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	<title>National Drug Prevention Alliance &#38; PPP</title>
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	<link>http://drugprevent.org.uk/ppp</link>
	<description>information collected by NDPA and PPP about drugs, prevention and support</description>
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		<title>Marijuana 2.0 &#8211; It is a Different Drug Now</title>
		<link>http://drugprevent.org.uk/ppp/2012/01/marijuana-2-0-it-is-a-different-drug-now/</link>
		<comments>http://drugprevent.org.uk/ppp/2012/01/marijuana-2-0-it-is-a-different-drug-now/#comments</comments>
		<pubDate>Sun, 22 Jan 2012 21:53:32 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Cannabis]]></category>
		<category><![CDATA[Uncategorized]]></category>
		<category><![CDATA[Youth]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8066</guid>
		<description><![CDATA[After alcohol, marijuana is the drug most abused by teens. In fact, marijuana is the most widely used illicit substance in the United States and recent data show an uptrend in teen marijuana use during 2009. Unfortunately, it is still viewed today by many as being the same drug it was 45 years ago, despite [...]]]></description>
			<content:encoded><![CDATA[<p>After alcohol, marijuana is the drug most abused by teens. In fact,<br />
marijuana is the most widely used illicit substance in the United<br />
States and recent data show an uptrend in teen marijuana use<br />
during 2009. Unfortunately, it is still viewed today by many as being the<br />
same drug it was 45 years ago, despite significant changes.</p>
<p><strong>Prevalence of Use by</strong><br />
<strong>Teens in the past 30 ays (2008)</strong><br />
<strong>monitoringthefuture.org</strong><br />
Marijuana:<br />
8th grade &#8211; 5.8%,<br />
10th grade- 13.8%,<br />
12th grade &#8211; 19.4%</p>
<p align="LEFT">It is a Stronger Drug Today. Delta9-tetrahydrocannabinol, A.K.A. &#8220;THC&#8221; is<br />
the active ingredient in marijuana that creates the intoxication. From the<br />
1960’s &#8211; 1970’s marijuana was around 1/2 % &#8211; 3% THC. For 35 years following the 70‘s, the potency of<br />
marijuana slowly increased to 4% by 1995.</p>
<p align="LEFT"><a href="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Marijuana-2.0-It-is-a-Different-Drug-Now-Graph1.jpg"><img class="alignnone size-full wp-image-8067" title="From 1995 to 2008 the percentage of THC went from 4% to just over 10% on average" src="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Marijuana-2.0-It-is-a-Different-Drug-Now-Graph1.jpg" alt="From 1995 to 2008 the percentage of THC went from 4% to just over 10% on average" width="600" height="295" /></a></p>
<p><strong>2. Average Age of First Use is Younger Today.</strong><br />
Replicated studies since 1997 have provided a convergence of data suggesting that “early onset of first<br />
intoxication,” as an independent variable, significantly increases the probability of developing addiction. 4<br />
Today the average age of first intoxication is 12 years old. This compares to the 1960’s when marijuana<br />
was primarily used by college students.<br />
One study by (1997) Grant &amp; Dawson, shows the probability of a person developing addiction based<br />
on age of first intoxication in the chart below. In addition to age as a variable if the drug-user has a<br />
genetic family history of addiction then the risk factor is increased by 15 percent. See chart below.</p>
<p><a href="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Marijuana-2.0-It-is-a-Different-Drug-Now-Graph21.jpg"><img class="alignnone size-full wp-image-8073" title="Marijuana-2.0---It-is-a-Different-Drug-Now---Graph2" src="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Marijuana-2.0-It-is-a-Different-Drug-Now-Graph21.jpg" alt="" width="400" height="300" /></a></p>
<p>&nbsp;</p>
<p align="LEFT"><strong>3. Marijuana Then vs. Today &#8211; A Picture is Worth a Thousand Words:</strong></p>
<p><strong><span style="font-family: LucidaGrande-Bold;">Marijuana Then:</span></strong></p>
<div class="mceTemp">
<dl id="attachment_8074" class="wp-caption alignnone" style="width: 90px;">
<dt class="wp-caption-dt"><a href="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Marijuana-Then.jpg"><img class="size-full wp-image-8074" title="Marijuana-Then" src="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Marijuana-Then.jpg" alt="Marijuana Then" width="298" height="287" /></a></dt>
</dl>
</div>
<p class="wp-caption-dd">
<p>&nbsp;</p>
<p><strong>Paraphernalia Then:</strong></p>
<p>&nbsp;</p>
<p><a href="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Paraphernalia-Then2.png"><img class="alignnone size-full wp-image-8076" title="Paraphernalia Then2" src="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Paraphernalia-Then2.png" alt="" width="80" height="160" /> </a>  <a href="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Paraphernalia-Then11.png"><img class="alignnone size-full wp-image-8078" title="Paraphernalia Then" src="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Paraphernalia-Then11.png" alt="" width="132" height="85" /> </a>    <a href="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Paraphernalia-Then3.png"><img class="alignnone size-full wp-image-8077" title="Paraphernalia Then" src="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Paraphernalia-Then3.png" alt="" width="156" height="116" /></a></p>
<p>&nbsp;</p>
<p><strong>Marijuana Today:</strong></p>
<p>20 &#8211; 25% THC)</p>
<p><a href="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Marijuana-Today1.png.jpg"><img class="alignnone size-medium wp-image-8081" title="Marijuana Today1.png" src="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Marijuana-Today1.png-300x200.jpg" alt="Marijuana Today" width="300" height="200" /></a>   <a href="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Marijuana-Today2.png1.jpg"><img class="alignnone size-medium wp-image-8083" title="Marijuana Today2.png" src="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Marijuana-Today2.png1-300x200.jpg" alt="Marijuana Today" width="300" height="200" /></a></p>
<p>&nbsp;</p>
<p><strong>Paraphernalia Today</strong></p>
<p>Vaporizer, Grinder, Blunt Wrap</p>
<p><a href="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Paraphernalia-Today-Vaporizer.jpg"><img class="alignnone size-medium wp-image-8084" title="Paraphernalia Today - Vaporizer" src="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Paraphernalia-Today-Vaporizer-300x225.jpg" alt="Vaporizer" width="300" height="225" /> </a>  <a href="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Paraphernalia-Today-Grinder.jpg"><img class="alignnone size-full wp-image-8085" title="Paraphernalia Today - Grinder" src="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Paraphernalia-Today-Grinder.jpg" alt="Grinder" width="152" height="79" /> </a>  <a href="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Paraphernalia-Today-Blunt-Wrap.jpg"><img class="alignnone size-full wp-image-8086" title="Paraphernalia Today - Blunt Wrap" src="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Paraphernalia-Today-Blunt-Wrap.jpg" alt="Blunt Wrap" width="96" height="124" /></a></p>
<p align="LEFT">Clearly this is not the same marijuana used 40 years ago or certainly prior to 1995. For many, this grade of<br />
marijuana has only been accessible from “cannabis clubs.” At the same time, because the cost of the marijuana<br />
in the clubs was so expensive, many card holders still purchased marijuana from dealers on the street.<br />
However, with the economic contraction high grade marijuana prices have fallen in many of the cannabis clubs<br />
and access is now easier. Moreover, seeds to grow highly potent marijuana are easily purchased via the internet.<br />
Clients in our program state that “the weed is so sticky I need to use a weed grinder to break it up if I want to<br />
roll a blunt.”</p>
<p align="LEFT">
<p><strong>4. Withdrawal From the Drug Can Occur Today:</strong></p>
<p>t the 2009 medical doctor’s CSAM conference in San Francisco, a focus was on how to manage marijuana<br />
withdrawal with Gabapentin. Withdrawal symptoms include loss of appetite, problems sleeping and anxiety.<br />
Clearly people did not experience withdrawal 40 years ago and medicines weren’t being explored to manage<br />
withdrawal symptoms. Finally, with regard to teens, any drug being abused inhibits normal neural, emotional<br />
and social development, which can create a pathological relationship to intoxication resulting in negative<br />
consequences with school, family, money, friendships, romantic attachments, health, mental health, sports,<br />
employment, etc.</p>
<p align="LEFT">
<p align="LEFT"><strong>Final Thoughts: Evaluations, Education</strong><br />
<strong>&amp;Treatment</strong></p>
<p align="LEFT"><strong></strong></p>
<p align="LEFT">Marijuana is not the innocuous drug that some believe it to be.<br />
Too often parents and professionals base their understanding of<br />
the drug from their own personal use 20 years ago. One of the<br />
<a href="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Final-Thoughts.jpg"><img class="alignleft size-full wp-image-8088" title="Final Thoughts" src="http://drugprevent.org.uk/ppp/wp-content/uploads/2012/01/Final-Thoughts.jpg" alt="" width="88" height="100" /></a>biggest challenges facing professionals<br />
who specialize in the treatment of teen<br />
and young adult addictive disorders is<br />
that the intervention is not only with the<br />
individual, but it is also with the family,<br />
other health care professionals, schools,<br />
and legal system, who might “minimize”<br />
or discount the severity of marijuana<br />
abuse. Statements such as “It is only<br />
marijuana,” “at least it isn’t oxycontin,<br />
meth, etc” are examples of the type of<br />
denial described as “minimizing.” These messages from<br />
various systems support denial for the individual who is having<br />
consequences in different areas of their life because of the drug.<br />
For this reason, intervention must occur with the individual,<br />
family and community in order to be effective. It is also<br />
important that if families are seeking help for their child who is<br />
abusing drugs, they should seek professionals who are specially<br />
trained in adolescent and young adult addiction. If you are a<br />
parent or a professional working with teens and it is discovered<br />
that they have used, regardless of the frequency, an evaluation<br />
by a specialist is warranted. The individual needs to become<br />
educated, explore their relationship to intoxication and examine<br />
how it has already impacted different areas of their life in<br />
addition to learning new affect regulation and relational skills to<br />
move beyond this in their life. In addition, the family needs<br />
education on teen addiction, an understanding on how the brain,<br />
emotional, and social development are thwarted by drug use.<br />
An examination of parental denial &amp; enabling is needed as well<br />
as help with developing and implementing a good home<br />
contract, drug testing and education regarding how to be both a<br />
supportive resource for their child meanwhile maintaining a<br />
zero tolerance of drug use.</p>
<p align="LEFT">
<strong>Sources:</strong><br />
1. Janet E. Joy, Stanley J. Watson, Jr., and John A Benson, Jr., &#8220;Marijuana and Medicine:<br />
Assessing the Science Base,&#8221; Division of Neuroscience and Behavioral Research, Institute of<br />
Medicine (Washington, DC: National Academy Press, 1999).<br />
2. <a href="http://www.monitoringthefuture.org/data/09data.html#2009data-drugs">http://www.monitoringthefuture.org/data/09data.html#2009data-drugs</a><br />
3. <a href="http://www.justice.gov/ndic/pubs37/37035/national.html">http://www.justice.gov/ndic/pubs37/37035/national.html</a><br />
4. (1997) Grant &amp; Dawson, Journal of Substance Abuse, Vol. 9<br />
5. <a href="http://www.oas.samhsa.gov/newUsers.html">http://www.oas.samhsa.gov/newUsers.html</a><br />
6. (1997) Grant &amp; Dawson, Journal of Substance Abuse</p>
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		</item>
		<item>
		<title>Failure of Portugal&#8217;s drug legalisation experiment</title>
		<link>http://drugprevent.org.uk/ppp/2011/12/failure-of-portugals-drug-legalisation-experiment/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/12/failure-of-portugals-drug-legalisation-experiment/#comments</comments>
		<pubDate>Sat, 10 Dec 2011 18:23:44 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Europe]]></category>
		<category><![CDATA[Political Sector (Drug Politics)]]></category>
		<category><![CDATA[Social Affairs (Drug Politics)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8063</guid>
		<description><![CDATA[Congratulations to our colleague Manuel Pinto Coelho in Portugal.  This is an enormous victory!  The very liberal drug policy of Portugal is crumbling. &#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212; Dear colleagues, As you can see, although in Portuguese, it is official &#8211; Portuguese IDT and all its staff including the president Goulão has been abolished. The mask fell down and [...]]]></description>
			<content:encoded><![CDATA[<p><em>Congratulations to our colleague Manuel Pinto Coelho in Portugal.  This is an enormous victory!  The very liberal drug policy of Portugal is crumbling.</em></p>
<p><em>&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;&#8212;</em></p>
<p>Dear colleagues,</p>
<p>As you can see, although in Portuguese, it is official &#8211; Portuguese IDT and all its staff including the president Goulão has been abolished.</p>
<p>The mask fell down and there is no more &#8220;magnificent Portuguese model &#8211; an example to the world&#8221;. I hope Portuguese authorities decision may arrive in time to dissuade the rest of the world don’t follow countries like Mexico, Argentina and Czech Republic &#8211; as you know unfortunately they did bite the hook and decriminalized drugs already.</p>
<p> The magnificent Health Minister Paulo Macedo (ex-responsible by the treasure and finances) is now trying to understand how it was possible the existence of so many holes of so many millions of euros, opening the eyes FINALY to some personal and/or corporate interests some years ago installed&#8230; and as you can imagine there is a (very) few people very worried about&#8230;!</p>
<p>Now there is the SICAD with the competencies of&#8230;&#8221;&#8230;planeamento e acompanhamento de programas de redução do consumo de substâncias psicoactivas, na prevenção dos comportamentos aditivos e na diminuição das dependências num novo serviço criado no âmbito da administração directa do Ministério da Saúde&#8221;</p>
<p>that means, the&#8221;&#8230; planning and following up of programs to reduce the consumption of psychoactive substances, prevention of addictive behaviours and diminishing of dependencies in a new service born in Health Ministry direct administration.&#8221;</p>
<p>Treatment and harm reduction structures are since today within the responsibility of the several structures in the ground of National Health Service untied to central services&#8230; So the licences to internments and other services became responsibility of each and every ARS &#8211; Health Regional Administration accordingly its needs in the ground.</p>
<p> This is a big victory of good sense and REASON and very good news to everyone who suffer with drug dependence, giving to all of us more wings to believe that our efforts must go on moving always forward a drug free society</p>
<p> Manuel Pinto Coelho, International Task Force on Strategic Drug Policy.  Dec. 2011</p>
<p>&nbsp;</p>
<p>&nbsp;</p>
]]></content:encoded>
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		</item>
		<item>
		<title>Marijuana Impaired Driving: A serious safety problem</title>
		<link>http://drugprevent.org.uk/ppp/2011/12/marijuana-impaired-driving-a-serious-safety-problem/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/12/marijuana-impaired-driving-a-serious-safety-problem/#comments</comments>
		<pubDate>Sat, 10 Dec 2011 18:10:44 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[USA]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8060</guid>
		<description><![CDATA[While “medical marijuana” and marijuana legalization are common topics in the news, little attention is given to a large and growing body of research showing that marijuana impaired driving is a major cause of crashes, injuries and deaths. The overall number of traffic fatalities has continued to decrease nationally over the past 40 years,1 reaching [...]]]></description>
			<content:encoded><![CDATA[<p>While “medical marijuana” and marijuana legalization are common topics in the news, little attention is given to a large and growing body of research showing that marijuana impaired driving is a major cause of crashes, injuries and deaths.</p>
<p>The overall number of traffic fatalities has continued to decrease nationally over the past 40 years,1 reaching its lowest level in decades of 33,808 deaths in 2009.2 While reductions in alcohol-related fatalities have led this favorable trend, over the past five years the number of drug-positive drivers, including those positive for marijuana, has increased.3 Of all drug-positive fatally injured drivers in 2009, 28% were positive for marijuana. This accounts for 9% of all fatally injured drivers who had confirmed drug test results. Because many states do not conduct routine – or in some instances any – drug testing of fatally injured drivers, the prevalence of drugs, and in particular of marijuana, among fatally injured drivers is likely to be higher.</p>
<p> Marijuana is a Schedule I drug of abuse that has serious impairing psychological and physiological effects.4 A recent meta-analysis of nine epidemiological studies concluded that drivers who test positive for marijuana or report driving within three hours of marijuana use are more than twice as likely as other drivers to be involved in a crash.5 Studies of drivers involved in motor vehicle crashes support this conclusion. A study of seriously injured drivers admitted to a Maryland Level-1 shock trauma center showed that 26.9% of all seriously injured drivers tested positive for marijuana.6 A study of fatally injured drivers inWashingtonStateshowed</p>
<p>12.7% tested positive for marijuana and that among alcohol-positive drivers, 17.3% also tested positive for marijuana. The combination of marijuana use and alcohol is of great concern as evidence shows that low doses of marijuana combined with low doses of alcohol causes severe impairment.7 These data also show that combining alcohol and marijuana is common among seriously injured and dead drivers.</p>
<p>Efforts to reduce drunk driving have included strong legislation, effective enforcement and massive national education campaigns, yielding impressive results. The number of fatally injured drivers with illegal blood alcohol concentrations (BAC) of 0.08 g/dL has decreased 49% from 21,113 deaths in 1982 to 10,839 deaths in 2009.8 Similar actions must be taken to reduce drugged driving, including marijuana-specific initiatives since marijuana is by far the leading cause of drugged driving crashes, injuries and deaths. The problem of drugged driving received national attention for the first time in 2010, when the White House Office of National Drug Control Policy (ONDCP) identified reducing drugged driving a national priority in the <em>National Drug Control Strategy</em>.9 In 2011, ONDCP renewed its commitment to work to reduce drugged driving by 10% over the next 5 years in the 2011</p>
<p><strong>Commentary </strong><strong>December 8, 2011</strong></p>
<p> The national rate of illicit drug use has increased in recent years after a long-term decline, largely due to increases in marijuana use, particularly among young adults.11 Increased marijuana use poses a heightened risk on the nation&#8217;s roads and highways. As perceived risk of marijuana use has decreased, particularly among youth, the rate of marijuana use has increased.12</p>
<p> The emergence of “medical marijuana” in 16 states and the District of Columbia have made national headlines, sending a strong, misleading message to the public that marijuana use is safe and that marijuana is a “medicine”, leading to increases in marijuana use. Adding to the more permissive state laws and to the changing perceptions of risk of marijuana use, a discussion paper released by the Institute for the Study of Labor recently has received significant international press attention for its conclusions that “medical marijuana” laws cause decreased traffic fatalities and decreased alcohol consumption.13 Analyzing three states which permit “medical marijuana” (Vermont, Rhode Island and Montana), the authors conclude that</p>
<p>“medical marijuana” increases adult marijuana use and not youth marijuana use; that increased adult marijuana use is associated with decreased alcohol use; and that the decrease in adult alcohol use in these states after their approval of “medical marijuana” led to fewer motor vehicle crashes and fatalities.</p>
<p> As stated by General Arthur Dean, Chairman and CEO of the Community Anti-Drug Coalitions of America (CADCA), there are three significant problems with this non-peer-reviewed discussion paper:</p>
<p> “(1) the study methodology is greatly flawed; and,</p>
<p>   (2) the study’s authors disregard a large body of evidence showing     that          marijuana and alcohol are compliments; and,</p>
<p> (3) The study’s authors disregard mounting evidence that marijuana use is linked with impaired driving.”14</p>
<p>Former White House Drug Policy Advisor Kevin Sabet, Ph.D. points out that this paper’s authors “clearly dismiss or ignore research about the effects of medical marijuana that happen to be inconsistent with their conclusions.”15 In particular, a recent peer-reviewed study showed that rates of youth marijuana use are higher in states with “medical marijuana” than in states without “medical marijuana,” noting need for further research.16</p>
<p> Marijuana is not a substitute for alcohol; rather, the use of marijuana and alcohol is complementary. People use both marijuana and alcohol, though not necessarily at the same time. The larger point is however, how could the introduction of “medical marijuana” laws have resulted in such large reductions on the states’ alcohol consumption and highway deaths when only tiny percentages of the states’ populations are “medical marijuana” users?Vermonthas 349 registered “medical marijuana” users, or 0.05% of the state population.Rhode Islandhas an estimated 3,000 users, less than 1% of the state population.Montanahas over 27,000 registered users, accounting for nearly 3% of the state population. These small percentages of the states’ populations could not conceivably account for the large reductions in alcohol use and traffic fatalities reported in this study. What is most noteworthy about this discussion paper is the media coverage it has received. There is a strong contrast between the widespread media coverage of this non-peer-reviewed, obviously misleading, paper and the virtual absence of media attention to the many scientifically 3 sophisticated, peer-reviewed studies showing the significant highway safety threat posed by marijuana use. The large and ever-growing evidence that marijuana use is a significant contributor to highway crashes and deaths should be highlighted in any discussion of “medical marijuana” laws which by all accounts, including the proponents of “medical marijuana,” increase this drug’s availability and use.</p>
<p> “Medical marijuana” states are not immune to the consequences of marijuana impaired driving.Montana, which had the second-highest rate of alcohol impaired fatalities in the nation in 2009, is no exception to the problems of marijuana and drugged driving.17 Like other states, among drivers arrested for Driving Under the Influence (DUI) inMontana, marijuana is the most widely detected drug. From 2007-2010, the presence of marijuana among DUI suspects inMontanaincreased over 100%.18 In addition, during this period of time, the number of DUI suspects who</p>
<p>tested positive for both marijuana and alcohol increased by over 180%. Among fatally injury crashes in 2010, 38% involved drugs, 33% involved alcohol, and 14% involved drugs and alcohol.</p>
<p> Two important and related national improvements are cause for celebration: a decreased number of fatal crashes and a decreased number of alcohol-related motor vehicle fatalities. Despite these notable public health and public safety achievements, fatal crashes remain a significant problem, with clear evidence that drug use, and in particular marijuana use, is causing a large proportion of these preventable deaths. While nationally alcohol use has remained stable in recent years, marijuana use has increased,19 particularly among young adults.20 Contrary to the conclusions of the recent discussion paper, increasing marijuana use increases highway fatalities. It does not decrease them.</p>
<p><strong>Robert L. DuPont, M.D. </strong><strong>President, Institute for Behavior and Health, Inc.  </strong><strong>First Director, National Institute on Drug Abuse (NIDA) 1973 to 1978</strong></p>
<p><strong> </strong>Source:  <a href="http://www.ibhinc.org/">www.ibhinc.org</a>. Dec 2011</p>
<p> References</p>
<p>1 National HighwayTraffic Safety Administration. (2009). 2008 Traffic Safety Annual Assessment. <em>Traffic Safety</em></p>
<p><em>Facts. </em>Washington,DC:NHTSANationalCenter for Statistics and Analysis. Retrieved December 8, 2011 from</p>
<p>http://www-nrd.nhtsa.dot.gov/pubs/811172.pdf</p>
<p>2 National HighwayTraffic Safety Administration. (n.d.). Fatality Analysis Reporting System (FARS)</p>
<p>Encyclopedia. Retrieved December 8, 2011 from http://www-fars.nhtsa.dot.gov/Main/index.aspx</p>
<p>3 National HighwayTraffic Safety Administration. (2010). Drug involvement of fatally injured drivers. <em>Traffic</em></p>
<p><em>Safety Facts</em>. DOT HS 811 415.</p>
<p>4Couper, F.J., &amp;Logan, B.K. (2004). Drugs and human performance fact sheets.Washington,DC: National</p>
<p>Highway Traffic Safety Administration. DOT HS 809 725. Retrieved December 8, 2011 from:</p>
<p>http://www.nhtsa.gov/people/injury/research/job185drugs/drugs_web.pdf</p>
<p>5 Li, M., Brady, J.E., DiMaggio, C.J., Lusardi, A.R., Tzong, K.Y., &amp; Li, G. (2011). Marijuana use and motor vehicle</p>
<p>crashes. <em>Epidemiological Reviews. </em>doi: 10.1093/epirev/mxr017</p>
<p>6 Walsh, M., Flegel, R., Atkins, R., Cangianelli,L.A., Cooper, C., Welsh, C., &amp; Kerns., T.J. (2005). Drug and</p>
<p>alcohol use among drivers admitted to a Level-1 Trauma Center. <em>Accident Analysis and Prevention</em>, <em>37</em>(5), 894-901.</p>
<p>7 Ramaekers, J.G., Robbe, H.W., O&#8217;Hanlon, J.F. (2000). Marijuana, alcohol and actual driving performance. <em>Human</em></p>
<p><em>Psychopharmacology</em>, <em>15</em>(7), 551-558.</p>
<p>8 The Century Council. (2010). State ofDrunkDriving Fatalities in America 2009.Arlington,VA: The Century</p>
<p>Council. Retrieved December 8, 2011 from: http://www.centurycouncil.org/files/material/files/SODDFIA.pdf</p>
<p>9 Office of National Drug Control Policy. (2010). National drug control strategy, 2010.Washington,DC: Office of</p>
<p>National Drug Control Policy. Retrieved December 8, 2011 from</p>
<p>http://www.whitehouse.gov/sites/default/files/ondcp/policy-and-research/ndcs2010_0.pdf</p>
<p>10 Office of National Drug Control Policy. (2011). National drug control strategy, 2011.Washington,DC: Office of</p>
<p>National Drug Control Policy. Retrieved December 8, 2011 from</p>
<p>http://www.whitehouse.gov/sites/default/files/ondcp/ndcs2011.pdf</p>
<p>11 Substance Abuse and Mental Health Services Administration. (2011, September 8). National survey shows a rise</p>
<p>in illicit drug use from 2008 to 2010. SAMHSA News Release. Retrieved December 8, 2011 from</p>
<p>http://www.samhsa.gov/newsroom/advisories/1109075503.aspx</p>
<p>12 Center for Substance Abuse Research. (2011). Marijuana use continues to increase as perceived risk of use</p>
<p>decreases among U.S.high school seniors. <em>CESAR FAX, 20</em>(3). Retrieved December 8, 2011 from</p>
<p>http://www.cesar.umd.edu/cesar/cesarfax/vol20/20-03.pdf</p>
<p>13Anderson, D.M., &amp; Rees, D.I. (2011). Medical marijuana laws, traffic fatalities, and alcohol consumption.</p>
<p>Discussion paper series IZA DP No. 6112.Germany: Institute for the Study of Labor.</p>
<p>14Dean, A. (2011, December 5). Why “study” linking medical marijuana and driving reductions is flawed.</p>
<p>Community Anti-Drug Coalitions ofAmerica. Retrieved December 6, 2011 from:</p>
<p>http://www.cadca.org/blogs/detail/why-%E2%80%9Cstudy%E2%80%9D-linking-medical-marijuana-drivingfatality-</p>
<p>reductions-flawed</p>
<p>15 Sabet, K.A. (2011, December 5). Does medical marijuana really reduce alcohol crash fatalities? <em>Huffington Post</em>.</p>
<p>Retrieved December 8, 2011 from http://www.huffingtonpost.com/kevin-a-sabet-phd/media-report-medicalmarijuana_</p>
<p>b_1129654.html?ref=politics</p>
<p>16 Wall, M.M., Poh, E., Cerda, M., Keyes, K.M., Galea, S., &amp; Hasin, D.S. (2011). Adolescent marijuana user from</p>
<p>2002 to 2008: higher in states with medical marijuana laws, cause still unclear. <em>Annals of Epidemiology, 21</em>(9):714-</p>
<p>716.</p>
<p>17MontanaDepartment of Transportation. (2011, August). Overview ofMontana’s Impaired Driving Problem.</p>
<p>Retrieved December 8, 2011 from: http://www.mdt.mt.gov/safety/docs/impaired_driving_prob_overview_2011.pdf</p>
<p>18MontanaDepartment of Transportation. (2011, August). Overview ofMontana’s Impaired Driving Problem.</p>
<p>Retrieved December 8, 2011 from: http://www.mdt.mt.gov/safety/docs/impaired_driving_prob_overview_2011.pdf</p>
<p>19 Substance Abuse and Mental Health Services Administration. (2011). Results from the 2010 National Survey on</p>
<p>Drug Use and Health: Summary of National Findings, NSDUH Series H-41, HHS Publication No. (SMA) 11-4658.</p>
<p>Rockville,MD: Substance Abuse and Mental Health Services Administration. Retrieved December 8, 2011 from:</p>
<p>http://www.samhsa.gov/data/NSDUH/2k10Results/Web/HTML/2k10Results.htm</p>
<p>20 Substance Abuse and Mental Health Services Administration. (2011, September 8). National survey shows a rise</p>
<p>in illicit drug use from 2008 to 2010. SAMHSA News Release. Retrieved December 8, 2011 from</p>
<p>http://www.samhsa.gov/newsroom/advisories/1109075503.aspx</p>
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		<title>Smoking and Binge Drinking Raises Oral-Cancer Risk</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/smoking-and-binge-drinking-raises-oral-cancer-risk-4/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/smoking-and-binge-drinking-raises-oral-cancer-risk-4/#comments</comments>
		<pubDate>Sun, 27 Nov 2011 16:01:45 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Alcohol]]></category>
		<category><![CDATA[Cannabis]]></category>
		<category><![CDATA[Health]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8055</guid>
		<description><![CDATA[New research suggests that people who smoke and drink heavily are more at risk for oral cancer, the Researchers from King&#8217;s College in London, England, found an increase in oral cancer among men and women in their 20s and 30s who smoke and binge drink. The researchers said that when tobacco smoke combines with alcohol, [...]]]></description>
			<content:encoded><![CDATA[<p>New research suggests that people who smoke and drink heavily are more at risk for oral cancer, the Researchers from King&#8217;s College in London, England, found an increase in oral cancer among men and women in their 20s and 30s who smoke and binge drink.</p>
<p>The researchers said that when tobacco smoke combines with alcohol, it produces dangerous levels of cancer-causing chemicals that attack the lining of the mouth.</p>
<p>&#8220;Our data show that smoking, drinking and poor diet are major risk factors, and that the younger people start smoking and drinking, the higher the risk,&#8221; said Newell Johnson, a professor of oral pathology at King&#8217;s College</p>
<p>Source: Daily Telegraph,  London  reported Nov. 9.2004</p>
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		<title>Skunk and Mental Illness</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/skunk-and-mental-illness/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/skunk-and-mental-illness/#comments</comments>
		<pubDate>Sun, 27 Nov 2011 15:47:42 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Cannabis]]></category>
		<category><![CDATA[Health]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8051</guid>
		<description><![CDATA[Record numbers of teenagers are requiring drug treatment as a result of smoking skunk, the highly potent cannabis strain that is 25 times stronger than resin sold a decade ago. More than 22,000 people were treated in 2007  for cannabis addiction &#8211; and almost half of those affected were under 18. With doctors and drugs [...]]]></description>
			<content:encoded><![CDATA[<p>Record numbers of teenagers are requiring drug treatment as a result of smoking skunk, the highly potent cannabis strain that is 25 times stronger than resin sold a decade ago.</p>
<p>More than 22,000 people were treated in 2007  for cannabis addiction &#8211; and almost half of those affected were under 18. With doctors and drugs experts are warning that skunk can be as damaging as cocaine and heroin, leading to mental health problems and psychosis for thousands  &#8211; an IoS editorial states that there is growing proof that skunk causes mental illness and psychosis.</p>
<p>The decision comes as statistics from the NHS National Treatment Agency show that the number of young people in treatment almost doubled from about 5,000 in 2005 to 9,600 in 2006, and that 13,000 adults also needed treatment.</p>
<p>The skunk smoked by the majority of young Britons bears no relation to traditional cannabis resin &#8211; with a 25-fold increase in the amount of the main psychoactive ingredient, tetrahydrocannabidinol (THC), typically found in the early 1990s. New research being published in this week&#8217;s Lancet (2008)  will show how cannabis is more dangerous than LSD and ecstasy. Experts analysed 20 substances for addictiveness, social harm and physical damage. The results will increase the pressure on the Government to have a full debate on drugs, and a new independent UK drug policy commission being launched next month will call for a rethink on the issue.</p>
<p>The findings last night reignited the debate about cannabis use, with a growing number of specialists saying that the drug bears no relation to the substance most law-makers would recognise. Professor Colin Blakemore, chief of the Medical Research Council, who backed the original Independent  campaign for cannabis to be decriminalised, has also changed his mind.</p>
<p>He said: &#8220;The link between cannabis and psychosis is quite clear now; it wasn&#8217;t 10 years ago.&#8221;</p>
<p>Many medical specialists agree that the debate has changed. Robin Murray, professor of psychiatry at London&#8217;s Institute of Psychiatry, estimates that at least 25,000 of the 250,000 schizophrenics in the UK could have avoided the illness if they had not used cannabis. &#8220;The number of people taking cannabis may not be rising, but what people are taking is much more powerful, so there is a question of whether a few years on we may see more people getting ill as a consequence of that.&#8221;</p>
<p>&#8220;Society has seriously underestimated how dangerous cannabis really is,&#8221; said Professor Neil McKeganey, from Glasgow University&#8217;s Centre for Drug Misuse Research. &#8220;We could well see over the next 10 years increasing numbers of young people in serious difficulties.&#8221;</p>
<p>Politicians have also hardened their stance. David Cameron, the Conservative leader, has changed his mind over the classification of cannabis, after backing successful calls to downgrade the drug from B to C in 2002. He abandoned that position last year, before the IoS revealed that he had smoked cannabis as a teenager, and now wants the drug&#8217;s original classification to be restored.</p>
<p>Source  IoS  Dec. 2008</p>
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		<title>Separate Genes Responsible for Drinking, Alcoholism</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/separate-genes-responsible-for-drinking-alcoholism-4/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/separate-genes-responsible-for-drinking-alcoholism-4/#comments</comments>
		<pubDate>Sun, 27 Nov 2011 15:41:41 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Alcohol]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8047</guid>
		<description><![CDATA[New research finds there are some genes that affect one but not the other WEDNESDAY, Aug. 18 (HealthDayNews) &#8212; Some people can drink a lot of alcohol without becoming addicted, and specific genes may help explain why, researchers say. In a new study of Australian twins, scientists found that separate genes appear to be responsible, [...]]]></description>
			<content:encoded><![CDATA[<p>New research finds there are some genes that affect one but not the other</p>
<p>WEDNESDAY, Aug. 18 (HealthDayNews) &#8212; Some people can drink a lot of alcohol without becoming addicted, and specific genes may help explain why, researchers say.</p>
<p>In a new study of Australian twins, scientists found that separate genes appear to be responsible, to some degree, for dependence on alcohol &#8212; addiction &#8212; and how much people drink. Understanding how these genetic factors work together should give researchers more insight into treatment of alcoholism in its various forms, said study co-author John B. Whitfield, a researcher at Royal Prince Alfred Hospital in Australia.</p>
<p>Alcoholism and alcohol consumption may appear to be similar, but researchers are increasingly studying them separately. Consumption refers to the amount of alcohol that someone drinks, while addiction refers to a person&#8217;s inability to go without a drink.</p>
<p>&#8220;The transition from social alcohol consumption to alcohol dependence is a gradual process, and it is often hard to notice it,&#8221; said Dr. Alexei B. Kampov-Polevoi, an assistant professor of psychiatry at Mount Sinai School of Medicine. &#8220;As a result, many alcoholics and their family members continue to think that a person &#8216;just drinks too much&#8217; while this person already developed alcohol dependence and requires treatment.&#8221;</p>
<p>Whitfield and his colleagues examined statistics about alcohol use from three studies of Australian twins completed between 1980 and 1995. The number of twins in the studies declined from 8,184 in 1980 to 3,378 in 1995.<br />
The findings appear in the August issue of Alcoholism: Clinical &amp; Experimental Research.</p>
<p>The researchers found twins who were genetically similar were more likely to consume similar amounts of alcohol. According to the study, some genes affected both addiction and alcohol intake, while some just affected addiction.</p>
<p>&#8220;We found (as others have also found) that alcohol dependence is partly, but not entirely, due to genetic differences between people who are affected by it and those who are not,&#8221; Whitfield said. &#8220;We also found that variation in the amount of alcohol that people habitually drink is subject to genetic influence, and that there is some &#8212; but not complete &#8212; overlap between the genes affecting these two things.&#8221;</p>
<p>Howard J. Edenberg, professor of biochemistry and molecular biology at Indiana University, said the findings &#8212; that genes separately affect alcoholism and drinking &#8212; are &#8220;reasonable.&#8221; But &#8220;that is a long way from identifying individual genes that actually are involved,&#8221; said Edenberg, whose own research is looking into that area.</p>
<p>So what should ordinary folks take from this study? &#8220;There is no direct and new message for people with alcoholism in their families; they are at higher risk than average but this has been known for some time and there is only a statistical risk, not a certainty by any means,&#8221; Whitfield said. &#8220;The more positive message for such people, and the community at large, is that we are learning more about alcohol use and alcohol-related problems and their causes.&#8221;</p>
<p>Source  By Randy Dotinga<br />
HealthDay Reporter    August  2004</p>
<p>&nbsp;</p>
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		<title>Research to Look at New Treatments for Heroin Addiction</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/research-to-look-at-new-treatments-for-heroin-addiction/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/research-to-look-at-new-treatments-for-heroin-addiction/#comments</comments>
		<pubDate>Sun, 27 Nov 2011 15:19:38 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Addiction]]></category>
		<category><![CDATA[Heroin/Methadone]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8043</guid>
		<description><![CDATA[A clinical trial to test better treatment options for chronic heroin addiction is expected to begin in Vancouver at the end of this year. Led by researchers from Providence Health Care and the University of British Columbia, it&#8217;s the only clinical trial of its kind in North America. The Study to Assess Longer-term Opioid Medication [...]]]></description>
			<content:encoded><![CDATA[<p>A clinical trial to test better treatment options for chronic heroin addiction is expected to begin in Vancouver at the end of this year. Led by researchers from Providence Health Care and the University of British Columbia, it&#8217;s the only clinical trial of its kind in North America.</p>
<p>The Study to Assess Longer-term Opioid Medication Effectiveness (SALOME) is a carefully controlled three-year clinical trial that will test whether hydromorphone (Dilaudid(R)), a licensed pain medication, is as effective as diacetylmorphine, the active ingredient of heroin, at engaging the most vulnerable long-term street heroin users, so they will enroll in treatment programs and end their use of illicit drugs.</p>
<p>The intent of the SALOME project is to determine whether some participants become healthier and reduce their illicit drug use or are able to switch to other forms of treatment. SALOME also intends to test if, after stabilizing patients on injectable medications, they can transition to oral formulations without losing effectiveness.</p>
<p>This study builds on the North American Opiate Medication Initiative (NAOMI), which was North America&#8217;s first-ever clinical trial of prescribed heroin that took place from 2005 to 2008. NAOMI, which also was led by researchers from Providence Health Care and UBC, was a randomized trial aimed at testing whether medically prescribed heroin (diacetylmorphine) was more effective than methadone therapy for individuals with chronic heroin addiction who were not benefiting from other conventional treatments.</p>
<p>The results, published in the New England Journal of Medicine, showed that patients treated with the prescribed heroin were more likely to stay in treatment or quit heroin altogether and more likely to reduce their use of illegal drugs and other illegal activities than patients treated with oral methadone.</p>
<p>In the NAOMI study, the researchers also provided a small sample of patients with injectable hydromorphone, (Dilaudid(R)). An unexpected finding was that injection patients could not accurately discriminate whether they were receiving prescribed heroin or hydromorphone. The researchers also observed similar results and benefits with both these drugs although the small number of participants receiving hydromorphone did not permit any definite and scientifically valid conclusions to be drawn as to the efficacy of hydromorphone as a viable treatment option.</p>
<p>Should hydromorphone be proven to be as affective as heroin, the benefits of this form of injectable treatment may be more feasible and achievable without the emotional and regulatory barriers often presented by heroin maintenance.<br />
SALOME, led by Dr. Michael Krausz, the Providence Health Care/UBC B.C. Leadership Chair in Addiction Research and Dr. Eugenia Oviedo-Joekes, Providence Health Care researcher and an assistant professor in UBC&#8217;s School of Population and Public Health, will enroll 322 individuals with chronic heroin dependency who currently are not sufficiently benefiting from conventional therapies, such as methadone treatment, at one site based in Vancouver, BC.</p>
<p>In the first stage, half of the 322 participants will receive injectable prescribed heroin, and the other half will receive injectable hydromorphone. Stage I will involve six-months of treatment. All volunteers retained in injection treatment at the end of Stage I will be eligible to enter Stage II.</p>
<p>In Stage II, half of the participants will then continue injection treatment exactly as in Stage I on a blinded basis while the other half will switch to the oral equivalent of the same medication (prescribed heroin or hydromorphone). Stage II will also involve six-months of treatment.</p>
<p>Throughout the treatment period, social workers will be assigned to both groups to assist them with reaching other addiction services and community resources such as counseling, housing and job training services.</p>
<p>Some 60,000 to 90,000 persons are affected by opioid addiction in Canada. This study will enroll the most chronically drug-dependent members of Vancouver&#8217;s population &#8212; those who are not benefiting from other treatments, such as methadone therapy and abstinence-based programs, and continue injecting street heroin.</p>
<p>The SALOME study is funded by the Canadian Institutes of Health Research, the Government of Canada&#8217;s agency responsible for funding health research in Canada, Providence Health Care and the InnerChange Foundation.</p>
<p><strong>Quotes:</strong><br />
Dr. Perry Kendall, BC&#8217;s Provincial Health Officer -<br />
&#8220;SALOME addresses critical social and ethical concerns dealing with addiction. Opioid-dependent people are in need of treatment options to avoid marginalization from the health care system and this study aims to answer questions that could lead to improvements in the health of persons with chronic addictions and identify new ways of reintegrating this population into society.&#8221;</p>
<p>&#8220;If the SALOME study shows that hydromorphone can go head-to-head with heroin as an alternative therapy for people who have failed optimally provided methadone, then I think this should be part of the treatment continuum that&#8217;s available through licensed physicians.&#8221;</p>
<p>Dianne Doyle, Providence Health Care President and CEO -<br />
&#8220;Providence Health Care is supporting this research because it is so aligned with our mission, vision and values. We have a very long tradition of providing compassionate care to the most marginalized and needy in our community, including those suffering from addictions.&#8221;</p>
<p>&#8220;What we need to get from this research is a better understanding of what the right approaches are to treating addicted populations. In particular our hope would be that we could find a new approach for those people who are addicted and not benefiting from current approaches to care. This treatment option would be one more component of a range of services offered by Providence Health Care and Vancouver Coastal Health, all of which are intended to reduce the harm to individuals and others from drug use, and to support recovery from addiction and mental illness.&#8221;</p>
<p><strong>About Providence Health Care </strong></p>
<p>Providence Health Care is one of Canada&#8217;s largest faith-based health care organizations, operating 15 facilities within Vancouver Coastal Health. Guided by the principle &#8220;How you want to be treated,&#8221; PHC&#8217;s 1,200 physicians, 6,000 staff and 1,500 volunteers deliver compassionate care to patients and residents in British Columbia. Providence&#8217;s programs and services span the complete continuum of care and serve people throughout B.C. PHC operates one of two adult academic health science centres in the province, performs cutting-edge research in more than 30 clinical specialties, and focuses its services on six &#8220;populations of emphasis&#8221;: cardiopulmonary risks and illnesses, HIV/AIDS, mental health, renal risks and illness, specialized needs in aging and urban health.</p>
<p><strong>About the University of British Columbia </strong></p>
<p>The University of British Columbia (UBC) is one of North America&#8217;s largest public research and teaching institutions, and one of only two Canadian institutions consistently ranked among the world&#8217;s 40 best universities. Surrounded by the beauty of the Canadian West, it is a place that inspires bold, new ways of thinking that have helped make it a national leader in areas as diverse as community service learning, sustainability and research commercialization. UBC offers more than 50,000 students a range of innovative programs and attracts $550 million per year in research funding from government, non-profit organizations and industry through 7,000 grants.</p>
<p>To view the first video of the SALOME project, please visit the following link: <a href="http://www.youtube.com/watch?v=fFgV_bt8QAU&amp;feature=youtu.be">http://www.youtube.com/watch?v=fFgV_bt8QAU&amp;feature=youtu.be</a></p>
<p>To view the second video of the SALOME project, please visit the following link: <a href="http://www.youtube.com/watch?v=S8xfkkeHpdE&amp;feature=related">http://www.youtube.com/watch?v=S8xfkkeHpdE&amp;feature=related</a></p>
<p>Source:  <a href="http://www.marketwatch.com/">www.marketwatch.com</a>  13th Oct. 2011</p>
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		<title>CAMH study suggests increased risk of schizophrenia in heavy methamphetamine users</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/camh-study-suggests-increased-risk-of-schizophrenia-in-heavy-methamphetamine-users-2/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/camh-study-suggests-increased-risk-of-schizophrenia-in-heavy-methamphetamine-users-2/#comments</comments>
		<pubDate>Sun, 27 Nov 2011 15:15:09 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Brain and Behaviour]]></category>
		<category><![CDATA[Methamphetamine/GHB/Hallucinogens/Oxycodone]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8039</guid>
		<description><![CDATA[Canadian scientists also confirm previous research showing possible link between cannabis dependence and schizophrenia In the first worldwide study of its kind, scientists from Toronto&#8217;s Centre for Addiction and Mental Health (CAMH) found evidence that heavy methamphetamine users might have a higher risk of developing schizophrenia. This finding was based on a large study comparing [...]]]></description>
			<content:encoded><![CDATA[<p>Canadian scientists also confirm previous research showing possible link between cannabis dependence and schizophrenia</p>
<p>In the first worldwide study of its kind, scientists from Toronto&#8217;s Centre for Addiction and Mental Health (CAMH) found evidence that heavy methamphetamine users might have a higher risk of developing schizophrenia. This finding was based on a large study comparing the risk among methamphetamine users not only to a group that did not use drugs, but also to heavy users of other drugs.</p>
<p>The report will be published online on Nov. 8, 2011, at AJP in Advance, the advance edition of the American Journal of Psychiatry, the official journal of the American Psychiatric Association.</p>
<p>Methamphetamine and other amphetamine-type stimulants are the second most common type of illicit drug used worldwide.</p>
<p>&#8220;We found that people hospitalized for methamphetamine dependence who did not have a diagnosis of schizophrenia or psychotic symptoms at the start of our study period had an approximately 1.5 to 3.0-fold risk of subsequently being diagnosed with schizophrenia, compared with groups of patients who used cocaine, alcohol or opioid drugs,&#8221; says Dr. Russ Callaghan, the CAMH scientist who led the study. Dr. Callaghan also found that the increased risk of schizophrenia in methamphetamine users was similar to that of heavy users of cannabis.</p>
<p>To establish this association, the researchers examined California hospital records of patients admitted between 1990 and 2000 with diagnosis of dependence or abuse for several major abused drugs: methamphetamine, cannabis, alcohol, cocaine or opioids. They also included a control group of patients with appendicitis and no drug use. The methamphetamine group had 42,412 cases, while cannabis had 23,335.</p>
<p>Records were excluded if patients were dependent on more than one drug or had a diagnosis of schizophrenia or drug-induced psychosis during their initial hospitalization. Readmission records within California hospitals were analyzed for up to 10 years after the initial admission. The researchers then identified patients who were readmitted with a schizophrenia diagnosis in each drug group.</p>
<p>There has been a longstanding debate as to whether there is a connection between methamphetamine use and schizophrenia. Many Japanese clinicians have long believed that methamphetamine might cause a schizophrenia-like illness, based on their observations of high rates of psychosis among methamphetamine users admitted to psychiatric hospitals. However, they lacked long-term follow-up studies of methamphetamine users initially free of psychosis. In North America, this link has mostly been discounted, as psychiatrists believed that the psychosis was already present and undiagnosed in these methamphetamine users.</p>
<p>&#8220;We really do not understand how these drugs might increase schizophrenia risk,&#8221; says Dr. Stephen Kish, senior scientist and head of CAMH&#8217;s Human Brain Laboratory. &#8220;Perhaps repeated use of methamphetamine and cannabis in some susceptible individuals can trigger latent schizophrenia by sensitizing the brain to dopamine, a brain chemical thought to be associated with psychosis.&#8221; Dr. Kish also cautions that the findings do not apply to patients who take much lower and controlled doses of amphetamines or cannabis for medical purposes.</p>
<p>Since this is the first such study showing this potential link, the researchers emphasize that the results need to be confirmed in additional research involving long-term follow-up studies of methamphetamine users.</p>
<p>&#8220;We hope that understanding the nature of the drug addiction-schizophrenia relationship will help in developing better therapies for both conditions,&#8221; says Dr. Callaghan.</p>
<p>In an earlier study using California hospital records, the researchers found evidence for a possible association between heavy methamphetamine use and Parkinson&#8217;s disease.</p>
<p>Source:www.eurekalert.org.  8th Nov. 2011</p>
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		<title>Pot Shock</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/pot-shock-2/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/pot-shock-2/#comments</comments>
		<pubDate>Sun, 27 Nov 2011 15:12:42 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Brain and Behaviour]]></category>
		<category><![CDATA[Cannabis]]></category>
		<category><![CDATA[Youth]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8036</guid>
		<description><![CDATA[PATIENTS suffering the effects of cannabis abuse are being treated by Tasmanian public hospitals every day, says a leading health authority. People with short-term drug-induced psychosis and longer-term mental illness, compounded by pot smoking, are seeking medical help at an increasing rate.   Mental Health Services clinical statewide director Peter Norrie said the Royal Hobart Hospital [...]]]></description>
			<content:encoded><![CDATA[<p>PATIENTS suffering the effects of cannabis abuse are being treated by Tasmanian public hospitals every day, says a leading health authority.</p>
<p>People with short-term drug-induced psychosis and longer-term mental illness, compounded by pot smoking, are seeking medical help at an increasing rate.   Mental Health Services clinical statewide director Peter Norrie said the Royal Hobart Hospital was seeing many cannabis cases.</p>
<p>First-time pot smokers were turning up at the Royal with full-blown psychosis &#8212; delusional, confused and anxious.   Other more regular pot smokers with long-term mental illness were fronting for treatment for episodes likely to have been triggered or related to using cannabis. </p>
<p>&#8220;These days it&#8217;s close to every day,&#8221; said Dr Norrie, who is a senior clinical consultant psychiatrist at the Royal.   He said he was talking about &#8220;drug-induced psychosis or long-term mental illness associated with pot smoking&#8221;.   Dr Norrie said it was &#8220;very common&#8221; for first-time users to present with &#8220;floridly psychotic&#8221; behaviour.</p>
<p>He said psychiatrists were increasingly concerned with the link between substance abuse and mental illness.   Cannabis use had been linked with depression, anxiety and schizophrenia. International studies show modern strains of marijuana are from three to 10 times stronger than those used by previous generations.</p>
<p>&#8220;Clinically psychiatrists have suspected a link for many years and the latest research seems to confirm this,&#8221; Dr Norrie said.</p>
<p>&#8220;The chicken-and-egg debate has raged for years whether pot causes psychosis or people with a tendency to psychotic illness are predisposed to smoke pot.&#8221;</p>
<p>Dr Norrie said the first signs of schizophrenia were often a lack of engagement with society. But those symptoms could also be what is commonly known as &#8220;typically teenage&#8221; or a sign of the onset of depression.</p>
<p>Disengaged teenagers could then turn to cannabis.</p>
<p>If psychosis did occur it was hard to tell whether smoking pot was a cause or a symptom. Dr Norrie said some pot smokers appeared to be able to continue the habit without serious mental illness but others were prone to individual cases of psychosis or longer-term mental disease.</p>
<p>&#8220;There&#8217;s a certain group of people who smoke pot who are unlikely to develop mental illness but there&#8217;s certainly a significant number of the population who suffer from mental illness and pot smoking adds to the risk,&#8221; Dr Norrie said.</p>
<p>Drug-induced psychosis usually consists of paranoia, confusion and anxiety.</p>
<p>Sufferers present with memory problems and delusions. They can believe they have special powers, hear and see things that are not there and are unable to distinguish what is real.</p>
<p>Source: Sunday Tasmanian 30th January 2005</p>
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		<title>Oral cannabis induces psychosis at low levels</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/oral-cannabis-induces-psychosis-at-low-levels-2/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/oral-cannabis-induces-psychosis-at-low-levels-2/#comments</comments>
		<pubDate>Sun, 27 Nov 2011 15:08:34 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Cannabis]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8032</guid>
		<description><![CDATA[Last Updated: 2005-04-01 9:09:08 -0400 (Reuters Health) NEW YORK (Reuters Health) &#8211; Even in clinical situations where cannabis is administered orally at low doses, psychotic reactions can occur, Swiss researchers report the current issue of BMC Psychiatry. Recreational cannabis use has been associated with psychotic reactions, but this is the first such report in closely [...]]]></description>
			<content:encoded><![CDATA[<p>Last Updated: 2005-04-01 9:09:08 -0400 (Reuters Health)</p>
<p>NEW YORK (Reuters Health) &#8211; Even in clinical situations where cannabis is administered orally at low doses, psychotic reactions can occur, Swiss researchers report the current issue of BMC Psychiatry.</p>
<p>Recreational cannabis use has been associated with psychotic reactions, but this is the first such report in closely monitored subjects participating in a clinical trial, note Dr. Bernard Favrat and colleagues at Institut Universitaire de Medicine Legale in Lausanne.</p>
<p>Favrat&#8217;s group was conducting a study to examine the effects of ingestion of THC (delta-9-tetrahydrocannabinol) on psychomotor function and driving performance in eight occasional cannabis users.</p>
<p>The first case of psychosis was in a 22-year-old man given 20 milligrams of dronabinol, a synthetic THC. Ninety minutes after dronabinol administration he experienced severe anxiety and symptoms of psychosis, and was unable to perform the two psychometric tests.</p>
<p>Levels of THC and its active metabolite 11-OH-THC in the blood at the time of the strong adverse effects were 1.8 and 5.2 nanograms per milliliter, respectively.</p>
<p>The second case was also a 22-year-old man who developed severe anxiety one hour after taking 16.5 milligrams of a THC compound, when his THC blood level was 6.2 nanograms per milligram and 11-OH-THC was 3.9 nanograms per milligram. For several hours he was unable to perform psychometric tests</p>
<p>The authors note that smoking a 3.5-percent marijuana cigarette leads to blood concentrations of THC in the range of 50 to 100 nanograms per milliliter. They believe that oral administration produces higher levels of 11-OH-THC, with slower elimination.</p>
<p>Alternatively, they suggest that &#8220;consuming oral cannabis may produce more potent, yet unknown psychotomimetic metabolites of THC.&#8221;</p>
<p>&#8220;Doctors and users should be aware of the increasing availability of oral cannabis in &#8216;special&#8217; drinks or food as well as in medications under development,&#8221; which can result in &#8220;significant psychotic reactions,&#8221; Favrat&#8217;s group cautions.</p>
<p>SOURCE: BMC Psychiatry, April 1,2005.</p>
<p>&nbsp;</p>
<p>&nbsp;</p>
<p>&nbsp;</p>
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		<title>Myocardial Infarction Associated With Use of the Synthetic Cannabinoid K2</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/myocardial-infarction-associated-with-use-of-the-synthetic-cannabinoid-k2/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/myocardial-infarction-associated-with-use-of-the-synthetic-cannabinoid-k2/#comments</comments>
		<pubDate>Sun, 27 Nov 2011 15:07:16 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Cannabis]]></category>
		<category><![CDATA[Health]]></category>
		<category><![CDATA[Youth]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8029</guid>
		<description><![CDATA[Physicians who encounter myocardial infarction in teenagers should consider the possibility that the teens may have ingested K2, a form of synthetic cannabinoid, researchers said. &#8220;Although chest pain is a common presenting complaint of teenagers seen in emergency departments, chest pain from cardiac causes remains exceedingly rare,&#8221; Colin Kane, MD, a pediatric cardiologist at the [...]]]></description>
			<content:encoded><![CDATA[<p>Physicians who encounter myocardial infarction in teenagers should consider the possibility that the teens may have ingested K2, a form of synthetic cannabinoid, researchers said.</p>
<p>&#8220;Although chest pain is a common presenting complaint of teenagers seen in emergency departments, chest pain from cardiac causes remains exceedingly rare,&#8221; Colin Kane, MD, a pediatric cardiologist at the UT Southwestern Medical Center in Dallas, and colleagues wrote in the December issue ofPediatrics. &#8220;Use of illicit drugs causing chest pain and myocardial ischemia, however, must remain part of the differential diagnosis.&#8221;</p>
<p>The researchers reported on three cases of myocardial infarction in teenagers following ingestion of K2. Designer drugs containing synthetic cannabinoids have become more popular among teens, but little is known about their health implications.</p>
<p>K2 is a collection of herbs and spices that have been treated with a synthetic cannabinoid. The effects are said to be stronger than naturally occurring cannabis.</p>
<p>&#8220;These types of drugs give a marijuana-like effect but do not show up on drug screens,&#8221; Kane explained to MedPage Today. Therefore, careful questioning may be needed to elicit information about K2 exposure, the authors suggested.<br />
All three cases involved 16-year-old males with no previous health problems. Each complained of chest pains of at least three days&#8217; duration and presented between August and November of 2010.</p>
<p>Initial electrocardiograms revealed ST-segment elevation and high troponin levels. There was no personal or family history of early cardiac problems. Urine drugs screens noted the presence of THC in two patients. No other drugs, including cocaine and amphetamines, were found.</p>
<p>&#8220;When the first patient came we initially thought it was a virus that was affecting his heart,&#8221; said Kane. &#8220;The day after he was hospitalized, the chest pain, ECG, and laboratory test all changed dramatically. We went back to the patient and were more persistent about anything else he might have done. It just isn&#8217;t normal for a 16-year-old to have a heart attack.&#8221;<br />
Shortly thereafter, two new cases presented with similar findings. After establishing that these males also had smoked K2, Kane and colleagues became concerned because their patients were not having just chest pains, but actual heart attacks.</p>
<p>&#8220;I have since then seen a number of kids in my practice who have smoked K2 and complained of chest pains,&#8221; said Kane. &#8220;I haven&#8217;t seen any other frank heart attacks.&#8221;</p>
<p>This led them to wonder if there was something different about the K2 that was in circulation at that time. Another option is that teenagers were showing up in the emergency room, but the heart attacks were not found because it is so atypical in the age group.</p>
<p>&#8220;It is disconcerting and frightening that K2 is relatively easy to obtain and could have such serious health consequences,&#8221; said Kane. &#8220;Emergency and primary care doctors need to ask patients specifically about the use of K2 and synthetic marijuana. If the clinical findings fit, physicians should take the extra step and look for heart damage, even in previously healthy teenagers.&#8221;</p>
<p>Source:   <a href="http://www.pediatrics.aappublications.org/">www.pediatrics.aappublications.org</a> at University of Florida on November 14, 2011</p>
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		<title>Marijuana and Schizophrenia</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/marijuana-and-schizophrenia-3/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/marijuana-and-schizophrenia-3/#comments</comments>
		<pubDate>Sun, 27 Nov 2011 15:02:54 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Brain and Behaviour]]></category>
		<category><![CDATA[Marijuana and Medicine]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8025</guid>
		<description><![CDATA[Marijuana causes disruptions in concentration and memory similar to those that occur in people with schizophrenia, according to a new study. U.K. researchers measured the electrical activity from hundreds of neurons in the brains of rats given a drug that mimics the effects of cannabis, the psychoactive ingredient of marijuana. The effects of the drug [...]]]></description>
			<content:encoded><![CDATA[<p>Marijuana causes disruptions in concentration and memory similar to those that occur in people with schizophrenia, according to a new study.</p>
<p>U.K. researchers measured the electrical activity from hundreds of neurons in the brains of rats given a drug that mimics the effects of cannabis, the psychoactive ingredient of marijuana.</p>
<p>The effects of the drug on individual brain regions were subtle but the drug completely disrupted the coordinated brain waves across the hippocampus and prefrontal cortex. Both of these brain structures are essential for memory and decision-making and play a key role in schizophrenia.</p>
<p>Due to the &#8220;decoupling&#8221; of the hippocampus and prefrontal cortex, the rats were unable to make accurate decisions while attempting to find their way through a maze, the University of Bristol researchers said.</p>
<p>&#8220;Marijuana abuse is common among sufferers of schizophrenia and recent studies have shown that the psychoactive ingredient of marijuana can induce some symptoms of schizophrenia in healthy volunteers. These findings are therefore important for our understanding of psychiatric diseases, which may arise as a consequence of &#8216;disorchestrated brains&#8217; and could be treated by re-tuning brain activity,&#8221; lead author Matt Jones said in a university news release.</p>
<p>The study appears Oct. 25 in the Journal of Neuroscience.<br />
&#8220;These results are an important step forward in our understanding of how rhythmic activity in the brain underlies thought processes in health and disease,&#8221; study first author Michal Kucewicz said</p>
<p>Source: <a href="http://www.everydayhealth.com/">www.everydayhealth.com</a> Oct. 25, 2011</p>
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		<title>Marijuana Use and Adolescents: What Clinicians Need to Know</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/marijuana-use-and-adolescents-what-clinicians-need-to-know/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/marijuana-use-and-adolescents-what-clinicians-need-to-know/#comments</comments>
		<pubDate>Sun, 27 Nov 2011 15:00:07 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Marijuana and Medicine]]></category>
		<category><![CDATA[Youth]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8021</guid>
		<description><![CDATA[As marijuana use among teenagers increases and its perceived danger among this age group decreases, clinicians need to know the latest science about the harmful effects of the drug on the adolescent brain, according to a researcher at the University of Colorado, Denver. Paula Riggs, PhD, Professor of Psychiatry, notes the most recent Monitoring the [...]]]></description>
			<content:encoded><![CDATA[<p>As marijuana use among teenagers increases and its perceived danger among this age group decreases, clinicians need to know the latest science about the harmful effects of the drug on the adolescent brain, according to a researcher at the University of Colorado, Denver.</p>
<p>Paula Riggs, PhD, Professor of Psychiatry, notes the most recent Monitoring the Future Survey shows a significant increase in marijuana use, including daily marijuana use among U. S. high school students and a decrease in perceived risk of use. “There are a number of indicators, including the increasing number of states that have passed ‘medical marijuana’ legislation, and that society as a whole tends to view marijuana as a relatively benign, recreational drug. However, scientific research does not support this.”</p>
<p>A growing body of research shows that adolescent marijuana use can be detrimental to the brain development and may produce long-lasting neurocognitive deficits and increased risk of mental health problems including psychosis, said Dr. Riggs, who spoke about this topic at the recent California Society of Addiction Medicine meeting.</p>
<p>Marijuana is the most commonly used illicit drug in the United States. Although some have questioned whether marijuana is an addictive drug, scientific research shows that one in 10 people overall, and one in six adolescents, who use marijuana develop dependence or addiction, Dr. Riggs says. Research shows that marijuana can cause structural damage, neuronal loss and impair brain function on a number of levels, from basic motor coordination to more complex tasks, such as the ability to plan, organize, solve problems, remember, make decisions and control behavior and emotions.</p>
<p>Dr. Riggs also cited recent studies indicating that adolescents may be more vulnerable to addiction, in part due to rapid brain development. “Emerging research suggests that individuals who start using marijuana during their teenage years may have longer-lasting cognitive impairments in executive functioning than those who start later,” she says. “Animal studies also suggest that exposure to marijuana during adolescence compared to adulthood may increase the vulnerability or risk of developing addiction to other substances of abuse such as cocaine and methamphetamine.”</p>
<p>She adds, “It is important for pediatricians, psychiatrists and other mental health clinicians to be aware of current research because they are on the front line to identify teens when they first start to experiment. They need to be able to effectively screen adolescents for marijuana use, and be armed with the scientific facts to educate teens and families about associated risks.”</p>
<p>Source   <a href="http://www.partnershipatdrugfree.org/">www.partnershipatdrugfree.org</a>  Nov. 2011</p>
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		<title>CAMH study suggests increased risk of schizophrenia in heavy methamphetamine users</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/camh-study-suggests-increased-risk-of-schizophrenia-in-heavy-methamphetamine-users/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/camh-study-suggests-increased-risk-of-schizophrenia-in-heavy-methamphetamine-users/#comments</comments>
		<pubDate>Sun, 27 Nov 2011 14:56:14 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Brain and Behaviour]]></category>
		<category><![CDATA[Methamphetamine/GHB/Hallucinogens/Oxycodone]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8017</guid>
		<description><![CDATA[Canadian scientists also confirm previous research showing possible link between cannabis dependence and schizophrenia In the first worldwide study of its kind, scientists from Toronto&#8217;s Centre for Addiction and Mental Health (CAMH) found evidence that heavy methamphetamine users might have a higher risk of developing schizophrenia. This finding was based on a large study comparing [...]]]></description>
			<content:encoded><![CDATA[<p>Canadian scientists also confirm previous research showing possible link between cannabis dependence and schizophrenia</p>
<p>In the first worldwide study of its kind, scientists from Toronto&#8217;s Centre for Addiction and Mental Health (CAMH) found evidence that heavy methamphetamine users might have a higher risk of developing schizophrenia. This finding was based on a large study comparing the risk among methamphetamine users not only to a group that did not use drugs, but also to heavy users of other drugs.</p>
<p> The report will be published online on Nov. 8, 2011, at AJP in Advance, the advance edition of the American Journal of Psychiatry, the official journal of the American Psychiatric Association.</p>
<p>Methamphetamine and other amphetamine-type stimulants are the second most common type of illicit drug used worldwide.</p>
<p>&#8220;We found that people hospitalized for methamphetamine dependence who did not have a diagnosis of schizophrenia or psychotic symptoms at the start of our study period had an approximately 1.5 to 3.0-fold risk of subsequently being diagnosed with schizophrenia, compared with groups of patients who used cocaine, alcohol or opioid drugs,&#8221; says Dr. Russ Callaghan, the CAMH scientist who led the study. Dr. Callaghan also found that the increased risk of schizophrenia in methamphetamine users was similar to that of heavy users of cannabis.</p>
<p>To establish this association, the researchers examined California hospital records of patients admitted between 1990 and 2000 with diagnosis of dependence or abuse for several major abused drugs: methamphetamine, cannabis, alcohol, cocaine or opioids. They also included a control group of patients with appendicitis and no drug use. The methamphetamine group had 42,412 cases, while cannabis had 23,335.</p>
<p>Records were excluded if patients were dependent on more than one drug or had a diagnosis of schizophrenia or drug-induced psychosis during their initial hospitalization. Readmission records within California hospitals were analyzed for up to 10 years after the initial admission. The researchers then identified patients who were readmitted with a schizophrenia diagnosis in each drug group.</p>
<p>There has been a longstanding debate as to whether there is a connection between methamphetamine use and schizophrenia. Many Japanese clinicians have long believed that methamphetamine might cause a schizophrenia-like illness, based on their observations of high rates of psychosis among methamphetamine users admitted to psychiatric hospitals. However, they lacked long-term follow-up studies of methamphetamine users initially free of psychosis. In North America, this link has mostly been discounted, as psychiatrists believed that the psychosis was already present and undiagnosed in these methamphetamine users.</p>
<p>&#8220;We really do not understand how these drugs might increase schizophrenia risk,&#8221; says Dr. Stephen Kish, senior scientist and head of CAMH&#8217;s Human Brain Laboratory. &#8220;Perhaps repeated use of methamphetamine and cannabis in some susceptible individuals can trigger latent schizophrenia by sensitizing the brain to dopamine, a brain chemical thought to be associated with psychosis.&#8221; Dr. Kish also cautions that the findings do not apply to patients who take much lower and controlled doses of amphetamines or cannabis for medical purposes.</p>
<p>Since this is the first such study showing this potential link, the researchers emphasize that the results need to be confirmed in additional research involving long-term follow-up studies of methamphetamine users.</p>
<p>&#8220;We hope that understanding the nature of the drug addiction-schizophrenia relationship will help in developing better therapies for both conditions,&#8221; says Dr. Callaghan.</p>
<p>In an earlier study using California hospital records, the researchers found evidence for a possible association between heavy methamphetamine use and Parkinson&#8217;s disease.</p>
<p>Source:www.eurekalert.org.  8th Nov. 2011</p>
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		<title>Implementation of evidence-based substance use disorder continuing care interventions.</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/implementation-of-evidence-based-substance-use-disorder-continuing-care-interventions/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/implementation-of-evidence-based-substance-use-disorder-continuing-care-interventions/#comments</comments>
		<pubDate>Sun, 27 Nov 2011 14:53:04 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Solvent abuse]]></category>
		<category><![CDATA[Treatment/Addiction]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8012</guid>
		<description><![CDATA[As this review comments, people treated for substance use often remain precariously balanced between recovery and relapse. Widely seen as valuable if not essential, aftercare is nevertheless more the exception than the rule. How to reverse that ratio is the issue addressed by these leading US analysts. Summary Continuing care or aftercare is the stage [...]]]></description>
			<content:encoded><![CDATA[<p>As this review comments, people treated for substance use often remain precariously balanced between recovery and relapse. Widely seen as valuable if not essential, aftercare is nevertheless more the exception than the rule. How to reverse that ratio is the issue addressed by these leading US analysts.</p>
<p><strong>Summary</strong><br />
Continuing care or aftercare is the stage of treatment following initial, more intensive, treatment. This review focused on psychosocial continuing care interventions (such as individual, telephone, couples, and group therapy; case management; home visits; and brief check-ups) and 12-step mutual aid support groups. Studies of brief continuing care interventions (up to six months) have usually involved standard programmes provided after residential treatment. In contrast, most longer interventions are adapt their frequency or nature in response to systematic assessments of how well the client is doing.</p>
<p>Despite a broadly supportive research record, few efforts have been made to implement and sustain these interventions, and in practice few clients who might benefit from continuing care services actually receive a sufficient dose, either because they do not complete the initial treatment, do not start continuing care, or do not remain in it for a significant time. Among other things, this review seeks to better understand this discrepancy and make recommendations for future implementation efforts.</p>
<p><strong>Effectiveness of continuing care</strong><br />
Though this review and studies have focused on either continuing care treatment or mutual aid groups, it should be remembered that many individuals participate in both and that using both sources of support is associated with improved treatment outcomes.</p>
<p>Studies have shown that receiving continuing care services is generally but not always associated with improved long-term substance use outcomes. This small and varied corpus of studies precludes conclusions about which approaches work best. However, the findings support certain general principles. Among these are increasing the duration of care to at least a year, ongoing monitoring of clients, reaching out actively to engage and link clients to care, and using incentives to improve treatment outcomes. Relatively low-cost practices can dramatically improve rates of sustained engagement in continuing care such as low level incentives and active outreach following discharge or drop-out. In contrast, the theoretical orientation and intensity of the interventions appear less important.</p>
<p>As well as or instead of continuing care treatment services, mutual aid groups are important continuing care resources. The most prevalent like Alcoholics Anonymous and Narcotics Anonymous follow 12-step principles. Several studies have shown that attending these groups after initial treatment is associated with positive substance use outcomes, though they are unable to prove that attendance causes these gains. Additional to attendance as such, being more involved in the groups (such as getting a sponsor or reading 12-step literature) has also been associated with better substance use outcomes. In practice though, while most US patients start attending groups, most of these are no longer attending a year later.</p>
<p>Interventions to promote participation in 12-step mutual aid groups can be traced to the Twelve Step Facilitation therapy trialled in Project MATCH. This large US study of treatment for alcohol dependence found this approach achieved significantly higher rates of continuous abstinence (and equivalent outcomes on other drinking measures) than cognitive-behavioural therapy and a therapy based on motivational interviewing, and did so because it led more patients to engage in 12-step activities. Similar results have emerged from other studies.</p>
<p><strong>Implementing continuing care</strong><br />
A search for studies not of the effectiveness of continuing care, but of how to implement it, uncovered 28 relevant articles and others known to the authors of the review or referenced in the literature. To organise the analysis of these studies, the reviewers used the Consolidated Framework for Implementation Research. In respect of health care innovations in general, this model identifies five implementation domains, each divided in to several sub-domains. The five main domains with relevant examples are:</p>
<p>• Characteristics of the intervention (in this case, continuing care) such as the strength of the evidence for its effectiveness and how far it was adapted to fit the particular circumstances in which it was being implemented.<br />
• Outer setting, which refers to the economic, political, and social environment surrounding and influencing the organisation undertaking the implementation – in this case, typically substance use treatment services; included here might be national political drivers, availability of funding, the demand from patients, and (especially in the case of 12-step groups) the degree to which the broader society is receptive to the intervention&#8217;s philosophy.<br />
• Inner setting is pertinent features of the implementing organisation including the degree to which its structures, internal communication mechanisms, resources, leadership, and culture facilitate the adoption of continuing care or the particular continuing care intervention being implemented.<br />
• Characteristics of the individuals conducting the intervention – in this case, typically addiction counsellors – such as what they believe about the intervention and how enthusiastic and ready they are to implement it.<br />
• Process of implementation – the extent and quality of the implementation effort, including the degree to which relevant staff are actively engaged, the efficiency with which the implementation is carried out, the extent to which progress is appropriately monitored against specific goals and progress news fed back to the participants, and the extent to which this feedback is used to adapt and promote implementation.</p>
<p>Generally the few relevant studies have not developed or supported specific packages to promote continuing care implementation. The one clear example of a specific and manualised intervention is Twelve Step Facilitation therapy, an approach which has been successfully adapted to different circumstances and populations. More general evidence-based interventions for promoting mutual aid participation typically entail active and directive efforts to engage and retain clients, including education on the benefits of the groups, orientation to involvement with these groups, and connection with group members to help motivate involvement following initial treatment.</p>
<p>In more detail and organised under the main headings of the Consolidated Framework for Implementation Research, research offers the following guidance.</p>
<p>Intervention Characteristics Clinicians generally know that the evidence for continuing care is strong yet often continue to use interventions and practices without empirical support. A significant number of studies suggest that many interventions can be adapted to the needs of specific sites. Twelve Step Facilitation therapy has for example been successfully adapted to a group format, to focus on individuals&#8217; broader social networks rather than just 12-step groups, and to accommodate individuals with mental health as well as substance use problems. Similarly, treatment-based continuing care efforts have been conducted successfully using telephone and home-based visits and with different types of providers. One difficulty is the relative complexity of such interventions. Knowledge gaps include the relative advantages and cost-effectiveness of different continuing care interventions, and what are their core or essential components as opposed to those which can safely be adapted.</p>
<p>Outer Setting The most frequently cited factors related to successful continuing care implementation are located in the outer setting domain, especially the importance of client characteristics such as their needs and resources to support continuing care involvement. African-Americans (compared to Caucasians), and clients with more severe substance use problems, are more likely to engage in continuing care for a longer time. Psychiatric disorders seem no barrier to engagement in continuing care. Patients who see staff members as supportive and have more recovery resources are more likely to engage in treatment-based continuing care. Clients with beliefs consistent with a disease model or spiritual approach to recovery, women, and those with less prior experience with 12-step groups, may be more easily engaged in mutual aid groups, and those mandated to attend by courts may do as well as those who are not. In addition to client characteristics, the convenience of continuing care is an important facilitating factor while lack of funding is a common and significant barrier. Additionally, inviting mutual aid group members to contact patients in the initial treatment service facilitates post-treatment linkages. The role of external incentives and policies appears to be an extremely important area for future implementation efforts to address and better understand.</p>
<p>Inner Setting Focusing on the treatment service, those oriented to 12-step approaches facilitate linkage to 12-step mutual aid. Low rates of staff and supervisor turnover and multi-stakeholder involvement are important to sustaining continuing care treatment interventions. Goals or benchmarks that allow programmes to monitor performance and modify interventions in response are important factors in successful continuing care implementation. Mutual aid group engagement is facilitated by strong therapeutic alliances, greater supportiveness, and spirituality during initial treatment. Use of incentives with staff to promote implementation of continuing care practices appear to be a potentially powerful, but underused facilitator. Little is known about the implementation climate, including goals and benchmarks for continuing care interventions, or about the role of programme readiness for change (eg, resources and knowledge) as they relate to continuing care implementation.</p>
<p>Characteristics of the individual provider Treatment and mutual aid continuing care implementation are facilitated by providers and programme leaders with beliefs and attitudes supportive of the particular intervention, while a lack of knowledge about the effectiveness of interventions can be a significant barrier. Additionally, clinicians who are in recovery themselves, who have fewer concerns about religion or spirituality as a part of treatment, without allegiance to non-12-step approaches to treatment, and those who require abstinence during treatment, are more likely to facilitate 12-step mutual aid involvement following treatment. It is clear that future implementation efforts will need to address important characteristics such as the knowledge, beliefs, motivation, and self-efficacy of both providers and clients to maximise the potential for implementation success.</p>
<p>Implementation Process Successful continuing care implementation efforts have tended to address the important constructs of planning, engaging, executing, and reflecting and evaluating implementation efforts. These activities will be critical in the development and testing of implementation strategies.</p>
<p><strong>Implication for researchers and clinicians</strong><br />
Having summarised continuing care implementation research, the review ended by drawing out the implications of these findings for researchers and clinicians. Though scarce, viewed through the lens of the Consolidated Framework for Implementation Research, existing research provides a starting point for closing the gap between research and clinical practice. Formative evaluations intended to develop interventions to promote continuing care should be informed by this literature, and these evaluations should address all five domains, or deploy other comprehensive implementation models. Additionally, two primary recommendations emerge from this review.</p>
<p>Basic Continuing Care Implementation Research Is Needed Despite its clinical importance, continuing care implementation research has been relatively neglected. Both the treatment and mutual aid continuing care implementation literature have findings relevant to all five major domains of the Consolidated Framework for Implementation Research, but all the detailed strategies and factors within each domain have yet to be addressed. One of the most striking gaps is the lack of information on the relative advantages, disadvantages, and cost-effectiveness of continuing care interventions. Little is known too about and their core elements and the impact of incentives and/or consequences related to both the inner setting and outer setting domains.</p>
<p>Implementation Efforts Need to Address Multiple Domains The comprehensiveness of the Consolidated Framework for Implementation Research highlights that implementation efforts typically do not consider the importance of intervening across multiple domains. For instance, as already noted, the role of incentives and consequences in the inner setting and outer setting domains and at patient, counsellor and programme level, has been neglected. This review suggests that closing the gap between knowledge about continuing care interventions and their use will require a paradigm change in which both researchers and clinicians consider intervening across multiple domains rather than within a single domain, as has been typical thus far. Research-established interventions may have too few implementation facilitators and too many barriers for them to be adopted in particular settings without attention to all the relevant domains.</p>
<p>People treated for substance use often remain precariously balanced between recovery and relapse following initial treatment. As currently designed, the utility of treatment is limited by high post-treatment relapse and re-admission rates, and frequently prolonged addiction and treatment careers. Assertive linkage to continuing care helps individuals transition from brief experiments in sobriety (recovery initiation) to disease management and sustainable recovery maintenance, and an enhanced quality of life. It requires close connections between the worlds of professional treatment and community recovery support resources, and implementation of continuing care promotion procedures to enhance engagement and retention with these resources.</p>
<p>In the UK financial constraints and the recovery agenda have brought with them potentially conflicting expectations that treatment will end as soon as the patient seems able to manage on their own and rarely extend over years, yet will do more to reintegrate patients in society. More patients exiting briefer treatments would create an increasing potential caseload for aftercare services to ensure they remain safe and can rapidly re-enter treatment if relapse occurs or is threatened. How this configuration of forces will pan out and what it will mean for extended care in the form of aftercare or continuing care is unclear. Funders seeking to contain costs and maximise drug-free treatment exits may be reluctant to fund aftercare services, especially since UK research evidence that they make a difference is lacking, probably because studies have been few. On the other hand, low-cost, check-up style aftercare allied with free mutual aid groups may make it more acceptable to cut back on intensive and expensive initial treatment. These considerations are expanded on below.</p>
<p>The main recent British attempt to evaluate the contribution of aftercare was an analysis of the Scottish DORIS study. On several measures, it found that the few drug dependent patients who accessed aftercare after treatment in the early 2000s did better than the majority left to (or who chose to) fend on their own. However, it was unclear whether this could this be attributed to the aftercare, or whether these patients would have done well anyway. An attempt to statistically control for differences between patients still left recently being heroin free at the last 33-month follow-up associated with having received aftercare from the initial treatment agency. Having received aftercare following methadone maintenance or residential rehabilitation made little difference to whether patients had experienced a period of being entirely drug free. But consistently at each of the three follow-ups, aftercare following non-methadone community treatment like detoxification or psychosocial therapy was associated with about double the chance of having been drug free.</p>
<p>Formal aftercare from the treatment agency was not the only way patients sought to sustain their abstinence. Over the 33 months of the follow-up, nearly a quarter attended mutual aid groups like NA and AA. At each of the follow-ups, patients who had accessed aftercare and mutual aid were most likely to have been drug free for a period, generally those who accessed neither were least likely, and those who accessed one but not the other were in between.<br />
Whatever the meaning of these findings for aftercare&#8217;s effectiveness, it was clear that few patients received it, and neither was it targeted at those most at risk of relapse.</p>
<p>An English study of problem drinkers could more securely attribute the results to aftercare enhancements, because patients were randomly allocated to normal aftercare – up to three weekly support groups plus access to the unit&#8217;s recreational and social facilities – or to an additional 15 individual sessions modelled on an influential US approach called Early Warning Signs Relapse Prevention Training. During this, patients are helped to recognise personal warning signs of relapse by analysing their most recent attempts at recovery, and then to develop ways to manage these episodes without a return to drinking. Over the following year the benefits of more intensive aftercare were reflected in significantly fewer drinking days (22% of warning sign patients drank on a fifth or more of days compared to 40% in usual aftercare), fewer heavy drinking days (corresponding figures 18% and 28%), avoidance of any return to heavy drinking (45% v 26%), and improved mental well-being. In monetary terms, warning sign patients absorbed slightly less health service and rehabilitation resources, though slightly more if the warning sign regime was itself costed in. However, neither difference approached statistical significance.</p>
<p>In agreement with the featured review was a review of 11 studies which allocated patients at random or in a quasi-random manner to continuing care versus minimal or no continuing care. In terms of each study&#8217;s main substance use outcome measures, seven of the 11 found a clear and statistically significant advantage for continuing care. The review&#8217;s conclusions were endorsed by a panel of experts convened by the US Betty Ford Institute, who argued that extended and regular monitoring of the patient&#8217;s progress was the key component of continuing care and the one with the greatest evidence of effectiveness. Both review and recommendations were based largely on studies of aftercare following residential treatment.</p>
<p>While international and to a degree UK research is at least consistent with aftercare often being an aid to lasting remission, recommendations that it be implemented run up against a strong contrary trend in current UK policy, which emphasises not continuing care, but exit from the treatment system. Without denying the need for long-term care for some patients, the English strategy on drug misuse said services needed &#8220;to become much more ambitious for individuals to leave treatment free of their drug or alcohol dependence so they can recover fully &#8230; We will ensure that all those on a substitute prescription engage in recovery activities and build upon the 15,000 heroin and crack cocaine users who successfully leave treatment every year free of their drug(s) of dependence&#8221;. Scotland&#8217;s strategy too stressed the need for more patients to &#8220;move on from their addiction towards a drug-free life as a contributing member of society&#8221;, implying a corresponding shift away from extended and/or indefinite treatment.</p>
<p>Set against this drive to contain treatment, the recovery agenda has brought with it a greater emphasis on sustained and extensive life change, and an accompanying expectation that treatment services will do more for their patients than a brief treatment for their addiction. At the same time resources are no longer increasing and probably diminishing overall. One way to square this circle is to draw on the free resource of mutual aid groups which offer former patients 24-hour access to support, frequent support meetings, a new social circle, and a new way of life. It comes therefore as no surprise that they feature in recent commissioning guidance from England&#8217;s National Treatment Agency for Substance Misuse, which sees them as providing &#8220;valuable support and positive social networks for individuals who are addressing their dependency through treatment&#8221;. The advice to services is that &#8220;Details of how clients can access local recovery networks should be made available throughout their treatment journey. Services may wish to consider more active engagement with local mutual aid groups, for example making rooms within the treatment service or prisons available for meetings&#8221;. The agency now sees (see annual reports for 2009–10 and 2010–11) promoting mutual aid networks as a key way to achieve its objectives. Local service commissioners are being called on to ensure that the treatment system is better integrated with wider supportive services, among which mutual aid organisations are seen as the most prominent.</p>
<p>Source Psychology of Addictive Behaviors: 2011, 25(2), p. 238–251.</p>
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		<title>Drugs by Numbers</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/drugs-by-numbers/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/drugs-by-numbers/#comments</comments>
		<pubDate>Sun, 27 Nov 2011 14:42:18 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Cocaine]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8008</guid>
		<description><![CDATA[100% Three Andean countries – Colombia, Peru and Bolivia – are responsible for virtually all global coca leaf production, the raw material for cocaine. 149,100 In 2010, coca was cultivated on 149,100 hectares in the Andean countries – an area roughly one and a half times the size of Hong Kong – down from 221,300 [...]]]></description>
			<content:encoded><![CDATA[<p>100% Three Andean countries – Colombia, Peru and Bolivia – are responsible for virtually all global coca leaf production, the raw material for cocaine.</p>
<p>149,100 In 2010, coca was cultivated on 149,100 hectares in the Andean countries – an area roughly one and a half times the size of Hong Kong – down from 221,300 hectares in 2000.</p>
<p>6% In 2010, the global area under coca cultivation decreased by 6%, mainly due to a significant reduction in Colombia that was not entirely offset by a small increase in Peru.</p>
<p>732,000 The amount of cocaine seized worldwide in 2009 was 732,000kg – which refers to seizures unadjusted for purity. The United Nations Office on Drugs and Crime estimates that between 46% and 60% of cocaine produced was seized – an indication of the amount manufactured the previous year.</p>
<p>444,000 The best reading of data and estimates suggests that about 440,000kg of pure cocaine was consumed worldwide in 2009. This would be in line with a production estimate of about 1.1m kg and purity adjusted seizures of 615,000kg, plus agricultural and other losses of about 55,000kg (which represents 5% of production).</p>
<p>$85bn The value of the global cocaine market is lower than in the mid-1990s, when prices were much higher and the US market was strong. In 1995, the global market was worth about $165bn, while, in 2009, this had been reduced to just over half of that.</p>
<p>99% Of that $85bn income from global cocaine retail sales in 2009, traffickers are estimated to have reaped about $84bn (almost 99%). The rest went to Andean farmers.</p>
<p>5m The US has the highest prevalence of cocaine use (2.4% of the population, or five million people, aged 15-64), but there are indications of cocaine use declining in the last few years.</p>
<p>$33bn The amount of cocaine consumed in Europe has doubled in the last decade. The volume and value of the western and central European cocaine market, currently valued at $33bn, is now approaching parity with that of the US ($37bn).</p>
<p>80% Two thirds of European cocaine users live in three countries: the UK, Spain and Italy. With Germany and France, these countries represent 80% of European cocaine consumption.</p>
<p>272m Globally, the UN Office on Drugs and Crime estimates that between 149 and 272 million people – 3.3%-6.1% of the population aged 15-64 – used illicit drugs at least once in the previous year.</p>
<p>Source: United Nations Office on Drugs and Crime</p>
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		<title>Clever children more likely to end up on drugs</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/clever-children-more-likely-to-end-up-on-drugs/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/clever-children-more-likely-to-end-up-on-drugs/#comments</comments>
		<pubDate>Sun, 27 Nov 2011 14:35:40 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Youth]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8005</guid>
		<description><![CDATA[Scientists think they do so in part as a &#8220;coping strategy&#8221; to avoid bullying from their peers, and partially because they find life boring. The effect is more pronounced in girls than boys, with those exhibiting high IQs as children more than twice as likely to have tried cocaine or cannabis by the age of [...]]]></description>
			<content:encoded><![CDATA[<p>Scientists think they do so in part as a &#8220;coping strategy&#8221; to avoid bullying from their peers, and partially because they find life boring.</p>
<p>The effect is more pronounced in girls than boys, with those exhibiting high IQs as children more than twice as likely to have tried cocaine or cannabis by the age of 30, as those of lower intelligence.   The effect in boys with high IQs is also marked, with them being around 50 per cent more likely to have done so by that age as their less intelligent former classmates.</p>
<p>A team at Cardiff University analysed data from almost 8,000 people born in one week in April 1970, who were enrolled at birth in the ongoing British Cohort Study, which follows participants through life. All these children had their IQs tested between the age of five and 10.     Drug use, as reported by the participants themselves, was then recorded at 16 and 30 years of age.</p>
<p>At 16, 7.0 per cent of boys and 6.3 per cent of girls had used cannabis. This minority had &#8220;statistically significant higher mean childhood IQ scores&#8221; than non-users, according to the authors of the report, published in the Journal of Epidemiology and Community Health.    At 30, 35.4 per cent of men and 15.9 per cent of women had used cannabis, while the figures for cocaine were 8.6 and 3.6 per cent respectively.</p>
<p>The authors noted: &#8220;Across most drugs (except amphetamine in men), men and women who reported using in the past 12 months had a significantly higher childhood IQ score than those who reported no use.&#8221;<br />
They concluded: &#8220;High childhood IQ may increase the risk of substance abuse in early adulthood.&#8221;<br />
The study did not look into why this was the case, although it did not fine any relationship between the social class of the participants&#8217; parents and future drug use.</p>
<p>However, the authors noted that other studies suggested &#8220;intellectually &#8216;gifted children&#8217; [with an IQ higher than 130] report high levels of boredom and being stigmatised by peers, either of which could conceivably increase vulnerability to using drugs as an avoidant coping strategy&#8221;.</p>
<p>Dr James White of Cardiff University&#8217;s Centre for Development and Evaluation of Complex Interventions for Public Health Understanding, said: &#8220;Although it is not yet clear exactly why there should be a link between high IQ and illicit drug use, previous research has shown that people with a high IQ are more open to new experiences and keen on novelty and stimulation.&#8221;</p>
<p>Source: www.telegraph.co.uk  15th Nov.</p>
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		<title>Cannabis use and risk of lung cancer: a case–control study</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/cannabis-use-and-risk-of-lung-cancer-a-case%e2%80%93control-study/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/cannabis-use-and-risk-of-lung-cancer-a-case%e2%80%93control-study/#comments</comments>
		<pubDate>Sun, 27 Nov 2011 14:29:16 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Cannabis]]></category>
		<category><![CDATA[Health]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8002</guid>
		<description><![CDATA[ABSTRACT: The aim of the present study was to determine the risk of lung cancer associated with cannabis smoking. A case–control study of lung cancer in adults less than55 yrs of age was conducted in eight district health boards in New Zealand. Cases were identified from the New Zealand Cancer Registry and hospital databases. Controls [...]]]></description>
			<content:encoded><![CDATA[<p>ABSTRACT: The aim of the present study was to determine the risk of lung cancer associated with cannabis smoking.</p>
<p>A case–control study of lung cancer in adults  less than55 yrs of age was conducted in eight district health boards in New Zealand. Cases were identified from the New Zealand Cancer Registry and hospital databases. Controls were randomly selected from the electoral roll, with frequency matching to cases in 5-yr age groups and district health boards. Interviewer-administered questionnaires were used to assess possible risk factors, including cannabis use. The relative<br />
risk of lung cancer associated with cannabis smoking was estimated by logistic regression.</p>
<p>In total, 79 cases of lung cancer and 324 controls were included in the study. The risk of lung cancer increased 8% (95% confidence interval (CI) 2–15) for each joint-yr of cannabis smoking, after adjustment for confounding variables including cigarette smoking, and 7% (95% CI 5–9) for each pack-yr of cigarette smoking, after adjustment for confounding variables including cannabis smoking. The highest tertile of cannabis use was associated with an increased risk of lung cancer (relative risk 5.7 (95% CI 1.5–21.6)), after adjustment for confounding variables including cigarette smoking.</p>
<p>In conclusion, the results of the present study indicate that long-term cannabis use increases the risk of lung cancer in young adults.</p>
<p>Source  Eur Respir J 2008; 31: 280–286  2008</p>
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		<title>Marijuana as  Medicine ?</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/marijuana-as-medicine/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/marijuana-as-medicine/#comments</comments>
		<pubDate>Mon, 14 Nov 2011 12:13:54 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Drug Specifics (Drug Politics)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=8000</guid>
		<description><![CDATA[The attraction that the medical profession has for medical marihuana continues to mystify me. Many of the same physicians who will exercise exemplary caution in caring for their patients, will throw caution aside when it comes to pot. I know internists in private practice who refuse to accept new patients if they smoke tobacco. I [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;">The attraction that the medical profession has for medical marihuana continues to mystify me. Many of the same physicians who will exercise exemplary caution in caring for their patients, will throw caution aside when it comes to pot. I know internists in private practice who refuse to accept new patients if they smoke tobacco. I often wonder if they would have the same reaction if the patients smoke pot! Yesterday, I entered an online discussion by a medical group on this subject and I’ve pasted my comment below<br />
The medical profession needs to apologize for letting the public down on this one – once again. In the early 1900s, although medical organizations like the AMA were against patent medicines and refused to post ads in JAMA that did not list the ingredients of the products being promoted, there were quite a few doctors who nonetheless sold and promoted the use of patent medicines, most of which were worthless elixirs of cocaine or morphine or heroin or cannabis or combinations thereof, laced with copious amounts of alcohol, coloring agents and flavorings. They were promoted as curing everything from the common cold to cancer. Although the docs knew better, they argued that they were giving their patients what they wanted and if they didn’t, the patients would buy them on the street from sidewalk vendors who were not trained healthcare professionals. Ethical?<br />
As best we know, any “positive” effects of these nostrums came in the form of intoxication, a normal reaction to psychotropic substances, including alcohol. Therapeutic they were not. Even during alcohol prohibition (1919-1933), the federal government issued special prescriptions to physicians –only– that allowed them to prescribe “medicinal alcohol” in the form of wine, whiskey, and beer. Overnight, pharmacies became liquor stores. And doctors did, indeed, prescribe alcohol for medicinal purposes and plenty of it during Prohibition. Ethical?<br />
Fast-forward to the 1980s and 1990s and along comes the return of “medical marihuana.” This time, however, it’s not in the form of a tincture but, instead, promoted for use in its crude form as smoked marihuana. Not surprisingly, smoked pot today is touted as a cure-all for anything that ails one, from stress, to headache, to multiple sclerosis, to cancer pain and even cancer itself. How could a drug that’s so great be overlooked for so long by so many? Moreover, as in the case of alcohol prohibition, only doctors in certain states can prescribe (or recommend) it for medicinal purposes only. Ethical?<br />
What these brief histories have in common is the promotion and use of intoxicants for therapeutic purposes. In all three cases, doctors promoted the use of these substances knowing that the anecdotal evidence of efficacy was weak at best, unsupported by unbiased clinical trial data, and not likely to improve the patient’s condition but only mask symptoms temporarily through intoxication. Incidentally, we could add tobacco to this list, too. A favorite ad of mine comes from a 1950s magazine that shows a photo of a physician holding a cigarette with a caption proclaiming that in a national survey of physicians, more preferred Camels over any other brand of cigarette. Ethical?<br />
Wake up, America, and realize that whatever therapeutic molecules we might be able to squeeze out of the pot plant must be synthesized, purified, and manufactured to measured standards and dosing units before being used in medical treatment. Consider morphine and codeine. We don’t recommend that people grow opium poppies, harvest them, extract and chew the gum to get pain relief. Instead, we have synthesized and standardized pharmaceutically pure opiate medicines. Current pot research is underway to isolate and restructure the genetic pathways that provide pot’s psychic effect. This, scientists say, will be accomplished without interfering or reducing in any way the therapeutic properties of the beneficial cannabinoids in the plant. The final product will be safe and effective – far more effective as a medicine than smoking pot because dosing will be concentrated and stronger – and not controlled because there will be no psychotropic response. In effect, if pot truly has medicinal benefits independent of its intoxicating effects, they should be more readily available and useful in a finished pharmaceutical form. Also, users will be spared the toxic effects of inhaling smoke. Smoking anything &#8212; paper, tobacco, dry leaves, or pot &#8212; is not good for lung tissue of any living organism. Finally, the new pot without its psychic effect can be compared to decaffeinated coffee. It will have many of the same properties of the real thing except the kick. And, let’s face it, a good cup of Starbuck’s decaf can’t be distinguished from the regular stuff.<br />
When all this happens in a few years, pot heads now desperately trying to promote pot for everything and anything will be left with nothing but the fact that their story of pot’s medicinal history will join the other historical artifacts described above. Someday, their kids and grand-kids will look back and say the same thing that we say now when we look at those old cigarette ads from the 1950s: What were you thinking?  </p>
<p><em>Source: John Coleman Drug-Watch International Feb.2010<br />
</em><br />
</span></p>
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		<title>Methadone or Not ?</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/methadone-or-not/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/methadone-or-not/#comments</comments>
		<pubDate>Mon, 14 Nov 2011 12:12:37 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Drug Specifics (Drug Politics)]]></category>
		<category><![CDATA[Social Affairs (Drug Politics)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7998</guid>
		<description><![CDATA[Jay’s story has a familiar ring. The pre-teen experimentation with cannabis after his father walked out on the family, followed by flirtation with ecstasy and cocaine. He had smoked his first wrap of heroin before he was old enough to buy a pint of beer. But it was only when he was off the street, [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;">Jay’s story has a familiar ring. The pre-teen experimentation with cannabis after his father walked out on the family, followed by flirtation with ecstasy and cocaine. He had smoked his first wrap of heroin before he was old enough to buy a pint of beer. But it was only when he was off the street, safely incarcerated in a young offender institution, that methadone was added to Jay’s palate. As the gaunt teenager with grey skin shuffled from foot to foot in the West London drizzle, uncaringly dressed in a hooded tracksuit, his pin-pricked pupils scanned the streets.<br />
“I was running wild with a raging [heroin] habit when they got me,” he said. “They tried to detox me inside but as soon as I complained they put my dose of methadone up again. I came out needing drugs as much as when I went in.”<br />
His six-month stretch inside passed in a methadone-induced daze with, according to Jay, little attempt by prison staff to offer him a pathway to drug-free recovery. When he was released two years ago, Jay, whose only family contact is an elder brother he occasionally stays with, swiftly returned to the messy chaos of an opiate-obsessed existence. He thinks that he will be back in prison within weeks. “Most junkies I know want to be clean but if you can’t do it when you’re inside, when can you?” he says.<br />
Methadone, a heroin substitute that is more addictive than heroin itself, has assumed a dominant position in the State’s drug-control armoury. It is given to half the country’s estimated 300,000 heroin addicts while parliamentary answers have revealed that 65,000 prisoners were prescribed it in the past year, including nearly 20,000 on a maintenance programme which can last years — an annual rise of 57 per cent. In some patches of “broken Britain” it is responsible for more fatal overdoses than any other substance.<br />
Supporters say it stabilises addicts and protects society by removing the need for drug-financing crime sprees. Opponents argue that the State is happy to “park” people on methadone for years, giving up hope that addicts will ever lead a productive, drug-free life.<br />
One aspect most agree on is that drug addiction is a lucrative business. Professor Neil McKeganey, a leading opponent of mass methadone medicating, said: “There’s considerable financial incentive that drug users remain drug dependent.” Drug companies make millions from producing methadone, GPs in many parts of the country get paid in the region of £220 per methadone patient per year, pharmacists can get £200 administration fees plus about £1.50 per administered dose, while more than 150,000 people are employed in drug-action teams funded largely from the public purse.<br />
Mark Johnson, a former drug user who founded the charity Uservoice, said that although prisons are the ideal location for rehabilitation because they are “the only place that removes some people from dysfunction and gives them a respite”, the authorities are increasingly opting for the methadone route. “All we’re doing is containing the problem, not solving it,” he said.<br />
Several studies have shown that a residential-based abstinence programme lasting at least a month has a roughly one in four success rate, while a recent study on addicts in society showed that after three years on methadone only 3 per cent are drug-free.<br />
Despite this, however, the Government, backed by a cadre of policy experts and health professionals, is increasing its multi-million annual spend on methadone maintenance programmes. At the same time, at least 20 residential rehabilitation centres have closed in the past two years because primary care trusts have stopped referring clients. Last month Middlegate Lodge, the only residential rehab centre specifically for teenagers, closed.<br />
Just 850 prisoners were put on the relatively succesful 12-step abstinence programme last year. No figures are available for how many young offenders are prescribed methadone.<br />
Inspectors’ reports into young offenders’ institutions record that while alcohol and cannabis are the biggest substance problems, the use of methadone is being encouraged and is increasing.<br />
Kathy Gyngell, a drugs policy analyst for the Centre for Policy Studies, said that prescribing methadone to young offenders had become routine. She added: “It might appear the easier option but it leads to longer term problems. Individuals who historically used their short sentences to gain clean time now feel the necessity to carry on using methadone, as it takes no effort other than presenting themselves at the healthcare door to get it.”<br />
David Burrowes, a Tory justice spokesman, said that drug treatment was “characterised by methadone” and that a variety of treatment options needed to be available.<br />
Katherine, a former addict, whose descent into heroin addiction began after she was raped as a teenager, said that after a decade ricocheting between methadone in prison and heroin outside, she had finally kicked her habit after becoming one of the few prisoners to be offered a place on a RAPt (Rehabilitation for Addicted Prisoners Trust) abstinence programme.<br />
“Methadone is not a solution,” said Katherine, who left prison drug-free in 2008. “The message it gives is, ‘You come in with a habit and we’ll keep the habit and let you back out into society with no changes whatsoever.” She said that even in prison, addicts are able to exploit the system by using cotton wool to absorb the sickly-sweet green methadone linctus, before selling it on to other inmates and buying heroin with the proceeds.<br />
Rosie, who started taking heroin at the age of 14, was prescribed methadone after leaving a young offenders’ institution and said that she had never seen a succesful methadone-led withdrawal from drug use. “It’s almost more of a poison than heroin, there doesn’t ever seem to be an end to it,” she said. She became drug-free after attending an abstinence-based treatment centre provided by the Nelson Trust.<br />
To its advocates, though, methadone is a useful tool. At best, it stabilises addicts before they are weaned off; at worst, it can be used to maintain addicts long term, minimising the need for them to commit crime to pay for street heroin. Overall, drug-related crime is estimated to cost the country more than £13 billion a year.<br />
There are also risks associated with forcing prisoners to go cold turkey. Cynics suggest the prison authorities’ increasing enthusiasm for methadone may have something to do with the £750,000 it was forced to pay out in 2006 after almost 200 drug-addicted prisoners sued the Government, claiming that their rights were infringed when they were forced to withdraw suddenly.<br />
Even for those who claim to have benefited from it, methadone is at best a stopgap. James, 30, from Renfrewshire, had been a heroin user for nine years when he was given methadone in Barlinnie Prison, Glasgow. “Everything in prison was all about drugs,” he said. “Sometimes you couldn’t get any heroin and you couldn’t eat your dinner, you were in bed with all your clothes on, teeth rattling. They put me on 30ml of methadone, a low dose, and it settled me. I was a lot calmer; it was like a safety net.”<br />
Roger Howard, the chief executive of the UK Drug Policy Commission, an advocate for methadone, admits that it could not alone cure drug addicts. “What everyone wants is to reduce deaths from dangerous street heroin and to reduce criminality,” he said. “Methadone is not the problem. These people come with a bucketful of problems: abuse, unemployment, homelessness, family.”<br />
Professor McKeganey, who works at the Centre for Drug Misuse at the University of Glasgow, warned that Britain was sleepwalking into a situation similar to that in the Netherlands, where the Government provided places at old people’s homes for those with long-term methadone habits: the so-called “geriaddicts”. Mr Howard agreed: “There is a cohort who are probably so damaged and with such profound health problems that they will never get a job and will for ever rely on the State.”<br />
As he prepared to pad the darkened streets of West London in shoes as punctured as his bony, needle-marked forearm in an all-consuming search for his next hit, Jay pondered a parting question: if you could survive in prison on methadone alone, why not, when outside, give your daily, drug-free urine sample, take the supervised dose of methadone and shun street drugs?<br />
“But where would it get me? All right, the craving for smack’s not there but you soon get the craving for the meth. Nobody I know on a heoin ’script is getting any better. They’re just surviving.” </p>
<p><em>Source: Times Online 17th March 2010<br />
</em><br />
The cost of a quick fix<br />
2.4m       Methadone prescriptions written in 2007, a rise of 60 per cent since 2003<br />
£1.2bn     Amount spent annually by government (central and local) tackling drug use in England in 2009-10<br />
£15.3bn    The cost per year to society of problem drug use<br />
£13.9bn     The estimated cost of drug-related offending in 2003, made up of a £9.9 billion cost to victims of crime and £4 billion costs incurred by the criminal justice system<br />
330,000      Estimated number of problem drug users in England, of whom 166,000 are in some form of treatment programme </p>
<p><em>Sources: NAO, Drugscope, Home Office<br />
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		<title>Reliance on methadone a dangerous game for both users and the Government</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/reliance-on-methadone-a-dangerous-game-for-both-users-and-the-government/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/reliance-on-methadone-a-dangerous-game-for-both-users-and-the-government/#comments</comments>
		<pubDate>Mon, 14 Nov 2011 12:11:06 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Drug Specifics (Drug Politics)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7996</guid>
		<description><![CDATA[Whichever way you look at it, the Government’s increasing reliance on methadone to treat heroin addicts involves moral issues. Predominant among these is that the State is in effect cast in the role of drug dealer — conceivably for as long as the addicts live. The uneasy relationship becomes especially problematic when users die of [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;">Whichever way you look at it, the Government’s increasing reliance on methadone to treat heroin addicts involves moral issues. Predominant among these is that the State is in effect cast in the role of drug dealer — conceivably for as long as the addicts live.<br />
The uneasy relationship becomes especially problematic when users die of overdoses, having supplemented methadone with other street drugs.<br />
Never forget how dangerous this is. When official figures show that in some areas a third of the people who die from drug-related causes have methadone in their bodies, put there by the taxpayer, and that this proportion doubled from 2006-08, we are on dodgy ethical ground.<br />
Increasingly, it means the substance that is supposed to be a primary solution appears to be an intrinsic part of the problem. What methadone also represents is the transfer of personal responsibility for addiction away from the drug user. In this sense, the heroin substitute symbolises the cultural shift in modern drug policy: the addict is a victim who needs support and maintenance, rather than someone who should change their behaviour.<br />
This official non-judgmentalism is interesting, especially when there is public debate about the resources devoted to the consequences of smoking, alcohol and overeating — which are not illegal. The merits of a humane approach to drug addiction are apparent. No one argues that methadone is not a useful part of the weaponry. It’s relatively cheap; it can stabilise the lives of addicts who shoplift or supply heroin to others; and of course, rather importantly, it allows the Government to say that it is doing something.<br />
But what worries critics of methadone is not only its excessive use, but the lack of an exit strategy. In parts of the country there are addicts who have been taking it for decades. Even advocates concede that people are being kept on the drug for too long without any target to get them off.<br />
All of which makes it troubling to hear that young offenders are being prescribed it, if only because, without any commitment to get them off drugs, they may end up “parked” for many years of dependency.<br />
Professor Neil McKeganey, in his latest book, laments the lack of consensus about the goals of treatment, pointing out that although the majority of addicts want to be free of drugs, this is not facilitated by government policy. He wants to see a target limiting use to two years.<br />
Methadone is a smokescreen for the absence of alternatives when it comes to problem use. There appears to be no new thinking, no initiatives, few open minds; and indeed little political will.<br />
In a sense, the ubiquity of the heroin substitute is an admission that not only have social policies failed, but that we have no solutions for the consequences.</p>
<p><em>Source:  Times Online 17th March 2010<br />
</em><br />
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		<title>Methadone: &#8216;Too many use it as part of their drugs routine&#8217;</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/methadone-too-many-use-it-as-part-of-their-drugs-routine/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/methadone-too-many-use-it-as-part-of-their-drugs-routine/#comments</comments>
		<pubDate>Mon, 14 Nov 2011 12:00:45 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Social Affairs (Drug Politics)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7994</guid>
		<description><![CDATA[THE Conservative&#8217;s Holyrood justice spokesman Bill Aitken is no stranger to controversy and his plain-spoken attack on the methadone programme has re-ignited the debate about how best to tackle Scotland&#8217;s appalling epidemic of drug addiction. The debate about the effectiveness of the methadone programme has raged since its inception and there has always been opposition [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;">THE Conservative&#8217;s Holyrood justice spokesman Bill Aitken is no stranger to controversy and his plain-spoken attack on the methadone programme has re-ignited the debate about how best to tackle Scotland&#8217;s appalling epidemic of drug addiction.<br />
The debate about the effectiveness of the methadone programme has raged since its inception and there has always been opposition to the principle of handing out free opium-based drugs like methadone to addicts. But there is much in the basis of the scheme to commend it, not least that it has the potential to place those on the programme outwith the reach of criminals. Something that means addicts no longer have to steal to manage their habit and keeps them out of the clutches of gangsters should be a good thing. However, too many just use the methadone as part of their daily drugs routine and find ways of selling it on, despite measures like forcing them to take it in front of the pharmacist.</p>
<p>But the biggest flaw in the current system is that there is no incentive for the addicts to wean themselves off drugs altogether. The methadone programme is only a means to manage the habit, not break it and that must change. There is a great deal of truth in the belief that addicts must genuinely want to give up before any treatment can be successful, and that applies as much to alcohol, nicotine and gambling as it does to drugs. But therein lies the weakness in the system – following the logic, why should alcoholics not get free booze if it helps prevent them following a life of crime? Of course, that would be absurd, but so too is supplying junkies with more drugs for as long as they want without any prospect of a cure.</p>
<p>The extent of drug addiction across the whole of Scotland is only one facet of a wider social malaise, especially in the sprawling sink estates. Edinburgh has its own well-documented drug problems, but its scale is dwarfed  by the problems affecting places like Easterhouse. Why is it that some of these places have lower life-expectancy than deprived Third World countries? Why are thousands of people in a prosperous country able to see out their lives without ever doing a useful day&#8217;s work? And why is it necessary to lock up more people here than in most comparable Western countries? That there is a deep social malaise in much of Central Scotland is not in any doubt and the answer does not lie in throwing more public money at the problems without a radical re-think.</p>
<p>Bill Aitken&#8217;s description of drug addicts sitting &#8220;fat and happy&#8221; on the methadone programme might be over-blown – few of them are what any normal person would recognise as happy – but he does have a point. Free drugs on the state should only be part of a habit-breaking programme – anything less is little more than state-funded dealing.</p>
<p><em>Source: Edinburgh Evening News 17 March 2008<br />
</em><br />
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		<title>What Mr. Barnes failed to mention</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/what-mr-barnes-failed-to-mention/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/what-mr-barnes-failed-to-mention/#comments</comments>
		<pubDate>Mon, 14 Nov 2011 11:58:05 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Effects of Drugs (Drug Politics)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7992</guid>
		<description><![CDATA[“National Drug Treatment Monitoring System (NDTMS) data on treatment modalities shows that 131,110 people received substitute prescribing treatment. During the same period, 5,350 people received PTB-funded treatment in residential rehabilitation centres.” “We should be justly proud of what has been achieved in drug treatment. The sustained investment in recent years has resulted in significantly increased [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;">“National Drug Treatment Monitoring System (NDTMS) data on treatment modalities shows that 131,110 people received substitute prescribing treatment. During the same period, 5,350 people received PTB-funded treatment in residential rehabilitation centres.”</p>
<p> “We should be justly proud of what has been achieved in drug treatment. The sustained investment in recent years has resulted in significantly increased capacity, accessibility and take-up of drug treatment services. However, there is both the need and opportunity to further improve retention and treatment outcomes, not least by ensuring that problem drug users are able to access core services such as housing, employment and training opportunities. It is the time to evaluate where we are and how we can make drug treatment even better.&#8221;  Martin Barnes Drugscope</p>
<p>posted by Peter O&#8217;Loughlin on 14 Mar 2009 at 5:05 am </p>
<p>What Mr. Barnes failed to mention. </p>
<p>1.	Drug related deaths in accordance with the UK official definition are at their highest for 5 years. (Health Statistics Quarterly 39. Office of National Statistics) </p>
<p>2.	The level of HIV and other blood born diseases among Injecting Drug Users is higher now than at the start of the decade. </p>
<p>3	.In London where the prevalence of HIV is higher than anywhere else in England, 1 in 20 Injecting Drug Users is infected. </p>
<p>4	.In the remainder of England and Wales HIV among IDUs has risen from approximately one in 400 to 1 in 250 in 2006. </p>
<p>5	.The prevalence of hepatitis C among IDUs has increased from 33% in 2000 to 42% in 2006. </p>
<p>6.	Approximately on in 5 IDUs has hepatitis B, which represents an increase of something like 200 per cent since 1997. </p>
<p>The foregoing is neither ‘uninformed’, or ‘unwarranted’ criticism, they are however the inescapable facts which Mr. Barnes seems either keen to suppress or is unaware of, In either event his opinion that “we should be justly proud of what has been achieved in drug treatment”, is hardly a balanced judgment of the escalation in both drug related deaths and disease which is being inflicted on our society. Nor the increasing level of drug offences and drug related crime. </p>
<p>Whether or not this catastrophic outcome of our drug treatment strategy can be wholly attributed to the harm reduction treatment protocols which has dominated it for so many years, and of which Mr. Barnes is an enthusiast, is the principle cause of the seemingly out of control increase in death, disease and crime, is debatable, what is not debatable is that we have no reason or justification to be proud that we have presided over an escalating and avoidable loss of life, death and criminal activity; nor is Mr. Barnes justified in claiming that we have.</p>
<p>Follow-Up Opinions </p>
<p>Failings Found In Needle Exchange Services.<br />
posted by Mary Brett on 17 Mar 2009 at 1:49 pm<br />
Among other failings found in a survey by the NTA of needle exchanges in England 2006, 50% of DATS had no access to virus testing on site, 40% no immunisation in place, about a third lacked hygiene and safer technique discussions. </p>
<p>Data collection was poor &#8211; DATS able to provide numbers of clients and visits, quantity of equipment distributed and returned were in the minority. Only 74% of DATS, 55% of needle exchange service providers and 48% of pharmacies provided information. There was a lack of training for co-ordinators and access to facilities was mostly limited to the working week. Very few operated at weekends or during the evening or night. Largely missing was any monitoring of discarded needles or injuries arising from them to the public.<br />
Has anything been done to improve this situation? From the latest figures, quoted here, it would appear not to be the case.<br />
| </p>
<p>Quantity V Quality<br />
posted by Peter O&#8217;Loughlin on 18 Mar 2009 at 6:11 am<br />
Thank you for your revealing and interesting contribution Mary. </p>
<p>It seems as if the NTA&#8217;s obsession with numbers treated, rather than treatment outcomes could be a contributory factor to the spread of blood born disease. </p>
<p>It is also depressing to learn that those hardy souls in the front line for whom I have considerable respect and admiration, are being deprived of the fundamental training and facilities needed to improve outcomes. </p>
<p>No doubt the apparent focus on numbers is to enable those responsible to issue gushing reports of achievement through the simplistic process of counting the numbers of needles issued, rather than positive outcomes of how those who use the facilities might be engaged in recovery. </p>
<p>A case of &#8216;never mind the quality, feel the width&#8217;. </p>
<p>If we add to that the seeming disregard of the danger to the public caused by discarded needles, then harm reduction as it is being practised in this country is creating more problems than it is resolving. </p>
<p>It seems to me that those people who sit in their &#8216;ivory towers&#8217; dreaming up &#8216;harm reduction&#8217; solutions have failed to realise that addiction is not confined to office hours and that when the addicted are craving for a fix, the lack of a clean needle will not prevent them from using. </p>
<p>Now exactly what is it that Mr Barnes of DrugScope feels we have reason to be &#8216;justly proud of&#8217;? </p>
<p>Is it the number of needles issued? </p>
<p>The injury to children and others arising from discarded needles? </p>
<p>The lack of training and supervision and hygiene facilities? Or the escalation, in avoidable deaths and disease? </p>
<p>The one thing I do agree with Mr. Barnes on is that more, much more is needed to reduce both drug related deaths and disease, and the most realistic way of achieving that is through abstinence focused recovery. </p>
<p>What Mr Barnes seems unable to grasp is that there is a world of difference between abstinence and recovery. Nor does he seem willing to acknowledge that the outcome of addiction is always abstinence. The latter is not an option as Mr Barnes appears to be suggesting. It is achieved either through premature death, a reality which is already occurring, or abstinence focused treatment followed by on going after care; realities that neither Mr. Barnes or the NTA seem willing or able, to confront.</p>
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		<title>The grim reality of 574 addicts&#8217; wasted lives</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/the-grim-reality-of-574-addicts-wasted-lives/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/the-grim-reality-of-574-addicts-wasted-lives/#comments</comments>
		<pubDate>Mon, 14 Nov 2011 11:56:07 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Effects of Drugs (Drug Politics)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7990</guid>
		<description><![CDATA[EACH year the Scottish Drug Misuse Database releases statistics laying bare the grim reality of drug addiction in Scotland. For a few days politician show angst at the tragedy that lies behind the statistics but somehow attention moves on as if this problem will resolve itself. It is expected areas of high unemployment and poverty [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;">EACH year the Scottish Drug Misuse Database releases statistics laying bare the grim reality of drug addiction in Scotland.  For a few days politician show angst at the tragedy that lies behind the statistics but somehow attention moves on as if this problem will resolve itself.</p>
<p>It is expected areas of high unemployment and poverty will feature prominently in the SDMD and yesterday&#8217;s figures offer little change. Glasgow, Dundee, Inverclyde and West Dunbartonshire all feature as areas showing significant levels of problematic drug abuse, though in truth all of Scotland is affected. </p>
<p>What continues to shock, however, is the numbers of young people under 15 years of age who present as problematic drug users and this year, as the figure records more than 100, that shock does not lessen. </p>
<p>Their first involvement is likely to occur as early as primary school but most often in first or second year secondary, their drug use developing usually from a habit of the illicit drinking of alcohol with school friends. Accessing of drugs builds from that background of irresponsible risk taking in public areas such as parks, isolated school play areas and the likes. </p>
<p>From my experience and talking to young people in prisons, it seems to me likely that school absenteeism arising from heavy drinking and the abuse of drugs (usually cannabis) created for these youngsters a self-imposed understanding of exclusion and thereafter educational failure that ensured that any chances they may have had of early success is denied. </p>
<p>Opportunities for gainful employment were also denied. It is in these circumstances that many turned to heroin, diazepam and cocaine – drugs identified in the most recent statistics as the source of much of the problematic drug misuse recorded. A spiralling downturn in life chances, an increased likelihood of arrest and incarceration and real possibility of drugs-related death beckons. </p>
<p>The latest figures reflect a 131 per cent increase in drugs-related deaths over the ten-year period to 2008 giving us a new yearly total of 574 deaths. 574 tragedies. </p>
<p>It is not the writing of new drugs strategies that will bring about a change in this situation. It&#8217;s government leadership to ensure that enforcement, health, education and prisons all work with the voluntary sector towards the sole outcome of reducing problematic drug abuse.</p>
<p><em>Source: http://news.scotsman.com  31st March 2010<br />
</em></p>
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		<title>Prisons keeping inmates dependent on drugs, says new report</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/prisons-keeping-inmates-dependent-on-drugs-says-new-report/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/prisons-keeping-inmates-dependent-on-drugs-says-new-report/#comments</comments>
		<pubDate>Mon, 14 Nov 2011 11:54:28 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Legal Sector (Drug Politics)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7988</guid>
		<description><![CDATA[Almost 30,000 prisoners are being kept dependent on drugs by the prison service rather than being put through detox programmes, according to a new report. Methadone, along with similar drugs, is being prescribed too easily thanks to risk-averse clinical guidelines and inexperienced prescribers, concludes the Policy Exchange report, to be released on Monday. &#8220;Perversely, the [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;">Almost 30,000 prisoners are being kept dependent on drugs by the prison service rather than being put through detox programmes, according to a new report.<br />
Methadone, along with similar drugs, is being prescribed too easily thanks to risk-averse clinical guidelines and inexperienced prescribers, concludes the Policy Exchange report, to be released on Monday.<br />
&#8220;Perversely, the massive increase in opiate substitute medication has created a new kind of trade for drugs in prisons, as methadone and buprenorphine are readily traded among inmates,&#8221; said Max Chambers, author of the report, Coming Clean, Combating Drug Misuse in Prisons.<br />
The report criticises clinical guidelines for not taking into account the length of sentence a prisoner is serving when prescribing treatment for drug addiction.<br />
&#8220;Maintenance treatment, which is when a stable dose is prescribed often continued indefinitely, should only be given to prisoners serving 13 weeks or less and who don&#8217;t have time to complete a detoxification programme,&#8221; said Chamber.<br />
Under current practices, however, every prisoner who has been receiving methadone in the community will have their drug habit maintained in prison, regardless of the length of their sentence.<br />
Almost 20,000 maintenance prescriptions were made in 2008 to 2009. By 2011, when the Integrated Drug Treatment System is rolled out to all prisons in England and Wales, an additional 8,788 prisoners a year will be receiving methadone maintenance treatment.<br />
The report also cites research showing that around £100m of drugs are smuggled into prisons each year. The majority of drug-dealing in prison involves the collusion of about 1,000 corrupt members of staff – equating to seven prison officers per prison. &#8220;They are able to smuggle drugs due to lax security arrangements and, given the inflated value of drugs in prison, are able to make substantial profits without fear of detection,&#8221; said Chambers. &#8220;A prison officer bringing a gram of heroin into prison every week – about the size of two paracetamol tablets – could expect to more than double his basic salary.&#8221;<br />
Chambers cites evidence that accusations of corruption by prison officers are not routinely investigated by the Serious Organised Crime Agency or the Prison Service. &#8220;Information on the number of officers accused, charged, prosecuted or convicted of smuggling drugsor other contraband is apparently not collected at all by central government,&#8221; he said.<br />
The report reveals that the number of prisoners using drugs is hugely underestimated. Mandatory drug testing figures indicate 7.7% of prisoners are using drugs but in a survey of prisoners conducted for the new report, the figure was found to be 35%, with 16% using drugs at least once a week – equivalent to about 14,000 prisoners.<br />
Harry Fletcher, assistant general secretary of probation union Napo, said officers who smuggled drugs into jail routinely avoided detection. &#8220;It&#8217;s a serious problem but the government doesn&#8217;t keep statistics on how many staff are caught, which is extraordinary,&#8221; he said.<br />
Fletcher said there were more than 6,000 prison officers convicted of disciplinary offences over the past four years, with 19 of them currently serving sentences. &#8220;Because there is no data on the extent of the problem we can&#8217;t devise solutions,&#8221; he said.</p>
<p><em>Source: www.guardian.co.uk   28th May 2010<br />
</em><br />
</span></p>
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		<title>Dawn is almost here,but hope seems far away.</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/dawn-is-almost-herebut-hope-seems-far-away/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/dawn-is-almost-herebut-hope-seems-far-away/#comments</comments>
		<pubDate>Sun, 13 Nov 2011 23:53:31 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Uncategorized]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7983</guid>
		<description><![CDATA[Dear  Friend, They wait to welcome their child into the world. They wait in cars after the team practice. They wait in line to buy their family’s dinner. They wait for texts to pick up their child from birthday parties, sleepovers and play dates. Others wait for thick college acceptance envelopes. But imagine this: You are one of the millions [...]]]></description>
			<content:encoded><![CDATA[<p>Dear  Friend,</p>
<p>They wait to welcome their child into the world. They wait in cars after the team practice. They wait in line to buy their family’s dinner. They wait for texts to pick up their child from birthday parties, sleepovers and play dates. Others wait for thick college acceptance envelopes. But imagine this: You are one of the millions of parents with a teen or young adult abusing drugs or alcohol. You? You wait for a phone call. You wonder if your child will come home. You worry she’s hurt.</p>
<p>Like so many others, you don&#8217;t know where to start to find the answers you need, let alone how you will confront your child, if she needs help, or if there are others like you, parents who have stood in your shoes, paced beside doors or waited for a call. Having a child with an alcohol or drug problem can be emotionally taxing and financially devastating for families.</p>
<p>Suddenly, a lifeline.</p>
<p>You are searching the Internet for help and find <a href="http://www.drugfree.org/">The Partnership at Drugfree.org</a>. The website, created with and for parents, allows you to connect with others, tap into expert advice and find support as you find help for your child. You find insight and tools you can use. You find a community who understands. You find hope. That’s what The Partnership at Drugfree.org exists to do – be a partner to parents all along their journey of parenting a teen, whether that’s prevention, intervention or treatment.</p>
<p>The<a href="http://www.drugfree.org/"> </a><a href="http://www.drugfree.org/">The Partnership at Drugfree.org</a> is there, a steady, supportive hand, offering services to help parents and caring adults. Not a government agency, but a nonprofit made up of partners in science, parenting and communications, and one that relies on the generosity of individuals.</p>
<p>Right now&#8230;</p>
<ul>
<li>We are building support for <a href="http://www.drugfree.org/give-get-involved/you-are-not-alone"><strong>You Are Not Alone</strong></a>&#8230; a campaign dedicated to letting families of teens and young adults who are struggling with addiction know that they are not alone. By enlisting and uniting the millions who have been affected by addiction, we are removing barriers to seeking treatment and creating a new dialogue around addiction. </li>
<li>We are growing the staff and services of our Parents Toll-Free Hotline&#8230; <a href="http://timetogethelp.drugfree.org/parents-toll-free-helpline-0"><strong>1-855-DRUGFREE</strong></a>… a new, nationwide support service that offers assistance to parents and other primary caregivers of children who want to talk to someone about their child’s drug use and drinking. Our trained and caring parent specialists will help parents plan a course of action for teens who are struggling with substance abuse and lead them to resources or treatment facilities in their area.</li>
<li>We have <a href="http://timetogethelp.drugfree.org/"><strong>downloadable e-books</strong></a> <strong>and</strong> <a href="http://www.timetotalk.org/ParentTalkKit/"><strong>kits</strong></a> available&#8230; which equip parents with conversation starters and step-by-step direction to talk to their kids or take action if they think or know their child is using. Kids who learn a lot about the risks of drugs from their parents are up to <strong>50% less likely to use</strong>.</li>
</ul>
<p>Meeting these specific needs of parents, in conjunction with the everyday services we provide to families, will take a huge effort, cost money, and stretch our resources to the limit.</p>
<p>Stephen J. Pasierb, President &amp; CEO</p>
<p>Source: www.<a href="http://www.drugfree.org/">drugfree.org</a> to learn more</p>
<p align="center"> </p>
<p>&nbsp;</p>
<p align="center"> </p>
<p>&nbsp;</p>
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		<title>Parents: Know warning signs of drug abuse</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/parents-know-warning-signs-of-drug-abuse-3/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/parents-know-warning-signs-of-drug-abuse-3/#comments</comments>
		<pubDate>Sun, 13 Nov 2011 23:45:01 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Cannabis]]></category>
		<category><![CDATA[Parents]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7980</guid>
		<description><![CDATA[ Q: How can I tell if my child has been using marijuana? A: There are some signs you might be able to see. If someone is high on marijuana, he or she might:  Seem dizzy and have trouble walking; Seem silly and giggly for no reason; Save very red, bloodshot eyes; and Have a hard [...]]]></description>
			<content:encoded><![CDATA[<p> <strong>Q: </strong>How can I tell if my child has been using marijuana?</p>
<p><strong>A:</strong> There are some signs you might be able to see. If someone is high on marijuana, he or she might:</p>
<p> Seem dizzy and have trouble walking;</p>
<ul>
<li>Seem silly and giggly for no reason;</li>
<li>Save very red, bloodshot eyes; and</li>
<li>Have a hard time remembering things that just happened.</li>
</ul>
<p> When the early effects fade, the user can become very sleepy.</p>
<p> Parents should be aware of changes in their child&#8217;s behavior, although this may be difficult with teens. Parents should look for withdrawal, depression, fatigue, carelessness with grooming, hostility and deteriorating relationships with family members and friends.</p>
<p> In addition, changes in academic performance, increased absenteeism or truancy, lost interest in sports or other favourite activities, and changes in eating or sleeping habits could be related to drug use. However, these signs may also indicate problems other than using drugs.</p>
<p>&nbsp;</p>
<p>In addition, parents should be aware of:</p>
<p> Signs of drugs and drug paraphernalia, including pipes and rolling papers;</p>
<ul>
<li>Odour on clothes and in the bedroom;</li>
<li>Use of incense and other deodorizers;</li>
<li>Use of eye drops; and</li>
<li>Clothing, posters, jewellery, etc., promoting drug use.</li>
</ul>
<p><em> </em><em>Source: The National Institute on Drug Abuse  2010</em></p>
<p>&nbsp;</p>
<p>&nbsp;</p>
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		<title>Characteristics Of Effective Prevention</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/characteristics-of-effective-prevention/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/characteristics-of-effective-prevention/#comments</comments>
		<pubDate>Sun, 13 Nov 2011 23:38:56 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Education Sector (Papers)]]></category>
		<category><![CDATA[Papers]]></category>
		<category><![CDATA[Prevention (Papers)]]></category>
		<category><![CDATA[Social Affairs (Papers)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7976</guid>
		<description><![CDATA[Written by Bonnie Benard, NIDA, USA.(Originally in training manuals for Project Snowball, Illinois Teen Institutes, 1987) Published in Britainin ‘Drug Prevention – Just say Now’ by Peter Stoker, pub. David Fulton Publishers,London, 1992. Programme comprehensiveness/intensity A.        Multiplicity: the causes of drug/alcohol abuse are multiple: personality, environmental, behavioural (Kandel, Logan, 1984; Hawkins et al, 1951).  Programmes [...]]]></description>
			<content:encoded><![CDATA[<p><strong>Written by Bonnie Benard</strong>, NIDA, USA.(Originally in training manuals for Project Snowball, Illinois Teen Institutes, 1987)</p>
<p>Published in Britainin ‘Drug Prevention – Just say Now’ by Peter Stoker, pub. David Fulton Publishers,London, 1992.</p>
<h1>Programme comprehensiveness/intensity</h1>
<p>A.        <strong>Multiplicity:</strong> the causes of drug/alcohol abuse are multiple: personality, environmental, behavioural (Kandel, Logan, 1984; Hawkins et al, 1951).  Programmes tackling only one area usually fail.  You should target multiple systems (youth, families, schools, community, workplace, media, etc).  Also use multiple strategies (information, lifestyles, positive alternatives, community policies) (Botvin, 1982).</p>
<p>B.         <strong>Target whole community.</strong>  School-based programmes achieve less than community-based approaches.</p>
<p>C.         <strong>Target all youth for prevention</strong> &#8211; not just “high risk”.  Adolescence is seen to be a high-risk time for all youth in terms of health-compromising behaviour.  Labelling “high risk” youth can provoke stigmatisation and lead to self-fulfilling prophecies.  There is however an argument for defining “high risk” communities where an additional resource over and above the general prevention effort could be justified.</p>
<p>D.        <strong>Build drug prevention into general health promotion.</strong>  Drug abuse has been found by several (Lofquist, 1993) to be part of a complex pattern of interrelated factors – e.g. delinquency, truancy, school failure, precocious sexuality, which share common antecedents.</p>
<p>E.         <strong>Start at an early age and keep going!</strong>  Even in infancy there are influences in later behaviour.  Developmental difficulties by age 3 are difficult to overcome (Burton, White).  Here, it is of course relevant to mention Trevor Williams, Noreen Wetton and Alysoun Moon of the Southampton University team who have demonstrated so graphically in their “Jugs and Herrings” research papers that primary age children are not blissfully ignorant of drugs and alcohol.  Prevention programmes starting from what children actually know are essential.  Many secondary schools still seem to regard years 11 and 12 as the age at which discussion of drugs (or indeed sexuality) should be facilitated.  Don’t wait until the horse has run away before you lock the stable doors!</p>
<p>F.         <strong>Adequate quantity.</strong>  ‘One-shot prevention efforts do not work” (Kumpfer, 1988) there must be a substantial number of interventions, each of a substantial duration.  Project D.A.R.E. (Drug Abuse Resistance Education) initiated by Los Angeles Police and now in several countries, delivers no less than seventeen one-hour lessons to any given year and this is only part of the school programme.</p>
<p>G.        <strong>Integrate family/classroom/school/community life.</strong>  This is easier to say than do, but where it has happened results have been enhanced.</p>
<p>H.        <strong>Supportive environment, empowerment.</strong>  Where young people are encouraged to participate and take responsibility their behavioural outcomes are improved.  InBritain now peer-education methods which have been proven elsewhere have been applied to good effect.</p>
<h1>Programme strategies</h1>
<p>J.          <strong>‘KAB’ -</strong> <strong>K</strong><strong>nowledge/Attitudes/Behaviour</strong>.  Address these as a set, i.e. each must be directly tackled rather than assuming one will flow from another.  The behavioural component is in part addressed by social skills development, but also supported by positive reinforcement activities – drug free social/sporting events, commendations (preferably with something for everyone), media coverage, etc.  Research suggests that social learning theory (Bandura, 1977) produces some of the most profound improvements.</p>
<p>K.         <strong>Drug specific curriculum.</strong>  Affective education programmes that had not specifically covered drugs (including alcohol) were found unsuccessful, even though they addressed knowledge, attitudes and lifeskills.</p>
<p>L.         <strong>Gateway drugs.</strong>  So-called because people now using heavy-end drugs almost always started on these.  Gateway drugs can be tobacco, alcohol and cannabis or, these days inBritain, even heroin!  Concentration on prevention of these is therefore likely to prevent all substances.  British research by PaT (Parents against Tobacco, 1990), showed that of youth who smoked tobacco 50% had also tried an illegal drug compared with only 2% of those who did not smoke tobacco.  It should be particularly noted that cannabis is far from harmless; physical, mental and social damage is now being increasingly accepted as a reality.</p>
<p>M.        <strong>Salient material.</strong>  Whatever is used needs to identify with the audience, including:</p>
<p>•          ethnic/cultural sensitivity</p>
<p>•          appeal to youth’s interests</p>
<p>•          short term outcomes to be emphasised as important to youth as well as long term</p>
<p>•          appropriate language, readability</p>
<p>•          appealing graphics</p>
<p>•          appropriate to real age/reading age – a key factor</p>
<p>In a survey of 3, 700, 000 young American children, 25% of 9 year olds felt “some” to “a lot” of peer pressure to try drugs or alcohol (Weekly Reader, 1987).</p>
<p>N.        <strong>Alternatives.</strong>  Activities have to be plausible, be more highly valued than the health-compromising behaviour.  Too often these alternatives are poorly thought through. ( ‘Ping-pong = prevention’? No!)</p>
<p>P.         <strong>Lifeskills.</strong>      Development of these will be of wider benefit than drug prevention.  Included will be</p>
<p>communication, problem solving, decision-making, critical thinking, assertiveness, peer pressure reversal, peer selection, low-risk choice making, self-improvement, stress reduction and consumer awareness (Botvin, 1985).</p>
<p>Choosing your friends has been found in some research to be more effective than resisting the peer pressure of said friends.  Consumer awareness is a “companion” to resisting peer pressure, i.e. resisting media pressure.</p>
<p>Q.        <strong>Training prevention workers.</strong>  For the school setting the greater emphasis on experiential and interactive work requires teacher training to extend into youth work skills.  Community development skills are valuable in taking school initiatives into the community.  Imported “prestige” role models are all very well, but good results have been achieved with parents, peers, teachers, or outside agency workers.</p>
<p>R.         <strong>Community norms.</strong>  Consistency of policies throughout schools, families and communities can greatly enhance impact.</p>
<p>S.         <strong>Alcohol norms.</strong>  Because of its dual status as a beverage and as a culturally accepted drug, alcohol is problematic for prevention.  However, heart disease and tobacco prevention programmes have shown that societal norms can be changed.</p>
<p>T.         <strong>Improve schooling!</strong>  Listed here as a target because of its important correlation with healthy lifestyle.  Within the current British economic and academic climate one realistic hope may lie with co-operative learning, see the ‘Tribes’ programme, for example.</p>
<p>U.        <strong>Change society.</strong>  Don’t just stop with improving schools; add your voices to pressure for improvement in employment, housing, recreation and self-development; it is naïve to suppose that prevention can take place in a political vacuum.  Jessor recognises that failing to acknowledge the need for macro-environmental improvement while at the same time placing the responsibility for health solely with the individual is tantamount to “blaming the victim”.</p>
<h1>The planning process</h1>
<p>V.         <strong>Design, implementation, evaluation.</strong>  Evaluations have generally concentrated on outcomes rather than the quality of design.  However, implementation is as much dependent on engaging all sectors of the community (be it a school, a workplace, or a town) as it is on quality of design.  Evaluation should therefore measure process as well as outcome.</p>
<p>W.        <strong>Goal-setting.</strong>  Unrealistic or immeasurable goals help no-one.  It is important to set not only long-term outcome goals (for prevention is long-term) but also “process goals” such as increased involvement of parents and community, academic success, increased student-teacher interaction, and so on.</p>
<p>X.         <strong>Evaluation and amendment.</strong>  Prevention workers have been criticized for giving too little attention to this area, the crushing shortage of funds has much to do with it (inAmerica the ratio of funding between interdiction-policy and prevention is about 200:1).  This lack of emphasis on evaluation has been the Achilles heel that pro-drug campaigners have gleefully attacked.  Effective evaluations have been those including longitudinal design, multiple measures of process as well as outcome (Tobler, 1986), and cost-benefit analysis (CBA).  CBA is perhaps the greatest marketing tool prevention has; where CBA has been applied substantial cost effectiveness has been demonstrated.</p>
<table width="100%" cellspacing="0" cellpadding="0">
<tbody>
<tr>
<td>
<div>
<h2 align="center">National Drug PreventionAlliance</h2>
<p align="center">PO Box 594, Slough  SL1 1AA   J  <em>Tel / Fax: </em>+44 (0)1753 677917</p>
<p align="center"><em>E-mail: </em>NDPA@drugprevent.org.uk   J  <em>Internet: </em>www.drugprevent.org.uk</p>
</div>
</td>
</tr>
</tbody>
</table>
<p>&nbsp;</p>
<p>&nbsp;</p>
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		<title>Number of Prescription Painkiller Deaths More Than Tripled in Last 10 Years</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/number-of-prescription-painkiller-deaths-more-than-tripled-in-last-10-years/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/number-of-prescription-painkiller-deaths-more-than-tripled-in-last-10-years/#comments</comments>
		<pubDate>Sat, 05 Nov 2011 21:05:17 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Addiction]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7972</guid>
		<description><![CDATA[The number of Americans who died from overdoses of prescription painkillers more than tripled in the past decade, according to the Centers for Disease Control and Prevention (CDC). More people now die from painkillers than from heroin and cocaine combined. An estimated 14,800 people died in the United States from painkiller overdoses in 2008, a [...]]]></description>
			<content:encoded><![CDATA[<p>The number of Americans who died from overdoses of prescription painkillers more than tripled in the past decade, according to the Centers for Disease Control and Prevention (CDC). More people now die from painkillers than from heroin and cocaine combined.</p>
<p>An estimated 14,800 people died in the United States from painkiller overdoses in 2008, a more than threefold jump from the 4,000 deaths recorded in 1999, the CDC said in a new report. Prescription and illegal drugs caused 36,450 deaths in 2008, compared with 39,973 deaths from motor vehicle crashes, according to the Associated Press.</p>
<p>The CDC said painkiller abuse and deaths are rising because the drugs are easier than ever to obtain. They cited the growth of “pill mills,” clinics that prescribe opioids without first conducting medical exams, and “doctor shopping,” or receiving multiple prescriptions from different doctors.   According to the CDC, enough painkillers were prescribed in 2010 to medicate every American adult around the clock for a month. “Right now, the system is awash in opioids—dangerous drugs that got people hooked and keep them hooked,” said CDC Director Thomas Frieden.</p>
<p>“Prescription drug abuse is a silent epidemic that is stealing thousands of lives and tearing apart communities and families across America,” Gil Kerlikowske, Director of National Drug Control Policy, said in a CDC news release. He noted health care providers and patients should be educated on the risks of prescription painkillers. “Parents and grandparents should properly dispose of any unneeded or expired medications from the home and to talk to their kids about the misuse and abuse of prescription drugs,” he noted.</p>
<p>Source: ww.drugfree.org.  2nd Nov.2011</p>
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		<title>Nicotine Acts as “Gateway” Drug to Cocaine, Study in Mice Finds</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/nicotine-acts-as-%e2%80%9cgateway%e2%80%9d-drug-to-cocaine-study-in-mice-finds/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/nicotine-acts-as-%e2%80%9cgateway%e2%80%9d-drug-to-cocaine-study-in-mice-finds/#comments</comments>
		<pubDate>Sat, 05 Nov 2011 20:56:51 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Cocaine]]></category>
		<category><![CDATA[Nicotine]]></category>
		<category><![CDATA[Youth]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7968</guid>
		<description><![CDATA[Nicotine appears to be a “gateway” drug that primes the brain to be susceptible to cocaine, according to a new study in mice.  The researchers say if further studies show the findings apply to humans, a decrease in smoking rates in young people would be expected to lead to a decrease in cocaine addiction, the [...]]]></description>
			<content:encoded><![CDATA[<p>Nicotine appears to be a “gateway” drug that primes the brain to be susceptible to cocaine, according to a new study in mice.  The researchers say if further studies show the findings apply to humans, a decrease in smoking rates in young people would be expected to lead to a decrease in cocaine addiction, the Los Angeles Times reports.</p>
<p>The study found mice exposed to nicotine in drinking water for at least seven days showed an increased response to cocaine. The researchers also looked at data on cocaine use among a group of high school students, and found 81 percent of those who started using cocaine did so in a month when they were smoking tobacco.</p>
<p>The findings appear in the journal Science Translational Medicine.<br />
Previous studies have shown that most illegal drug users report using tobacco products or alcohol before they started illicit drug use, according to a news release by the National Institute on Drug Abuse, which funded the study. Until now, studies have not shown a biological mechanism through which exposure to nicotine increases vulnerability to illegal drug use, the release notes.</p>
<p>“Now that we have a mouse model of the actions of nicotine as a gateway drug this will allow us to explore the molecular mechanisms by which alcohol and marijuana might act as gateway drugs,” lead author Eric Kandel, MD, of Columbia University Medical Center, said in the release. “In particular, we would be interested in knowing if there is a single, common mechanism for all gateway drugs or if each drug utilizes a distinct mechanism.”</p>
<p>Source:   <a href="http://www.drugfree.org/">www.drugfree.org</a>.  4th Nov.</p>
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		<title>Record high: Gallup poll shows FIFTY per cent of Americans favour legalising marijuana</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/record-high-gallup-poll-shows-fifty-per-cent-of-americans-favour-legalising-marijuana/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/record-high-gallup-poll-shows-fifty-per-cent-of-americans-favour-legalising-marijuana/#comments</comments>
		<pubDate>Sat, 05 Nov 2011 20:54:16 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Cannabis]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7965</guid>
		<description><![CDATA[The headline below from the Daily Mail on the 18th October 2011, suggests that 50% of the population of America favour legalizing marijuana.   However  Jose Paulo Carneiro, a statistics expert from Brazil,  writes a critique of this survey and shows that the results issued by Gallup Poll are not what they seem to be suggesting. [...]]]></description>
			<content:encoded><![CDATA[<p>The headline below from the Daily Mail on the 18th October 2011, suggests that 50% of the population of America favour legalizing marijuana.   However  Jose Paulo Carneiro, a statistics expert from Brazil,  writes a critique of this survey and shows that the results issued by Gallup Poll are not what they seem to be suggesting.<br />
The saying ‘Lies, Damn Lies and Statistics’ comes to mind.    NDPA<br />
*  *  *  *  *  *<br />
Record high: Gallup poll shows FIFTY per cent of Americans favour legalising marijuana    -    18th October 2011<br />
• Up from 46 per cent last year<br />
• Liberals and those 18 to 29 most in favour<br />
• Americans 65 and older most oppose<br />
Read more: <a href="http://www.dailymail.co.uk/news/article-2050348/Legalisation-marijuana-50-Americans-favour.html#ixzz1boJx8Vwj">http://www.dailymail.co.uk/news/article-2050348/Legalisation-marijuana-50-Americans-favour.html#ixzz1boJx8Vwj</a><br />
*  *  *  *  *  *<br />
 <br />
&#8220;First of all, what is the methodology of this survey? What is the universe? Of course, it cannot be all the Americans.   Suppose it is the American population between 15 and 64 years (approximately 205.7 million).</p>
<p>If the sample were a simple random sample (that is, all the individuals have the same probability of being selected), the fact that the sample size is a tiny proportion of the population would not matter, because in the formula for the sample standard deviation (let us call it s) of a proportion to be estimated, the size N of the universe only appears in a factor, which, for a sample size of 1,005, equals 0.999995, which is practically 1. <br />
 <br />
Then, the maximum value for s, which occurs precisely for the 50% proportion and is independent of N, reduces to 1.6%. This means (supposing, as usual, the normality of the sampling distribution) that there is a probability of 95% that a real proportion of 50% will appear in the sample between 46.8% and 53.2% (this is the meaning of the phrase “the survey error is 3.2%”), which is a very acceptable value.<br />
 <br />
The problem is not in the sample size. The problem is that a telephone survey is not a simple sample survey, because not all individuals have the same chance of being selected. If you don’t have a telephone number, your probability of being selected is zero. If you have three telephone numbers and your neighbor has only one, your probability of being selected is three times his. Moreover, even inside a specific household, the probabilities are different. In certain households (mine, for instance), the probability that the husband answers the phone is very small compared with the probability that the wife does it. And, what is worse: the sample is biased, because there may be – and usually there is – a specific profile of people who answer, opposed to that of people who don’t answer the call.</p>
<p>In summary, it is very surprising that an Institute so renowned as Gallup, in a country so developed in matters of survey and research, makes a telephone survey and draws a conclusion about the opinion of “half of the Americans”.</p>
<p>Jose Paulo Carneiro, Expert in Statistics and Surveys, Rio de Janeiro, Brazil. Oct.2011</p>
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		<title>Medical Marijuana May Impair Thinking of MS Patients</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/medical-marijuana-may-impair-thinking-of-ms-patients/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/medical-marijuana-may-impair-thinking-of-ms-patients/#comments</comments>
		<pubDate>Sat, 05 Nov 2011 20:50:12 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Health]]></category>
		<category><![CDATA[Marijuana and Medicine]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7961</guid>
		<description><![CDATA[Feinstein says the marijuana users performed significantly worse than the non-users on tests measuring attention, speed of thinking, visual perception, and cognition related to planning and organizing.  Scores on one test measuring speed of processing information were about a third lower among marijuana users compared to non-users.  Thirty-two percent of non-users and 64% of users [...]]]></description>
			<content:encoded><![CDATA[<p>Feinstein says the marijuana users performed significantly worse than the non-users on tests measuring attention, speed of thinking, visual perception, and cognition related to planning and organizing.  Scores on one test measuring speed of processing information were about a third lower among marijuana users compared to non-users.  Thirty-two percent of non-users and 64% of users met the definition of globally cognitively impaired, meaning that they had measurable impairments in two or more aspects of intellectual functioning.<br />
 <br />
Neurologist Lily Jung Hensen, MD, of Seattle’s Swedish Neurosciences Institute, tells WebMD that the findings make a strong argument that the cognitive risks associated with marijuana use outweigh potential benefits for MS patients.<br />
 <br />
Source: <a href="http://www.cbs47.tv/webmd/ms/story/Medical-Marijuana-May-Impair-Thinking-of-MS/FmQ00ndFKkKhQ199RrPTDQ.cspx">http://www.cbs47.tv/webmd/ms/story/Medical-Marijuana-May-Impair-Thinking-of-MS/FmQ00ndFKkKhQ199RrPTDQ.cspx</a>  Oct.2011</p>
<p>&nbsp;</p>
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		<title>Impact On Lungs Of One Cannabis Joint Equal To Up To Five Cigarettes</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/impact-on-lungs-of-one-cannabis-joint-equal-to-up-to-five-cigarettes/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/impact-on-lungs-of-one-cannabis-joint-equal-to-up-to-five-cigarettes/#comments</comments>
		<pubDate>Sat, 05 Nov 2011 20:43:59 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Cannabis]]></category>
		<category><![CDATA[Health]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7954</guid>
		<description><![CDATA[A single cannabis joint has the same effect on the lungs as smoking up to five cigarettes in one go, indicates research published ahead of print in the journal Thorax. The researchers base their findings on 339 adults up to the age of 70, selected from an ongoing study of respiratory health, and categorised into [...]]]></description>
			<content:encoded><![CDATA[<p>A single cannabis joint has the same effect on the lungs as smoking up to five cigarettes in one go, indicates research published ahead of print in the journal Thorax.</p>
<p>The researchers base their findings on 339 adults up to the age of 70, selected from an ongoing study of respiratory health, and categorised into four different groups.</p>
<p>These comprised those who smoked only cannabis, equivalent to at least one joint a day for five years; those who smoked tobacco only, equivalent to a pack of cigarettes a day for at least a year; those who smoked both; and those who did not smoke either cannabis or tobacco.</p>
<p>All the participants had high definition x-ray scans (computed tomography) taken of their lungs and they took special breathing tests designed to assess how well their lungs worked.</p>
<p>They were also questioned about their smoking habits.<br />
Seventy five people smoked only cannabis, and 91 smoked both. Eighty one people did not smoke either, and 92 smoked only tobacco.</p>
<p>Combined smokers tended to use less tobacco, the findings showed.<br />
Cannabis smokers complained of wheeze, cough, chest tightness and phlegm. But emphysema, the progressive and crippling lung disease, was only seen in those who smoked tobacco, either alone or in combination.<br />
But cannabis still damaged the lungs and stopped them from working properly.</p>
<p>It diminished the numbers of small fine airways, which are important for transporting oxygen and waste products to and from the blood vessels effectively.<br />
And it damaged the large airways of the lung, blocking airflow, and forcing the lungs to work harder.<br />
The extent of this damage was directly related to the number of joints smoked, with higher consumption linked to greater incapacity.</p>
<p>The effect on the lungs of each joint was equivalent to smoking between 2.5 and five cigarettes in one go.<br />
The authors explain that the impact of cannabis is strongly associated with the way in which it is smoked. It is usually smoked without a filter, and at a higher temperature. Smokers tend to inhale more deeply and to hold their breath for longer.</p>
<p>Source:  Retrieved August 8, 2009, from <a href="http://www.sciencedaily.com/">http://www.sciencedaily.com</a></p>
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		<title>Marijuana Use and Motor Vehicle Crashes</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/marijuana-use-and-motor-vehicle-crashes/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/marijuana-use-and-motor-vehicle-crashes/#comments</comments>
		<pubDate>Sat, 05 Nov 2011 20:42:22 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Cannabis]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7956</guid>
		<description><![CDATA[Abstract Since 1996, 16 states and the District of Columbia in the United States have enacted legislation to decriminalize marijuana for medical use. Although marijuana is the most commonly detected non alcohol drug in drivers, its role in crash causation remains unsettled. To assess the association between marijuana use and crash risk, the authors performed [...]]]></description>
			<content:encoded><![CDATA[<p><strong>Abstract</strong></p>
<p>Since 1996, 16 states and the District of Columbia in the United States have enacted legislation to decriminalize marijuana for medical use. Although marijuana is the most commonly detected non alcohol drug in drivers, its role in crash causation remains unsettled. To assess the association between marijuana use and crash risk, the authors performed a meta-analysis of 9 epidemiologic studies published in English in the past 2 decades identified through a systematic search of bibliographic databases. Estimated odds ratios relating marijuana use to crash risk reported in these studies ranged from 0.85 to 7.16. Pooled analysis based on the random-effects model yielded a summary odds ratio of 2.66 (95% confidence interval: 2.07, 3.41). Analysis of individual studies indicated that the heightened risk of crash involvement associated with marijuana use persisted after adjustment for confounding variables and that the risk of crash involvement increased in a dose-response fashion with the concentration of 11-nor-9-carboxy-delta-9-tetrahydrocannabinol detected in the urine and the frequency of self-reported marijuana use. The results of this meta-analysis suggest that marijuana use by drivers is associated with a significantly increased risk of being involved in motor vehicle crashes.</p>
<p>Source:  Epidemiology Rev (2011) doi: 10.1093/epirev/mxr017</p>
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		<title>Impact of cannabis use on thalamic volume in people at familial high risk of schizophrenia</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/impact-of-cannabis-use-on-thalamic-volume-in-people-at-familial-high-risk-of-schizophrenia/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/impact-of-cannabis-use-on-thalamic-volume-in-people-at-familial-high-risk-of-schizophrenia/#comments</comments>
		<pubDate>Sat, 05 Nov 2011 20:33:28 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Brain and Behaviour]]></category>
		<category><![CDATA[Cannabis]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7951</guid>
		<description><![CDATA[1. Killian A. Welch, MD, MRCPsych  et al  Correspondence:: kwelch1@staffmail.ed.ac.uk Background No longitudinal study has yet examined the association between substance use and brain volume changes in a population at high risk of schizophrenia. Aims To examine the effects of cannabis on longitudinal thalamus and amygdala-hippocampal complex volumes within a population at high risk of schizophrenia. [...]]]></description>
			<content:encoded><![CDATA[<p><strong>1. Killian A. Welch, MD, MRCPsych  et al </strong></p>
<p>Correspondence:: <a href="mailto:kwelch1@staffmail.ed.ac.uk">kwelch1@staffmail.ed.ac.uk</a></p>
<p><strong>Background</strong><br />
No longitudinal study has yet examined the association between substance use and brain volume changes in a population at high risk of schizophrenia.</p>
<p><strong>Aims</strong><br />
To examine the effects of cannabis on longitudinal thalamus and amygdala-hippocampal complex volumes within a population at high risk of schizophrenia.</p>
<p><strong>Method</strong><br />
Magnetic resonance imaging scans were obtained from individuals at high genetic risk of schizophrenia at the point of entry to the Edinburgh High-Risk Study (EHRS) and approximately 2 years later. Differential thalamic and amygdala-hippocampal complex volume change in high-risk individuals exposed (n = 25) and not exposed (n = 32) to cannabis in the intervening period was investigated using repeated-measures analysis of variance.</p>
<p><strong>Results</strong><br />
Cannabis exposure was associated with bilateral thalamic volume loss. This effect was significant on the left (F = 4.47, P = 0.04) and highly significant on the right (F = 7.66, P = 0.008). These results remained significant when individuals using other illicit drugs were removed from the analysis.</p>
<p><strong>Conclusions</strong><br />
These are the first longitudinal data to demonstrate an association between thalamic volume loss and exposure to cannabis in currently unaffected people at familial high risk of developing schizophrenia. This observation may be important in understanding the link between cannabis exposure and the subsequent development of schizophrenia.</p>
<p> <br />
Source:  bjp.rcpsych.org   Sept.2011</p>
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		<title>Hepatitis C transmission via injecting drug use: look beyond needles and syringes</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/hepatitis-c-transmission-via-injecting-drug-use-look-beyond-needles-and-syringes/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/hepatitis-c-transmission-via-injecting-drug-use-look-beyond-needles-and-syringes/#comments</comments>
		<pubDate>Sat, 05 Nov 2011 20:25:35 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Health]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7946</guid>
		<description><![CDATA[Important insights into the continued spread of hepatitis C among injecting drug users are provided by two studies published in the online edition of the Journal of Infectious Diseases. An international team of investigators showed that infectious quantities of hepatitis C could survive on inanimate surfaces for up to seven days. However, the virus can [...]]]></description>
			<content:encoded><![CDATA[<p>Important insights into the continued spread of hepatitis C among injecting drug users are provided by two studies published in the online edition of the Journal of Infectious Diseases. An international team of investigators showed that infectious quantities of hepatitis C could survive on inanimate surfaces for up to seven days. However, the virus can be rendered inactive by commercially available disinfectants, or heating to a temperature of 65-70° for approximately 90 seconds.</p>
<p>In a separate study, French investigators detected the virus on 80% of alcohol swabs obtained from injecting drug users. They suggest that the swabs may be shared by users, risking the transmission of hepatitis C.</p>
<p>Holly Hagan of the New York University College of Nursing in an accompanying editorial stated: “The studies contribute new knowledge to our understanding of the mechanisms by which HCV [hepatitis C virus] may be transmitted among PWID [people who inject drugs] via injection-related materials.”</p>
<p>There are an estimated 130 million hepatitis C infections worldwide. Hepatitis C is a blood-borne infection and a major mode of transmission is injecting drug use. Needle and syringe exchange programmes have been introduced in many countries to control the epidemic. The have been highly effective at preventing new HIV infections, but hepatitis C transmissions still continue. This is possibly because viral load tends to be high in individuals with chronic hepatitis C infection, and even small quantities of contaminated blood are potentially infectious.</p>
<p>A team of investigators led by Juliane Doerrbecker wished to establish a clearer understanding of the survival of the virus, and the effectiveness of disinfectants and heat at rendering the virus non-infectious. Steel discs were contaminated with infectious quantities of hepatitis C which were then allowed to dry. Reassuringly, commercially available disinfectants were also shown to have “a high virucidal efficacy against HCV.”</p>
<p>Tests also showed that infectious quantities of hepatitis C of approximately 30 TCID50/ml could still be detected on inanimate surfaces up to seven days after contamination. However, the investigators emphasised that “all tested biocides were able to inactivate HCV infectivity to undetectable levels.”</p>
<p>The investigators then examined the effect of heat on the virus. Spoons and/or cookers are used to heat diluted heroin into solutions. The liquid is then drawn into a syringe, potentially contaminating the spoon if hepatitis C-infected blood is present in the syringe. The investigators therefore contaminated spoons with the virus, which were then heated to various temperatures using tea candles.</p>
<p>Infectivity started to decrease at temperatures of approximately 50°. Levels of the virus fell below the limit of detection when temperatures reached 67-70°. It generally took between 80 to 95 seconds for heating to produce small bubbles in the spoon.</p>
<p>“Reusing HCV contaminated cookers could lead to infection even if using sterile syringes,” comment the investigators. Holly Hagan emphasised that injecting drug users rarely heat spoons for more than 15 seconds.</p>
<p>In separate research, Dr Vincent Thibault and his colleagues collected drug-using paraphernalia from individuals known to be infected with hepatitis C. The used paraphernalia included syringes, filters and water cups, swabs for cleaning of skin before injecting and pads employed to stop bleeding after withdrawal of needles. A total of 160 pieces of equipment were collected.</p>
<p>The virus was detected on 44% of the pooled materials. A further 620 items used by individuals of unknown infection status were also obtained. Approximately 83% of the pools obtained from swabs had detectable hepatitis C. Moreover, viral load was highest (above 3 log10 iu/ml) within these swab pools. Hepatitis C was also commonly detected in syringes, but viral load tended to be at low levels (12 to 890 iu/ml). The investigators therefore believe that there is “a higher chance for PWID to be contaminated though sharing of a tainted spoon rather than a tainted syringe.”</p>
<p>They note that blood was often visible on swabs. The researchers therefore suggest that transmission of the virus could occur if swabs were being used inappropriately. “The chaotic and rushed atmosphere of the injection setting, where swab sharing and mixing could take place, is…an important factor that should be considered.”</p>
<p>Holly Hagan believes the two studies have important implications for hepatitis C prevention programmes. “Cleaning cookers or perhaps impregnating injection equipment with safe biocides may help reduce the incidence of new infections. Promoting safe swab use to emphasize avoidance of reuse seems a prudent measure.”</p>
<p><strong>Reference</strong><br />
Doerrbecker J et al. Inactivation and survival of hepatitis C virus on inanimate surfaces. J Infect Dis, online edition, doi: 101093/infdis/jir535 (click here for the abstract).</p>
<p>Thibault V et al. Hepatitis C transmission in injecting drug users: could swabs be the main culprit? J Infect Dis, online edition, doi: 101093/infdis/jir650 (click here for the abstract).</p>
<p>Source: www.aidsmap.com 4th Nov.2011</p>
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		<title>Drug advisers told no chance of decriminalising possession laws</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/drug-advisers-told-no-chance-of-decriminalising-possession-laws/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/drug-advisers-told-no-chance-of-decriminalising-possession-laws/#comments</comments>
		<pubDate>Sat, 05 Nov 2011 20:14:16 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Legal Sector (Drug Politics)]]></category>
		<category><![CDATA[Political Sector (Drug Politics)]]></category>
		<category><![CDATA[Stop Press]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7942</guid>
		<description><![CDATA[Theresa May, the Home Secretary, issued a humiliating rebuke to her drug advisers after they called for the possession of drugs to be decriminalised. The Home Office said there was no intention to give people a &#8220;green light&#8221; to use drugs because they &#8220;destroy lives and cause untold misery&#8221;. The Advisory Council on the Misuse [...]]]></description>
			<content:encoded><![CDATA[<p>Theresa May, the Home Secretary, issued a humiliating rebuke to her drug advisers after they called for the possession of drugs to be decriminalised.</p>
<p>The Home Office said there was no intention to give people a &#8220;green light&#8221; to use drugs because they &#8220;destroy lives and cause untold misery&#8221;. </p>
<p>The Advisory Council on the Misuse of Drugs (ACMD) risked a fresh row with the Home Office after suggesting those who possess any drug, including cocaine or heroin, for personal use should be taken out of the criminal justice system. </p>
<p>The Government issued a blunt statement insisting drug laws would not be liberalised and “decriminalisation is not the answer”.   It is the latest in a series of run-ins between Whitehall’s official drug advisory body and the Home Office. </p>
<p>In 2009, the then Home Secretary Alan Johnson, sacked the ACMD chairman Professor David Nutt after he openly criticised the Government’s stance on cannabis. He had also previously said taking Ecstasy was no more dangerous than riding a horse. </p>
<p>The ACMD called for a review on how those caught in possession of drugs are handled in a submission to the Sentencing Council, which is consulting on guidelines for courts on drug offences. </p>
<p>However, it is not in the remit of the Sentencing Council to consider what would effectively decriminalisation and the ACMD only included its comments in the final section asking for any further comments.     It wrote: “There is an opportunity to be more creative in dealing with those who have committed an offence by possession of drugs. </p>
<p>&#8220;For people found to be in possession of drugs (any) for personal use (and involved in no other criminal offences), they should not be processed through the criminal justice system but instead be diverted into drug education/awareness courses.&#8221; </p>
<p>The courses &#8220;would be the equivalent of the apparently successful &#8216;speed awareness&#8217; courses to which drivers can be referred as a diversion&#8221;, the council added.   It also suggested that those accused of possessing drugs could also face &#8220;more creative civil punishments&#8221;, such as the loss of a driving licence or passport. </p>
<p>A spokesman for the Home Office said: “We have no intention of liberalising our drugs laws. Drugs are illegal because they are harmful – they destroy lives and cause untold misery to families and communities.   &#8220;Those caught in the cycle of dependency must be supported to live drug free lives, but giving people a green light to possess drugs through decriminalisation is clearly not the answer.” </p>
<p>Source: www.telegraph.co.uk  18th Oct 2011</p>
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		<title>California Medical Association Not So Medical Says Drug Policy Experts</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/california-medical-association-not-so-medical-says-drug-policy-experts/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/california-medical-association-not-so-medical-says-drug-policy-experts/#comments</comments>
		<pubDate>Sat, 05 Nov 2011 20:10:17 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Drug Specifics (Drug Politics)]]></category>
		<category><![CDATA[Stop Press]]></category>
		<category><![CDATA[USA]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7939</guid>
		<description><![CDATA[The California Medical Association (CMA) took a major leap lacking science and common sense. With the issuance of a White Paper calling for the legalization of marijuana for medical and non-medial purposes, they have transitioned from a medical group into a lapdog of the drug legalization lobby. “I am thoroughly appalled by the CMA’s decision [...]]]></description>
			<content:encoded><![CDATA[<p>The California Medical Association (CMA) took a major leap lacking science and common sense. With the issuance of a White Paper calling for the legalization of marijuana for medical and non-medial purposes, they have transitioned from a medical group into a lapdog of the drug legalization lobby.<br />
“I am thoroughly appalled by the CMA’s decision to release this policy in an attempt to legalize a drug that we know causes so much harm to individuals and families,” said Eric Voth, M.D., F.A.C.P. and Chair of the Institute on Global Drug Policy.  “The CMA has managed to single-handedly make a mockery of modern medicine and the ethical practices of physicians. There is nothing scientific about this White Paper – it is total politics.” </p>
<p>The White Paper just released contains a number of incorrect statements. Contrary to what the paper states:<br />
•	According to the National Household Survey on Drug Abuse, the rate of current, past 30-day use of marijuana by Americans aged 12 and older in 1979 was 13.2%.  In 2008 that figure stood at 6.1%.  This 54% reduction over that 29-year period is a major public health triumph, not a failure! Now, we must not only push back against the drugs but, the advocates who seek to normalize and legalize them.<br />
•	The Netherlands reclassified high potency marijuana as a “hard drug” because of the harms that have occurred from the drug and moved to shut down hundreds of “coffee shops” that serve marijuana.  Their lenient policy caught up with them and they are moving back to more conservative actions.<br />
•	Portugal’s policy that decriminalized consumption and possession of illicit drugs in 2001 was a dismal failure.  The 2007 national drug survey showed an increase in life-time prevalence of drug use in the general population, especially regarding cannabis use and use of cocaine has nearly doubled. Cocaine seizures increased seven-fold between 2001and 2006 and murders increased 40%.  </p>
<p>“The CMA is dead wrong in asserting that the marijuana legalization movement is driven by the public. Instead it is driven by a group of well-financed legalization advocates. The ballot initiative to legalize pot was defeated in California and no other state has approved such an ill-advised policy, despite millions of dollars poured into this effort by ivory-tower elitists unaffected by the impact of drug use, like the rest of us. Even the issue of marijuana as medicine was rejected by two-thirds of the country,” stated Calvina Fay, Executive Director of Drug Free America Foundation.</p>
<p>“It is laudable that CMA supports more research and more education efforts to reduce marijuana use among children, adolescents, and young adults (although we believe it should include all adults). Ongoing research into potential medicines and cures is an important endeavor but, the solution should be to require marijuana to meet the standards of modern medicine, not by ballot initiatives or legislation and certainly not by legalizing it for recreational use” Fay concluded.<br />
Drug Free America Foundation, Inc. is dedicated to fighting drug use, drug addiction and drug trafficking and to promoting effective sound drug policies, education and prevention.</p>
<p>Source: www.dfaf.org October 17, 2011</p>
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		<title>Estimated expenditure on cannabis in Australia is twice that of wine</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/estimated-expenditure-on-cannabis-in-australia-is-twice-that-of-wine-2/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/estimated-expenditure-on-cannabis-in-australia-is-twice-that-of-wine-2/#comments</comments>
		<pubDate>Sat, 05 Nov 2011 20:03:04 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Cannabis]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7936</guid>
		<description><![CDATA[The price of cannabis has declined more than 40% (4.9% p.a.) in real terms during the 1990s, far greater than for most other agricultural products. Cannabis price may be declining because of increasing use of more efficient hydroponic cultivation techniques and also because decreasing law enforcement lowered the &#8216;full cost&#8217; of cannabis. The number of [...]]]></description>
			<content:encoded><![CDATA[<p>The price of cannabis has declined more than 40% (4.9% p.a.) in real terms during the 1990s, far greater than for most other agricultural products. Cannabis price may be declining because of increasing use of more efficient hydroponic cultivation techniques and also because decreasing law enforcement lowered the &#8216;full cost&#8217; of cannabis. The number of national arrests and prosecutions per 100,000 population fell by almost one third between 1996 and 2001. Penalties also became less severe. If cannabis price had been constant, consumption of beer would have been 2.4% higher, wine 4.9% higher, spirits 9.8% higher and cannabis 10.4% lower.</p>
<p>Comment: As the health, social and economic costs of alcohol are greater than for cannabis, decreasing cannabis prices may have reduced harm from legal drugs.</p>
<p>Source:  Clements KW.  The Australian Journal of Agricultural and Resource Economics. 2004. 48:2; 271-300</p>
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		<title>Drunk behaviour – a question of immunity</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/drunk-behaviour-%e2%80%93-a-question-of-immunity/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/drunk-behaviour-%e2%80%93-a-question-of-immunity/#comments</comments>
		<pubDate>Sat, 05 Nov 2011 20:00:24 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Alcohol]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7919</guid>
		<description><![CDATA[University of Adelaide researchers have found that immune cells in your brain may contribute to how you respond to alcohol. Lead researcher Dr Mark Hutchinson, ARC Research Fellow with the University’s School of Medical Sciences, said his team’s research provided new evidence that an immune response in the brain was involved in behavioural responses to [...]]]></description>
			<content:encoded><![CDATA[<p>University of Adelaide researchers have found that immune cells in your brain may contribute to how you respond to alcohol.   Lead researcher Dr Mark Hutchinson, ARC Research Fellow with the University’s School of Medical Sciences, said his team’s research provided new evidence that an immune response in the brain was involved in behavioural responses to alcohol. This immune response lies behind some of the well-known alcohol-related behavioural changes, such as difficulty controlling the muscles involved in walking and talking.</p>
<p>“It’s amazing to think that despite 10,000 years of using alcohol, and several decades of investigation into the way that alcohol affects the nerve cells in our brain, we are still trying to figure out exactly how it works,” says lead researcher Dr Mark Hutchinson from the University’s School of Medical Sciences.<br />
“Alcohol is consumed annually by two billion people world-wide with its abuse posing a significant health and social problem,” said Dr Hutchinson. “Over 76 million people are diagnosed with an alcohol abuse disorder.  “This work has significant implications for our understanding of the way alcohol affects us, as it is both an immunological and neuronal response. Such a shift in mindset has significant implications for identifying individuals who may have bad outcomes after consuming alcohol, and it could lead to a way of detecting people who are at greater risk of developing brain damage after long-term drinking.”</p>
<p>The research is published in the latest edition of the British Journal of Pharmacology by PhD student Yue Wu, supervisor Dr Hutchinson, and others. Laboratory mice were given a single shot of alcohol and the researchers studied the effect of blocking toll-like receptors, a particular element of the immune system, on the behavioural changes induced by alcohol.  The researchers studied the effects of blocking the receptors by drugs, and also the effects of giving alcohol to mice that had been genetically altered so that they were lacking the functions of the selected receptors.</p>
<p>“The results showed that blocking this part of the immune system, either with the drug or genetically, reduced the effects of alcohol,” Dr Hutchinson said. He believes similar treatment could work in humans.  “Medications targeting this specific receptor ‒ toll-like receptor 4 ‒ may prove beneficial in treating alcohol dependence and acute overdoses,” Dr Hutchinson said.</p>
<p>Source:  http://ahha.asn.au/news mark.hutchinson@adelaide.  29th Sept.2011</p>
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		<title>Do drug policies affect cannabis markets? A natural experiment in Switzerland, 2000–10.</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/do-drug-policies-affect-cannabis-markets-a-natural-experiment-in-switzerland-2000%e2%80%9310/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/do-drug-policies-affect-cannabis-markets-a-natural-experiment-in-switzerland-2000%e2%80%9310/#comments</comments>
		<pubDate>Sat, 05 Nov 2011 19:03:57 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Cannabis]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7914</guid>
		<description><![CDATA[Killias M., Isenring G.L., Gilliéron G. et al. European Journal of Criminology: 2011, 8(3), p. 171–186. Studies of a &#8216;natural experiment&#8217; in Switzerland in the 2000s suggested that the effective re-criminalisation of cannabis production and distribution did diminish availability and use of the drug. The results contradict other findings suggesting that national policies have little [...]]]></description>
			<content:encoded><![CDATA[<p>Killias M., Isenring G.L., Gilliéron G. et al.<br />
European Journal of Criminology: 2011, 8(3), p. 171–186.</p>
<p>Studies of a &#8216;natural experiment&#8217; in Switzerland in the 2000s suggested that the effective re-criminalisation of cannabis production and distribution did diminish availability and use of the drug. The results contradict other findings suggesting that national policies have little effect on cannabis use.</p>
<p><strong>Summary</strong><br />
A &#8216;natural experiment&#8217; in Switzerland in the 2000s revealed the impacts of changes in the enforcement of cannabis production and distribution laws. By 2001, in response to public sentiment Switzerland had already relaxed its enforcement of laws against the use and distribution of cannabis. At this time the government prepared reforms to enshrine this in law by officially tolerating the sale, possession and use of small amounts of cannabis (usually below 5g), and the production and sale of larger quantities as long as producers and retailers agreed to act under strict control by police and the Department of Agriculture. Though this change had yet to be implemented, in anticipation over the following years visible and quasi-official structures of production, distribution and sale emerged. Concerned over some of the consequences, in 2003 and again in 2004 the Swiss parliament rejected the proposed changes. Over the following months, police and prosecutors resumed former more repressive policies, especially in respect of production and distribution. As a result, shops and production centres were closed during 2005 and 2006. It was this reversal which offered the opportunity to evaluate the impact of tolerance of legal production and distribution versus lack of tolerance.</p>
<p><strong>Main findings</strong><br />
Early in 2004 shortly before most of their shops were closed, a survey of cannabis retailers suggested that competition between shops was quite stiff, particularly in respect of price. Nearly all felt they had to provide excellent products and service to keep their customers. Though many said they had never sold high strength and/or smokeable cannabis, this conflicted with the number of prosecutions for selling cannabis whose main active ingredient (THC) was above the legal limit.</p>
<p>In summer 2004 when many cannabis shops were still operating, two young men aged around 18 conducted &#8216;test purchase&#8217; operations at 50 shops. Of these, 29 sold cannabis without reservation and 26 did so regardless of the young men&#8217;s age. Usually, the fake clients asked for 5g or the quantity available for about 50 Swiss francs. The quantities actually sold generally varied between 3.8g and 6.5g and THC levels between 8% and 28%, averaging 16%. Overall, the study confirmed that minors easily obtained high-strength cannabis. Most samples contained THC close to the average of 16% and prices varied little around 11 Swiss francs per gram. In short, quality and prices were fairly well standardised.<br />
In 2009 when all known cannabis shops had closed, a second &#8216;test purchase&#8217; operation was conducted, but this time to test the availability of supplies on the now fully illicit market. Two young men walked through inner-city areas where police said cannabis was most available, looking for potential dealers. Over 15 afternoons they made 29 relevant contacts; during 27 they were able to obtain cannabis. All the sales took place in streets and parks. Usually the fake clients were able to spot a dealer in under 20 minutes. The quantity purchased varied far more than in 2004, ranging from 0.38 to nearly 13 grams. Equally inconsistent were prices, varying greatly between 8 and 200 francs per gram. A typical price was 28 francs. The THC content varied between 4% and 18% and averaged 12%, lower than in 2004. At every transaction, the fake clients asked whether the dealer might be able or willing to supply other substances. Only one said they could.</p>
<p>Compared to 2004, typical prices paid per gram had increased from 11 to 28 francs and the variability in price and quantity was much greater and THC content lower. From the relatively standardised market of 2004, by 2009 the price structure was, from the clients&#8217; point of view, relatively obscure and bore little relation to the origin or strength of the product.</p>
<p><strong>The authors&#8217; conclusions</strong><br />
The results of our studies suggest that legal policies can strongly affect production, supply, distribution and sale of cannabis. The switch from a liberal to a more repressive policy meant that large-scale agricultural was partly replaced by small-scale production on private premises, and sales moved back from shops to the streets. Formerly an export country, illegal import of cannabis in to Switzerland resumed, though probably not enough to compensate for lost local production. For users without links to home-based production networks, availability of cannabis may have decreased substantially, probably prompting decreased consumption. However, the market and its price structure became far more variable and obscure. Prices soared, possibly reflecting reduced supply and more marginal and criminal suppliers. Street sales favoured cheating because quantities cannot be accurately weighed and suppliers had little interest in repeat sales to unknown customers, feeling little need to gain their trust. On the other hand, and contrary to a widely held view, markets for cannabis and other substances seem to have remained separated.</p>
<p>Surveys in Switzerland and abroad suggest that policies making cannabis more easily available were followed by increasing rates of use, whereas Switzerland&#8217;s opposite policy after 2004 was associated with a drop in both the prevalence and frequency of cannabis use. Establishing to what extent policy changes caused changes in use is for the moment impossible, but data is consistent with the assumption that policies affect the availability and (indirectly) use of cannabis.</p>
<p>This draft entry is currently subject to consultation and correction by study authors.<br />
Last revised 06 October 2011<br />
Source : European Journal of Criminology: 2011, 8(3), p. 171–186.</p>
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		<title>One in four at risk of cannabis psychosis</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/one-in-four-at-risk-of-cannabis-psychosis-3/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/one-in-four-at-risk-of-cannabis-psychosis-3/#comments</comments>
		<pubDate>Sat, 05 Nov 2011 18:54:15 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Brain and Behaviour]]></category>
		<category><![CDATA[Cannabis]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7910</guid>
		<description><![CDATA[ONE in four people carries genes that increases vulnerability to psychotic illnesses if he or she smokes cannabis as a teenager, scientists have found. A common genetic profile that makes cannabis five times more likely to trigger schizophrenia and similar disorders has been identified, increasing pressure on the Government to reverse the drug’s reclassification from [...]]]></description>
			<content:encoded><![CDATA[<p>ONE in four people carries genes that increases vulnerability to psychotic illnesses if he or she smokes cannabis as a teenager, scientists have found.</p>
<p>A common genetic profile that makes cannabis five times more likely to trigger schizophrenia and similar disorders has been identified, increasing pressure on the Government to reverse the drug’s reclassification from Class B to Class C.</p>
<p>The increased risk applies to people who inherit variants of a gene named COMT who also smoked cannabis as teenagers. About a quarter of the population have this genetic make-up, and up to 15 per cent of the group are likely to develop psychotic conditions if exposed to the drug early in life.</p>
<p>Neither the drug nor the gene raises the risk of psychosis by itself.</p>
<p>The study, led by Avshalom Caspi and Terrie Moffitt, of the Institute of Psychiatry at King’s College London, offers the best explanation yet for the way that cannabis has a devastating psychiatric impact on some users but leaves most unharmed. Scientists had suspected that genetic factors were responsible for this divide, but a gene had not been pinpointed.</p>
<p>The findings, to be published in Biological Psychiatry, also reinforce a growing consensus that nature and nurture are not mutually exclusive forces but combine to affect behaviour and health. The King’s team has previously identified genes that raise the risk of depression or aggression, but only in conjunction with environmental influences.</p>
<p>Mental health campaigners said that the results vindicated their concerns about the decision last year to downgrade cannabis to a Class C drug, which means that possession is no longer an arrestable offence.</p>
<p>Marjorie Wallace, chief executive of the mental health charity Sane, said that it was becoming clear that cannabis placed millions of users at risk of lasting mental illness. About fifteen million Britons have tried cannabis, and between two million and five million are regular users, according to the Home Office British Crime Survey. The research suggests that a quarter could be at risk.</p>
<p>The evidence will be considered by a review of the drug’s classification announced last month by the Home Secretary. It may be possible to develop a test for genetic susceptibility to cannabis. “If we were able genetically to identify the vulnerable individuals in advance, we would be able to save thousands of minds, if not lives,” Ms Wallace said.</p>
<p>Dr Caspi, however, rejected the idea of screening based on the COMT gene. “Such a test would be wrong more often than it is right. Cannabis has many other adverse effects, especially on developing teenagers, on respiratory health and possibly on cognitive function. Effects may be pronounced among a genetically vulnerable group but that doesn’t mean we should encourage others not genetically vulnerable to use cannabis.”</p>
<p>The King’s team tracked 803 men and women born in Dunedin, New Zealand, in 1972 and 1973, who were enrolled at birth in a research project. Each was interviewed at 13, 15 and 18 about cannabis use, tested to determine which type of COMT genes they had inherited, and followed up at 26 for signs of mental illness.</p>
<p>COMT was chosen as it is known to play a part in the production of dopamine, a brain-signalling chemical that is abnormal in schizophrenia. It comes in two variants, known as valine or methionine, and every person has two copies, one from each parent.</p>
<p>Among people with two methionine variants, the rate of psychotic illness was 3 per cent, the background rate for the general population, regardless of whether they had used cannabis as teenagers.</p>
<p>Among those with two valine variants the rate was 3 per cent for non-users but 15 per cent for those who had smoked cannabis in their teens.</p>
<p>Dr Caspi said research had shown that the valine gene variant and cannabis affect the brain’s dopamine system in similar fashion, suggesting that they deliver a “double dose” that can be damaging. The work needs to be replicated by others to confirm the findings, Dr Caspi said. It also is possible that the gene involved is not COMT but a neighbour.</p>
<p><strong>THE DRUG OF CHOICE FOR MILLIONS</strong></p>
<p>• Cannabis was reclassified from a Class B to a Class C drug in January 2004. Possession remains illegal, but is not an arrestable offence. The Home Secretary has asked for a review by November<br />
• The Home Office estimates that fifteen million people have tried cannabis, two million to five million are regular users and reclassification has saved 199,000 hours’ police work<br />
• Liberalisation campaigners argue that millions smoke the drug with fewer ill-effects than others suffer from alcohol or tobacco<br />
• A recent study at Maastricht University found that cannabis doubles the risk of schizophrenia, hallucinations and paranoia among a genetically susceptible group</p>
<p>Source: www.timesonline.co.uk 14 April 2005</p>
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		<title>&#8216;Cannabis causes chaos in the brain&#8217;</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/cannabis-causes-chaos-in-the-brain/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/cannabis-causes-chaos-in-the-brain/#comments</comments>
		<pubDate>Sat, 05 Nov 2011 18:40:55 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Brain and Behaviour]]></category>
		<category><![CDATA[Cannabis]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7907</guid>
		<description><![CDATA[Cannabis causes chaos in the brain as nerve activity becomes uncoordinated and inaccurate, a study has found. The results may help explain links between cannabis and schizophrenia, scientists believe. Researchers at the University of Bristol measured the brain responses of rats given a drug that mimics the psychoactive ingredient in cannabis. They found that the [...]]]></description>
			<content:encoded><![CDATA[<p>Cannabis causes chaos in the brain as nerve activity becomes uncoordinated and inaccurate, a study has found.    The results may help explain links between cannabis and schizophrenia, scientists believe.<br />
Researchers at the University of Bristol measured the brain responses of rats given a drug that mimics the psychoactive ingredient in cannabis.  They found that the drug completely disrupted co-ordinated brain waves across the hippocampus and prefrontal cortex.<br />
The first brain region plays a key role in the formation of memories. The second is essential to planning, decision making and social behaviour. Both are heavily implicated in schizophrenia.   Rats exposed to the cannabis-like drug became unable to make accurate decisions when navigating through a maze.<br />
The research is reported today in the Journal of Neuroscience.<br />
Study leader Dr Matt Jones said: &#8220;Marijuana abuse is common among sufferers of schizophrenia and recent studies have shown that the psychoactive ingredient of marijuana can induce some symptoms of schizophrenia in healthy volunteers.<br />
&#8220;These findings are therefore important for our understanding of psychiatric diseases, which may arise as a consequence of &#8216;disorchestrated brains&#8217; and could be treated by retuning brain activity.&#8221;   Co-author Michal Kucewicz, also from the University of Bristol, said: &#8220;These results are an important step forward in our understanding of how rhythmic activity in the brain underlies thought processes in health and disease.&#8221;<br />
The research was part of a Medical Research Council-funded collaboration between the university and drug company Eli Lilly &#038; Co. </p>
<p>Source:  The Independent.   26th October 2011</p>
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		<title>Children of addicts &#8216;more likely to experience problems in adulthood&#8217;</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/children-of-addicts-more-likely-to-experience-problems-in-adulthood/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/children-of-addicts-more-likely-to-experience-problems-in-adulthood/#comments</comments>
		<pubDate>Sat, 05 Nov 2011 18:08:43 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Alcohol]]></category>
		<category><![CDATA[Parents]]></category>
		<category><![CDATA[Youth]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7902</guid>
		<description><![CDATA[Children of drug addicts are suffering in desperation as shame and secrecy shroud the substance misuse in families, it was claimed today. Youngsters whose parents take drugs are also more likely to have problems with substances, as well as their mental health, social skills and academically, a seminar heard. Joan O’Flynn, director of the National [...]]]></description>
			<content:encoded><![CDATA[<p>Children of drug addicts are suffering in desperation as shame and secrecy shroud the substance misuse in families, it was claimed today.</p>
<p>Youngsters whose parents take drugs are also more likely to have problems with substances, as well as their mental health, social skills and academically, a seminar heard.  Joan O’Flynn, director of the National Advisory Committee on Drugs (NACD), said there is a need for more integration between addiction services, children’s services and medical professionals.</p>
<p>“Alcohol and drugs misuse by parents can impact negatively on a child’s experience of positive parenting and can create stressful family circumstances that impact on child development,” she said.  “For many of the affected children, the effect of their parents’ substance misuse continues into their adult lives.</p>
<p>“For some, the impact can be multifaceted and persist not only into adult life but even into the lives of the next generation.” She added that stress, combined with the increased likelihood of the child being in care or homeless, leaves young people at a high risk of emotional isolation or social marginalisation.</p>
<p>Alcohol Action Ireland estimates between 61,000 and 104,000 children aged under 15 are living with parents who misuse alcohol.  Director Fiona Ryan said: “Shame and secrecy shroud the issue of substance misuse in families with children living lives of quiet desperation.</p>
<p>“Alcohol Action Ireland has spent the past three years campaigning for children affected by parental alcohol problems to be seen and heard.”  An NACD report – &#8216;Parental Substance Misuse: Addressing its Impact on Children&#8217; &#8211; was launched at a seminar it jointly hosted with the Health Service Executive (HSE) and Alcohol Action Ireland, the national charity for alcohol-related issues.</p>
<p>The report reviewed all major international research on the impact of parental substance misuse on children and identified what steps can be taken in Ireland to reduce its impact.</p>
<p>It recommended additional research and data be collected to properly estimate the number of children whose parents have substance misuse problems.  It also wants an assessment of which adult alcohol and drug treatment services are supporting parents and liaising with child support services.  Women should also be educated on the adverse effects of consuming alcohol and drugs during pregnancy, it added.</p>
<p>Source: www.IrishExaminer.com  26th October 2011 </p>
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		<title>Apparent Success of Drug Treatment Aimed at Heroin is Misleading</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/apparent-success-of-drug-treatment-aimed-at-heroin-is-misleading/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/apparent-success-of-drug-treatment-aimed-at-heroin-is-misleading/#comments</comments>
		<pubDate>Fri, 04 Nov 2011 13:35:48 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Political Sector (Drug Politics)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7898</guid>
		<description><![CDATA[Irish research shows addicts on methadone programme still abusing crack cocaine and other substances. The Irish Government drugs policy needs to change There has been an apparent levelling off of the need for opiate centred drug treatment. However the researchers believe their findings show that this is misleading. Their evidence suggests that multiple drug use [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;">Irish research shows addicts on methadone programme still abusing crack cocaine and other substances. The Irish Government drugs policy needs to change<br />
There has been an apparent levelling off of the need for opiate centred drug treatment. However the researchers believe their findings show that this is misleading. Their evidence suggests that multiple drug use is the norm among many addicts.</p>
<p><strong>Realities of Drug Misuse Investigated<br />
</strong><br />
The study was led by Dr A. Jamie Saris (Principal Investigator) and Fiona O’Reilly (Primary Field Researcher), Dept of Anthropology at NUI Maynooth and is the result of a long-term study which closely examined the realities of drug misuse in three adjacent neighbourhoods.<br />
Of 92 abusers surveyed, 98% were on a methadone drugs treatment programme yet almost two thirds claimed to have used heroin within the past 3 months. Whilst over half were on prescription tranquilisers almost as many had used illegally obtained tranquilisers. Nearly one third had used crack cocaine and more than one in five powder cocaine. “Multiple drug use is the reality for nearly all users, and official policy needs to have this understanding at its centre”, claims Dr Saris.</p>
<p><strong>Stigma Against Heroin Among the Young<br />
</strong><br />
A surprising finding was that there is a stigma against heroin among many of the younger users (aged 16 to 25). But these individuals still abuse what the study team describe as a “dizzying array” of other substances. The established approach to treatment, being so heavily focused on heroin, means that the issues faced by such people are not being addressed.<br />
Another problem with the focus on crack and heroin is that it sets the users of those drugs apart from society when, in fact, such people are rarely defined solely by their addiction. A lot of local community activities aimed at assisting users recognise that they often lead lives that are not so very different from everyone else.<br />
Drug Treatment Services Should Focus on Individuals<br />
However it is often difficult to justify such activities to official funders under the rubric of ‘treatment’, as currently understood. Dr Saris believes that it is important to understand who users are, what they are taking and why, so that the authorities can assign the appropriate resources, treatments or management systems.<br />
Tony MacCarrthaigh chairs the Local Drugs Task Force that covers the area of the study and he agrees with Dr Saris. “Individuals and not chemicals, need to become the focal point of treatment, and that treatment needs to assist individuals in developing another orientation not just to drugs, but to life,” he said. (A Dizzying Array of Substances; An Ethnographic Study of Drug Use in the Canal Communities, Department of Anthropology, NUI Maynooth, 2010.)</p>
<p><em>Source: Apparent Success of Drug Treatment Aimed at Heroin is Misleading<br />
</em></p>
<p>http://news.suite101.com/article.cfm/apparent-success-of-drug-treatment-aimed-at-heroin-is-misleading-a259572#ixzz0tO3OAGXw</p>
<p></span></p>
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		<title>Abolist NTA to Cut Drug Addiction</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/abolist-nta-to-cut-drug-addiction/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/abolist-nta-to-cut-drug-addiction/#comments</comments>
		<pubDate>Fri, 04 Nov 2011 13:33:31 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Legal Sector (Drug Politics)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7896</guid>
		<description><![CDATA[&#8220;Methadone prescriptions for heroin addicts would be cut and the National Treatment Agency that runs the programme scrapped under plans from the Tories favourite think-tank,&#8221; reports Rosemary Bennett, social affairs correspondent of The Times newspaper. &#8220;The Centre for Social Justice, set up by Iain Duncan Smith, the Work and Pensions Secretary, said it was unacceptable [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;">&#8220;Methadone prescriptions for heroin addicts would be cut and the National Treatment Agency that runs the programme scrapped under plans from the Tories favourite think-tank,&#8221; reports Rosemary Bennett, social affairs correspondent of The Times newspaper.<br />
&#8220;The Centre for Social Justice, set up by Iain Duncan Smith, the Work and Pensions Secretary, said it was unacceptable that only 4% of addicts in treatment ever get “clean” and accused the agency of “pushing aside” proper rehabilitation. The Times has also learnt that the highly influential think-tank will use a report on Monday to throw its weight behind Ken Clarke, the Justice Secretary, who called for short prison sentences to be scrapped.The report will state that the CSJ agrees with him that short sentences of two months do nothing to help to rehabilitate offenders and should be replaced by community orders.&#8221;<br />
The CSJ&#8217;s Green Paper on Criminal Justice and Addiction comes as the government considers major changes to drug policy and the future of the National Treatment Agency. Set up in 2001, the NTA oversees the controversial “harm reduction” strategy &#8211; most recent NTA treatment statistics show that of the 207,000 addicts a year who use &#8216;treatment&#8217; services, only 8,980 completed their treatment drug free.4,600 addicts have access to residential rehabilitation.Numerous residential drug rehabilitation centres have closed because of lack of patients, despite no sharp fall in the number of addicts.<br />
The CSJ said that the NTA, the running costs of which have spiralled to £18million a year, merely processes addicts with a “fatalistic” belief that they can never get clean. It wants it scrapped and replaced by an Addiction Recovery Board, chaired by a minister and charged with getting addicts off drugs altogether, using the best local private sector and charity programmes, or “recovery communities”.<br />
The report says there is a role for methadone, but it should be used only as part of a wider treatment programme, with abstinence the goal.<br />
&#8220;There is no strategy or incentive to reduce the numbers on maintenance treatment and move people into recovery,&#8221; the CSJ said. The report is also highly critical of how drug use is tolerated in prison: 55% of prisoners received into custody each year are classified as problematic drug users. According to the Ministry of Justice, one in five men who reports using mainstream drugs first used them in prison.</p>
<p><em>Source:www.addictiontoday.org.  July 10th 2010<br />
</em><br />
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		<title>Experts Call New Strategies on AIDS Prevention Ineffective!</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/experts-call-new-strategies-on-aids-prevention-ineffective/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/experts-call-new-strategies-on-aids-prevention-ineffective/#comments</comments>
		<pubDate>Fri, 04 Nov 2011 13:31:26 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Political Sector (Drug Politics)]]></category>
		<category><![CDATA[USA]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7894</guid>
		<description><![CDATA[On Tuesday, July 20th 2010, internationally recognized anti-drug experts from every region of the world united to oppose a set of dangerous unproven global strategies recommended in the Vienna Declaration. The declaration is based on three false premises: 1) that the criminalization of illegal drug use fuels the HIV/AIDS epidemic, 2) that criminal justice and [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;">On Tuesday, July 20th 2010, internationally recognized anti-drug experts from every region of the world united to oppose a set of dangerous unproven global strategies recommended in the Vienna Declaration. The declaration is based on three false premises: 1) that the criminalization of illegal drug use fuels the HIV/AIDS epidemic, 2) that criminal justice and health promotion are conflicting approaches to drug policy, and 3) that the major costs of illegal drug use are those generated by the criminal justice system.<br />
This document was released in anticipation of the 18th International AIDS Conference and has been under scrutiny by several non-governmental organizations.  Calvina Fay, Executive Director of Drug Free America Foundation says, “There is no ‘reasonable evidence’ that supports the strategies outlined in the Vienna Declaration. Further, we should reject ineffective harm reduction tactics that are not based on scientific evidence while accepting drug use and creating an illusion that drugs can be used safely or responsibly. Such ill-conceived schemes foster the misunderstanding that drug use itself is not harmful and increases addiction.”<br />
Many of the experts who opposed the Vienna Declaration know from research and practical experience that the optimal way to truly beat addiction, prevent the spread of AIDS and other sexually transmitted diseases, and prevent drug-related harm are effective strategies that target drug use and include prevention, education, treatment and law enforcement efforts and do not trade one for the other.<br />
&#8220;The best foundation for prevention is policy. We know from experience that a balanced and restrictive drug policy is effective in keeping drug use at low levels. Since drug utilization in itself is an important risk factor for being infected by HIV, it is good AIDS-prevention to preclude illicit drug use. We must always strive to protect young people from getting involved with illegal drugs,” says Sven-Olov Carlsson, International President, World Federation Against Drugs.</p>
<p>To view the full joint statement issued opposing the Vienna Declaration, please visit www.wfad.se.  If you would like to conduct an interview with Ms. Fay, Mr. Carlsson and/or other drug policy and prevention experts on this statement, please contact Lana Beck, Director of Communications with Drug Free America Foundation, Inc. at 727-828-0211 or 727-403-7571.</p>
<p>The World Federation Against Drugs (www.wfad.se) is a multilateral community of non-governmental organizations and individuals. Founded in 2009, the aim of WFAD is to work for a drug-free world. Drug Free America Foundation (www.dfaf.org) is a national and international nonprofit organization dedicated to fighting drug use, drug addiction and drug trafficking and to promoting effective sound drug policies, education and prevention.  Drug Free America Foundation is a Non-Governmental Organization (NGO) in Special Consultative Status with the Economic and Social Council of the United Nations.</p>
<p>For More Info Contact  Lana Beck 727-828-0211 or 727-403-7571 after hours</p>
<p><em>Source: Joint  Press Release from www.wfad.se and www.wfad.se  July 2010<br />
</em><br />
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		<title>Joint Statement in Opposition to the Vienna Declaration Released July 20, 2010</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/joint-statement-in-opposition-to-the-vienna-declaration-released-july-20-2010/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/joint-statement-in-opposition-to-the-vienna-declaration-released-july-20-2010/#comments</comments>
		<pubDate>Fri, 04 Nov 2011 13:29:21 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Europe]]></category>
		<category><![CDATA[Legal Sector (Drug Politics)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7892</guid>
		<description><![CDATA[The criminalization of illicit drug use provides positive health and social benefits by deterring nonmedical use of substances that cause great harm to HIV/AIDS-affected individuals. Incarceration that respects human rights and provides drug treatment services can accelerate an individual’s recovery from drug dependence and prevent drug-related harms to HIV/AIDS-affected individuals and prevent further proliferation of [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;">The criminalization of illicit drug use provides positive health and social benefits by deterring nonmedical use of substances that cause great harm to HIV/AIDS-affected individuals. Incarceration that respects human rights and provides drug treatment services can accelerate an individual’s recovery from drug dependence and prevent drug-related harms to HIV/AIDS-affected individuals and prevent further proliferation of both diseases &#8211; HIV/AIDS and substance abuse.<br />
In anticipation of the International AIDS Conference (AIDS 2010) from July 18-23, 2010,i the Vienna Declarationii was released by a group of non-governmental organizations (NGOs) and signed by private individuals to outline a global strategy to deal with the modern drug epidemic. The Vienna Declaration is based on three false premises:<br />
 1) that the criminalization of illegal drug use fuels the HIV/AIDS epidemic,<br />
 2) that criminal justice and health promotion are conflicting approaches to drug<br />
     policy, and<br />
3) that the major costs of illegal drug use are those generated by the criminal justice        system. </p>
<p>The prohibition of illegal drug use does not encourage the spread of HIV/AIDS, but rather it reduces illegal drug use among HIV/AIDS patients, as well as the non-infected population and thereby reduces the population vulnerable to HIV/AIDS infection by contaminated needles. Illegal drug use exacerbates weaknesses of the immune system, making individuals with AIDS more susceptible to infection and death. iii Marijuana use causes impaired immunity,iv v vi vii and opens the door for the virus that causes Kaposi’s Sarcoma,viii life-threatening for individuals with HIV/AIDS. Marijuana also contains bacteria and fungi that put users at risk for infection. ix x xi Illegal drug use among AIDS patients is life-threatening because these drugs lessen the effectiveness of anti-retroviral (ARV) medications.xii Nonmedical drug use is associated with increased risky sexual behaviors which promote transmission of HIV/AIDS in a way that needle exchange cannot prevent. xiii xiv<br />
Illegal drug use also increases sexual violence which in turn results in more HIV infections, particularly among the most vulnerable members of society including womenxv as well as children. Mother-to-child transmission of HIV/AIDS now can be largely prevented by medical intervention; however, there is no protection for unborn fetuses from the adverse effects of a drug-using mother. xvi Joint Statement in Opposition to the Vienna Declaration Released July 20, 2010 Page 2 </p>
<p>There are 200 million illegal drug users globally, making up 5% of the world population aged 16-64,xvii and an estimated 33.4 million people living with HIV/AIDS.xviii Since the emergence of the HIV/AIDS epidemic in 1981, an estimated 25 million people have died of HIV/AIDS-related causes and two million people die each year from this disease.xix These numbers are tragically high, but so is the number of global drug-related deaths, estimated at 223,000 each year. xx As previously noted, illegal drug use increases the risks associated with both contracting and treating HIV/AIDS. Reducing drug use must be part of the solution to curb the distressingly high HIV/AIDS death toll<br />
.<br />
The Vienna Declaration concludes that “reorienting drug policies towards evidence-based approaches that respect, protect and fulfill human rights has the potential to reduce harms deriving from current policies and would allow for the redirection of the vast financial resources towards where they are needed most: implementing and evaluating evidence-based prevention, regulatory, treatment and harm reduction interventions.” Prevention and treatment are admirable goals which aim to reduce illegal drug use; however many so-called “harm reduction” interventions normalize illegal drug use and inevitably lead to more nonmedical use of drugs, leading to more drug-caused harm. Real harm reduction is achieved by rejecting illegal drug use to improve the health and safety of would-be drug users. </p>
<p>To promote public health and public safety, and to reduce both illegal drug use and HIV/AIDS, the World Federation Against Drugs (WFAD), Drug Free America Foundation, Inc. (DFAF), Institute for Behavior and Health, Inc. (IBH) and numerous other organizations and individuals support a balanced restrictive drug policy that uses the criminal justice system, and the illegal status of nonmedical drug use, to reinforce both prevention and treatment. The current globally-endorsed balanced drug abuse prevention policy can be improved. Treatment systems can work together with the criminal justice system by incorporating new, effective and evidence-based strategies to reduce illegal drug use among criminal offenders. These approaches also reduce the commission of new crimes and associated incarceration.<br />
The greatest costs of illegal drug use are not generated by the criminal justice system but by the nonmedical drug use itself. These costs include not only sickness and death but reduced productivity and the high healthcare costs generated by illegal drug use. </p>
<p>We are committed to efforts to improve current drug policy to further reduce illegal drug use by building on a balanced strategy that includes the criminal justice system. Rather than choosing between prevention and treatment on the one hand, and the criminal justice system on the other, it is important to find better ways for them to work together to achieve vital public health and public safety goals that neither can achieve alone. We know that the prevention of illegal drug use and HIV/AIDS prevention must go hand-in-hand; they are not in conflict with one another. </p>
<p>Organizations:<br />
Sven-Olov Carlsson, International President, World Federation Against Drugs, www.wfad.se<br />
Robert L. DuPont, M.D., President, Institute for Behavior and Health, Inc., www.ibhinc.org<br />
David Evans, Esq., Executive Director, Drug Free Projects Coalition,<br />
www.studentdrugtesting.org/<br />
Calvina Fay, Executive Director, Drug Free America Foundation, Inc., www.dfaf.org<br />
Members, International Task Force on Strategic Drug Policy, www.itfsdp.org Joint Statement in Opposition to the Vienna Declaration Released July 20, 2010 Page 3 </p>
<p><em>Source: Joint  Press Release www.dfaf.org and www.wfad.se  July 20 2010 </em> </p>
<p>REFERENCES:                                                                                                                                                                                                                                    XVIII International AIDS Conference. (2010). Retrieved July 12, 2010 from http://www.aids2010.org/<br />
ii The Vienna Declaration. (2010). Retrieved June 30, 2010 from http://www.viennadeclaration.com/the-declaration.html<br />
iii Antoniou, T., &#038; Tseng, L. (2002). Interactions between recreational drugs and antiretroviral agents. Annual of Pharmacotherapy, 36, 1598-1613.<br />
iv Cabral, G.A., &#038; Vasquez, R. (1992). Delta-9-Tetrahydrocannabinol suppresses macrophage extrinsic anti-herpes virus activity, Proceedings of the Society for Experimental Biology and Medicine, 199(2), 255-63.<br />
v American College of Allergy, Asthma and Immunology. (2004, November 17). Immunological changes associated with prolonged marijuana smoking.<br />
vi Tashkin, D.P., Baldwin, G.C., Sarafian, T., Dubinett, S., &#038; Roth, M.D. (2002). Respiratory and immunologic consequences of marijuana smoking. Journal of Clinical Pharmacology, 42(11 Suppl), 71S-81S.<br />
vii Wu, T.C., Tashkin, D.P., Djahed, B., &#038; Rose, J.E. (1988). Pulmonary hazards of smoking marijuana as compared with tobacco. New England Journal of Medicine, 318(6), 347-351.<br />
viii American Association for Cancer Research. (2007, August 2). Marijuana component opens the door for virus that causes Kaposi’s sarcoma. ScienceDaily. Retrieved July 7, 2010 from http://www.sciencedaily.com/releases/2007/08/070801112156.htm<br />
ix Fleisher, M., Winawer, S.J., &#038; Zauber, A.G. (1991). Aspergillosis and marijuana. [Letter]. Annals of Internal Medicine, 115, 578-579.<br />
x Ramirez, J. (1990). Acute pulmonary histoplasmosis: newly recognized hazard of marijuana plant hunters. American Journal of Medicine, 88(5), 60N-62N.<br />
xi Taylor, D.N., Wachsmuth, I.K., Shangkuan, Y.H., Schmidt, E.V., Barrett, T.J., et al. (1982). Salmonellosis associated with marijuana: A multi state outbreak traced by plasmid fingerprinting. New England Journal of Medicine, 306(21), 1249-1253.<br />
xii Ghaziani, A. (2005, October). Crystal methamphetamine use and antiretroviral drug resistance: A pilot study of behavioral and clinical correlates. International Association of Physicians in AIDS Care. IAPAC Monthly, 297-299. Retrieved July 9, 2010 from http://img.thebody.com/legacyAssets/22/36/meth.pdf<br />
xiii Wechsberg, W.M., Parry, C.D.H., &#038; Jewkes, R.K. (2010, May). Drugs, sex, gender-based violence, and the intersection of the HIV/AIDS epidemic with vulnerable women in South Africa. RTI Press. Retrieved July 9, 2010 from http://www.rti.org/pubs/pb-0001-1005-wechsberg.pdf<br />
xiv Colfax, G., Coates, T.J., Husnik, M.J., Huang, Y., Buchbinder, S., Koblin, B., et al. (2005). Longitudinal patterns of methamphetamine, popper (amyl nitrite), and cocaine use and high-risk sexual behavior among a cohort of San Francisco men who have sex with men. Journal of Urban Health, 82(1 Suppl 1), i62-i70.<br />
xv Wechsberg, W.M., Parry, C.D.H., &#038; Jewkes, R.K. (2010, May). Drugs, sex, gender-based violence, and the intersection of the HIV/AIDS epidemic with vulnerable women in South Africa. RTI Press. Retrieved July 9, 2010 from http://www.rti.org/pubs/pb-0001-1005-wechsberg.pdf<br />
xvi World Health Organization. (2010). PMTCT strategic vision 2010-2015: Preventing mother-to-child transmission of HIV to reach the UNGASS and millennium development goals. Retrieved July 9, 2010 from http://www.who.int/hiv/pub/mtct/strategic_vision.pdf<br />
xvii United Nations Office on Drugs and Crime. (2010). World Drug Report 2010. New York: United Nations. Retrieved July 7, 2010 from http://www.unodc.org/documents/wdr/WDR_2010/World_Drug_Report_2010_lo-res.pdf<br />
xviiiUNAIDS. (2009, December). Global facts &#038; figures. Retrieved July 7, 2010 from http://data.unaids.org/pub/FactSheet/2009/20091124_FS_global_en.pdf<br />
xixUNAIDS. (2009, December). Global facts &#038; figures. Retrieved July 7, 2010 from http://data.unaids.org/pub/FactSheet/2009/20091124_FS_global_en.pdf<br />
xx National Drug Research Institute. (2003, February 25). Tobacco, alcohol and illicit drugs responsible for seven million preventable deaths worldwide. Media release. Retrieved July 7, 2010 from http://db.ndri.curtin.edu.au/media.asp?mediarelid=40</p>
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		<title>Mexico looks to legalisation as drug war murders hit 28,000</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/mexico-looks-to-legalisation-as-drug-war-murders-hit-28000-2/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/mexico-looks-to-legalisation-as-drug-war-murders-hit-28000-2/#comments</comments>
		<pubDate>Fri, 04 Nov 2011 13:25:55 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Effects of Drugs (Drug Politics)]]></category>
		<category><![CDATA[Legal Sector (Drug Politics)]]></category>
		<category><![CDATA[South America]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7888</guid>
		<description><![CDATA[President joins calls for debate after figures reveal extent of violence since launch of military offensive against cartels in 2006. Murders in Mexico&#8217;s drug wars are becoming increasingly gruesome. Mexico&#8217;s president, Felipe Calderón, has joined calls for a debate on the legalisation of drugs as new figures show thousands of Mexicans every year being slaughtered [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;"> President joins calls for debate after figures reveal extent of violence since launch of military offensive against cartels in 2006.</p>
<p>Murders in Mexico&#8217;s drug wars are becoming increasingly gruesome. </p>
<p>Mexico&#8217;s president, Felipe Calderón, has joined calls for a debate on the legalisation of drugs as new figures show thousands of Mexicans every year being slaughtered in cartel wars.<br />
&#8220;It is a fundamental debate,&#8221; the president said, belying his traditional reluctance to accept any questioning of the military-focused offensive against the country&#8217;s drug cartels that he launched in late 2006. &#8220;You have to analyse carefully the pros and cons and key arguments on both sides.&#8221; The president said he personally opposes the idea of legalisation.<br />
Calderón&#8217;s new openness comes amid tremendous pressure to justify a strategy that has been accompanied by the spiralling of horrific violence around the country as the cartels fight each other and the government crack down. Official figures released this week put the number of drug war related murders at 28,000.<br />
Until recently the government regularly played down the general impact of the violence by claiming that 90% of the victims were associated with the cartels, with the remainder largely from the security forces. In recent months it has started to acknowledge a growing number of &#8220;civilian victims&#8221; ranging from toddlers caught in the cross fire to students massacred at parties.<br />
Momentum behind the idea that legalisation could be part of the solution has been growing since three prominent former Latin American presidents signed a document last year arguing the case.<br />
César Gaviria of Colombia, Fernando Cardoso of Brazil and Ernesto Zedillo of Mexico urged existing governments to consider legalising marijuana as a way of slashing cartel profits.<br />
This year Mexico&#8217;s national congress began a debate on the possibility that resurfaced again this week during a series of round table discussions between the Calderón, security experts, business leaders and civic groups.<br />
The &#8220;Dialogue for Security: Evaluation and Strengthening&#8221; is part of a new government effort to counter the growing perception in Mexico that the president&#8217;s drug war strategy is a disaster.<br />
&#8220;I&#8217;m not talking just about legalizing marijuana,&#8221; analyst and write Hector Aguilar Camin said during the Tuesday session, &#8220;rather all drugs in general.&#8221;<br />
After accepting the need to directly address the proposal, Calderón made it clear he did not support it. &#8220;It requires a country to take a decision to put several generations of young people at risk,&#8221; he said, citing a likely increase in consumption triggered by lower prices, greater availability and social acceptability.<br />
He added that the predicted &#8220;important economic effects by reducing income for criminal groups&#8221; would be limited by the integration of Mexican drug trafficking into international markets where drugs remain largely underground.<br />
Calderón did not mention current moves to soften drug laws in the US, including a planned vote in California in November on an initiative that would allow marijuana to be sold and taxed. Nor did he address the home grown argument that legalisation would remove the roots of the violence raging in the country.<br />
&#8220;Legalisation would render the war pointless as drugs would become just another product like tobacco or alcohol,&#8221; Jorge Castañeda, a legalisation advocate and former foreign minister, told W Radio. He added that even if it did prompt an increase in drug use. &#8220;It is worth considering whether this is preferable to having 28,000 deaths.&#8221;<br />
The new death toll, which was not broken down, is significantly higher than the informal counts kept by newspapers. Milenio newspaper put the number of drug-related deaths in July at 1,234.<br />
Some leading critics of Calderón&#8217;s strategy, however, do not believe legalisation is the key to reining in the cartels and the violence, preferring to emphasize the need to increase efforts to go after money laundering and political corruption.<br />
Edgardo Buscaglia, and expert in organised crime around the world, argues that the recent diversification of the Mexican cartels into other criminal activities ranging from systematic extortion to people trafficking would give them ample reason to keep fighting each other, even if drugs were legal. &#8220;Legalising drugs would be good public policy,&#8221; he said, &#8220;but it would not be a tool with which to combat organized crime.&#8221;</p>
<p><em>Source:  guardian.co.uk, Wednesday 4 August 2010 20.13 BST<br />
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		<title>NADCP and Drug Court Leaders Respond to Criticisms With the Facts</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/nadcp-and-drug-court-leaders-respond-to-criticisms-with-the-facts/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/nadcp-and-drug-court-leaders-respond-to-criticisms-with-the-facts/#comments</comments>
		<pubDate>Fri, 04 Nov 2011 13:22:56 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Legal Sector (Drug Politics)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7886</guid>
		<description><![CDATA[The following is an interesting article about Drug Courts in the USA and how successful they are. It is in response to criticisms by the NACDL about drug courts. Setting the Record Straight: Criticisms Answered The National Association of Drug Court Professionals (NADCP) Board of Directors has unanimously approved an official position statement regarding the [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;">The following is an interesting article about Drug Courts in the USA and how successful they are.  It is in response to criticisms by the NACDL about drug courts.<br />
Setting the Record Straight: Criticisms Answered</p>
<p>The National Association of Drug Court Professionals (NADCP) Board of Directors has unanimously approved an official position statement regarding the 2009 report by the National Association of Criminal Defense Lawyers (NACDL) purporting to identify deficiencies in the practices of Drug Courts. Following the release of their report last September, NACDL used attacks on Drug Courts to launch an aggressive media campaign. Each attack on Drug Courts was met with a thorough and factual response from NADCP. These responses, and others, are listed below. </p>
<p>NADCP CEO West Huddleston and NADCP Chief of Science, Law, and Policy Doug Marlowe authored the official position statement to correct assertions made in the NACDL report that are unsupported by research, as well as address some areas of common concern. NADCP encourages Drug Court professionals to use the statement as a tool for answering these criticisms and concerns should they arise. </p>
<p> Missouri Law Quarterly<br />
April, 2010</p>
<p>Drug Courts Save Lives and Money: So Why the Criticisms?<br />
by Dr. Douglas Marlowe, Chief of Science, Law and Policy, NADCP </p>
<p>More research has been published on the effects of Drug Courts than on virtually all other criminal justice programs combined.  By 2006, the scientific community had concluded beyond a reasonable doubt from what are called meta-analyses (highly advanced statistical procedures) that Drug Courts reduce crime and return financial benefits to society which are several times the initial investments.  A large-scale study funded by the National Institute of Justice and recently completed in 2009—called the Multi-Site Adult Drug Court Evaluation, or MADCE— has confirmed, once again, that Drug Courts reduce crime, reduce substance abuse, improve family relationships, and increase employment and school enrollment.</p>
<p>Yet, just as the scientific evidence is coming in decidedly in favor of Drug Courts, criticisms of Drug Courts appear to be reaching a surprising crescendo in opinion editorials and non-scientific law journals.  How can we explain this seeming paradox?  If the criminal justice system endorses evidence-based practices, why should negative sentiments be rising alongside favorable research findings?</p>
<p>The answer is at least two-fold.  One group of critics appears to be turning an intentionally blind eye to the research evidence to serve a drug-decriminalization policy agenda.  Although they may use scientific language to defend their objections, no amount of data could ever dissuade them from their position.  A second group of critics, however, recognizes the proven efficacy of Drug Courts, but worries that some Drug Courts might produce other negative side-effects which should also be taken into account, such as impeding zealous representation by defense counsel.  Because these latter critics are swayed by data, their concerns are capable of being empirically tested; and if confirmed, can point the way toward corrective measures that will advance the field rather than move it further and further behind. </p>
<p>One would be hard-pressed to point to a negative commentary on Drug Courts that does not, within the same pages, endorse a drug-decriminalization or legalization agenda.  For decades, drug legalizers could take steady aim at the so-called “War on Drugs” with its undue emphasis on mandatory sentencing and incarceration.  Such criticisms were easy to level, because the War on Drugs has been both prohibitively costly and largely ineffective at reducing drug abuse or crime.  </p>
<p>But Drug Courts throw a potential curve ball to these arguments.  Drug Courts prove that drug abuse can remain illicit without necessitating a costly and draconian punitive response.  We can hold people accountable for their dangerous behavior, while at the same time supervising them in the community and providing them with needed treatment and other services.  This finding could be seen by some as sweeping the legs out from under the strongest rationale for drug decriminalization.  And for this reason, it has elicited a steady stream of vehement antagonism framed in the guise of an objective scientific analysis. </p>
<p>Other critics, however, recognize that even beneficial treatments have the potential to cause unwanted side-effects.  For example, aspirin is proven to reduce pain but in some cases can cause unintended ulcers or blood thinning.  This has required the medical field to take remedial measures to reduce the likelihood that such side-effects will occur and to treat any negative symptoms that do emerge.  By analogy, there is always the possibility that some Drug Courts might misapply their authority or mishandle their operations to the detriment of their participants.  Moreover, there is the possibility that some types of addicted offenders might not respond well to the Drug Court model and should be treated in other ways.  </p>
<p>There are two problems, however, with how these arguments have typically been framed by critics of Drug Courts.  First, they assume facts not in evidence, and second, they often seek the wrong remedy.  A review of the research literature through February of 2010 failed to uncover a single empirical study confirming any of the untoward effects that have been attributed by critics to Drug Courts.  For example there is no reliable evidence (apart from some critics’ personal anecdotes) that Drug Courts impede adequate evidentiary discovery by defense counsel or sentence terminated defendants more harshly than if they had never entered the Drug Court.</p>
<p>It would not be a difficult matter, however, to study these questions in a scientifically defensible manner.  If such negative effects do exist, then corrective measures can be developed and tested to address them.  And finally, practice guidelines can be developed to ensure that all Drug Courts adhere to best practices and take reasonable efforts to avoid foreseeable injuries.  There is no need to “throw out the baby with the bath water.”  The indicated remedy is not to abandon the most successful program we have in the criminal justice system.  The appropriate course of action is to conduct more sophisticated research to improve the intervention and to develop standards to guide the actions of Drug Court professionals.</p>
<p>Drug Courts are here to stay not because they are politically palatable, but because they have withstood, time and again, rigorous empirical scrutiny.  They work where few other programs have.  The time has come for the Drug Court field to reach full maturity.  And like other mature disciplines, such as medicine or psychology, this means developing guidelines for effective and ethical practices.</p>
<p>The time has come for serious-minded constituencies to cease taking blind swipes at Drug Courts and vying for attention and limited resources.  We need to come together to determine who should be treated in Drug Courts, how to optimize Drug Court operations, and how to avoid or redress any potential harms.  This is what is meant by rational drug policy.</p>
<p> Governing Magazine<br />
January, 2010<br />
by West Huddleston, Chief Executive Officer, NADCP</p>
<p>John Buntin’s recent profile of Judge Stephen Alm and Hawaii’s promising H.O.P.E program is an encouraging sign that our nation’s probation system is ready for change (Swift and Certain, Hawaii&#8217;s Probation Experiment &#8211; November, 2009). In highlighting the development of the H.O.P.E. program, Mr. Buntin correctly identified systemic changes to our criminal justice system brought about by the growth and widespread success of Drug Courts, which now exceed 2,300 nationwide. In doing so, however, Mr. Buntin also raised serious questions about Drug Courts that rigorous research has already answered. </p>
<p>In the twenty years since the first Drug Court was founded there has been more research published on its effects than virtually all other criminal justice programs combined. The verdict? Drug Courts significantly reduce substance abuse and crime at less expense than any other justice strategy.</p>
<p>Mr. Buntin inferred that little is known about Drug Court participants once they leave the program. Here are the facts. Research demonstrates that nationwide, 70% percent of the 120,000 annual participants in Drug Court complete the program and 75% remain arrest-free. The longest study on Drug Courts to date shows that community reductions in drug abuse and improved employment and family functioning outcomes can last as long as 14 years.</p>
<p>Judge Alm suggested that most Drug Courts employ an “ineffective” reliance on future punishment. This is not the case. Drug Courts utilize close supervision, urine monitoring, and a system of graduated sanctions to ensure participants are immediately held accountable for not living up to their obligations. The approach is a vast improvement over traditional criminal justice responses, which are often applied inconsistently and in an all or nothing manner which emphasizes the draconian response of incarceration. This is just part of the reason why Drug Courts work better than probation, jails or prison and better than treatment alone.</p>
<p> The Sacramento Bee<br />
October 16, 2010</p>
<p>Drug courts unfairly attacked<br />
by West Huddleston, Chief Executive Officer, NADCP</p>
<p>Re &#8220;Fresh look at drug courts could also ease prison crisis&#8221; (Viewpoints, Nov. 9): In its latest attack on drug courts, the National Association of Criminal Defense Lawyers reveals a startling comfort with distorting facts and ignoring the truth. In misrepresenting its recent anecdotal report as a &#8220;study,&#8221; the NACDL chooses to ignore two decades of conclusive research, including hundreds of studies that prove drug courts reduce crime, reduce drug abuse, reunite families and save considerable money for taxpayers.</p>
<p>Here are the facts. Nationwide, 70 percent of the approximately 120,000 seriously addicted individuals who voluntarily enter drug courts with the assistance of their defense attorney complete it a year or more later and 75 percent of them remain arrest-free. A drug court participant is more than twice as likely to stay clean and remain arrest-free than is a newly released state inmate. Research also concludes that drug courts reduce drug abuse and improve employment and family functioning.</p>
<p>These effects are not short-lived. The longest study on drug courts to date shows these outcomes last as long as 14 years. Clearly, drug courts are not an experiment. They must be expanded to serve the 1.2 million substance-abusing arrestees before the courts. That is the real issue.</p>
<p>With every blind attack on drug courts, the National Association of Criminal Defense Lawyers calls into question only its own credibility.</p>
<p> The Miami Herald<br />
October 13, 2009</p>
<p>Keep drug courts &#8212; they&#8217;re effective<br />
by Dr. Douglas Marlowe, Chief of Science, Law and Policy, NADCP</p>
<p>The National Association of Criminal Defense Lawyers chooses to attack our nation&#8217;s most successful justice intervention for substance abusing offenders: drug courts (Cynthia Orr, Sept. 29 Other Views column, Rethink how we fight drugs).</p>
<p>It minimizes the impact of drug courts like the one in Miami-Dade, which has restored more than 12,000 lives and reunited tens of thousands of family members. NACDL only begrudgingly accepts drug courts as an interim improvement over the war on drugs until decriminalization is accomplished.</p>
<p>Two decades of research have proven that drug courts reduce crime, reduce drug abuse and save considerable money for taxpayers. The most conservative estimate is that every $1 invested in drug courts reaps between $2 to $3 in direct cost-savings to society.</p>
<p>Between 50 percent and 80 percent of all crimes are committed by substance abusers. NACDL&#8217;S assertion that drug courts are only treating low-level offenders is patently false. The majority of drug courts now treat serious offenders who have failed repeatedly in treatment and other dispositions.</p>
<p>NACDL recommends that drug courts treat high-risk offenders who would otherwise be in jail or prison bound in programs that do not require a guilty plea for entry.</p>
<p>But this would mean that serious and potentially violent offenders would face no legal repercussions whatsoever if they failed to complete treatment or even to attend it. When we consider the safety of our communities such recommendations cannot be taken seriously.</p>
<p>The Philadelphia Inquirer<br />
October 24, 2009</p>
<p>Drug courts are needed; New Jersey shows why<br />
by Yvonne Smith Segars, New Jersey Public Defender (As New Jersey Public Defender, Yvonne Smith Segars is the head of the New Jersey Office of the Public Defender, an agency overseeing the Public Defender offices throughout state.)</p>
<p>Last Saturday&#8217;s editorial, &#8220;Who needs drug courts?,&#8221; asks a simple question. In reality, the answer is far more complex. Drug courts are certainly not for everybody, and they were never intended to solve all of the problems plaguing the criminal-justice system.</p>
<p>In New Jersey, with all major stakeholders having a voice at the table, the judiciary, law enforcement, the defense bar, and the addiction-services community worked diligently to create a successful model. Nonviolent offenders clinically addicted to alcohol and drugs are given an opportunity to receive effective treatment.</p>
<p>The New Jersey Office of the Public Defender represents more than 90 percent of drug court participants, undermining the claim that drug courts favor a more privileged socioeconomic group. Of the 8,004 people who, with the advice of lawyers at their sides, participated in New Jersey&#8217;s drug-court program, 1,577 successfully graduated. While 61 percent of those entering the program complete it, the employment rate at the time of graduation is 90 percent and the percentage of negative drug tests is 96 percent. Within three years of graduating, only 3 percent return to prison for a new crime, compared with a 60 percent rate of recidivism for inmates who do not receive treatment.</p>
<p>Although there are serious concerns raised by the National Association of Criminal Defense Lawyers that need attention, we should not be dismayed nor distracted. Funding should continue for easily accessible substance-abuse education, prevention, and treatment. As a community, we all benefit each and every time a person triumphs over his addiction to alcohol or other drugs and becomes a law-abiding, tax-paying citizen. Who needs drug court? We all do.</p>
<p>Los Angeles Daily Journal<br />
October 22, 2009</p>
<p>Drug Courts Are the Most Sensible and Proven Alternative to Incarceration: So What’s the Problem?<br />
by West Huddleston, Chief Executive Officer, National Association of Drug Court Professionals</p>
<p>The National Association of Criminal Defense Lawyers recently released a report criticizing 2,100 (there are actually 2,369) Drug Courts that offer effective treatment instead of incarceration for drug addicted offenders. Instead, the NACDL calls for the decriminalization of highly addictive drugs such as methamphetamine, heroin and crack cocaine as the solution to the drug problem. According to Cynthia Orr, President of the NACDL, “Drug Courts have not stymied the rise in both drug abuse or exponentially increasing prison costs to taxpayers” because, according to the NACDL report, “Drug Courts focus on first-time or nonviolent offenders.” The evidence says differently.</p>
<p>It is now 20 years since the first Drug Court was initiated and there has been more research published on its effects than on virtually all other criminal justice programs combined. The scientific community has put Drug Courts under a microscope and concluded that Drug Courts work better than jail or prison, better than probation, and better than treatment alone. Most medications have less scientific evidence supporting their safety and benefit to the public. The research is unequivocal: Drug Courts significantly reduce drug abuse and crime and do so at less expense than any other justice strategy; and according to rigorous and replicated studies conducted by the University of Pennsylvania, the more serious the offender’s drug addiction and length of criminal record, the better Drug Courts work. Drug Courts are not for the fist time or the non-addicted offender.  Those individuals will do just as well by diverting them to a disposition that leads to record expungement upon successful completion of court conditions.  Drug Courts focus on high-value offenders; those who have the highest need for treatment and other wrap-around services, and who have the highest risk of failing out of those services without support and structure.  </p>
<p>Research demonstrates that nationwide, 70% of the approximately 120,000 seriously addicted individuals who voluntarily enter Drug Court with the assistance of their defense attorney complete it a year or more later and 75% of them remain arrest-free.  A Drug Court participant is over twice as likely to stay clean and remain arrest-free as a newly released state inmate.  Research also concludes that Drug Courts reduce drug abuse and improve employment and family functioning.   These effects are not short-lived.  The longest study on Drug Court to date shows these outcomes last as much as14 years.  And more research is coming out every day.  </p>
<p>Still, no one would argue that Drug Courts have realized their full potential. Drug Courts have not been made available to everyone who needs them.  Half of U.S. counties do not have a Drug Court and the Drug Courts that do exist only have capacity to serve 10% of the serious drug-abusing and addicted offenders estimated to be in need.   That’s the real issue.  </p>
<p>New York has implemented a Drug Court in every county in the state.  In a three year study, the New York State Court System estimates that $254 million in incarceration costs were saved by diverting 18,000 drug offenders into Drug Court. During the entire fifteen-year time period Drug Courts have been in operation throughout the state, New York has witnessed historic reductions in crime.  And through the first half of this year, crime has fallen another 4.7 percent.  According to a recent Northwestern University report, alternatives to incarceration like Drug Courts could lead to the closing of four half-empty adult prisons in New York. And a number of states such as Alabama, Missouri, New Jersey and Texas, among others, are following suit. In fact, in 2008, 44 state budgets included a specific appropriation for Drug Courts, totaling $208,000,000 nationwide.  The Obama Administration and Congress is also investing in new Drug Courts and increasing the capacity of the 2,369 Drug Court already in existence in all fifty states and U.S. territories with a 250% increase in federal appropriations from the year before.  That’s a great start, but far from what we need to reach the 1.2 million seriously drug abusing or addicted offenders who need treatment.  </p>
<p>If no other sentencing option can compare with its success, shouldn’t we finish the job and give everyone who needs it access to these life-saving courts?  It’s simple really. Drug Courts remain constrained by limited resources and by the more popular thinking that an alcoholic or addict can be punished out of their dependence.</p>
<p>It is no secret that prison has accomplished little to stem the tide of crime or drug abuse.  Upon their release from prison, between 60% and 80% of drug abusers commit a new crime (typically a drug-related crime)  and 85% to 95% relapse quickly to drug abuse.   In some states, such as California, more than 75% will be returned to prison. And amazingly, these disappointing figures have done little to curb prison spending.  National expenditures on corrections well exceed $60 billion annually.   On average, states spend $65,000 per bed, per year to build new prisons and $23,876 per bed, per year to operate them</p>
<p>Unfortunately, it is also not sufficient to simply offer more treatment.  Left to their own devices without intensive supervision by a judge, approximately 25% of offenders never arrive for a single treatment session. And among those who do show for treatment, most drop out prematurely before receiving any benefits.  The power and authority of the Court is necessary to keep them engaged in treatment long enough to experience any lasting gains.</p>
<p>Drug Courts are judicially supervised court dockets that strike the proper balance between the need to help addicted offenders get free from the gasp of drugs and the need to protect community safety; between the need for effective treatment and the need to hold people accountable for their actions; between hope and redemption on the one hand and productive citizenship on the other. Drug Courts keep drug-addicted individuals engaged in treatment for long periods of time, while supervising them closely and holding them accountable for their obligations to society, their families and themselves. Participants are regularly and randomly tested for drug use, required to appear frequently in court for the judge to review their progress, and immediately receive rewards for doing well and sanctions for not living up to their obligations. All of this with one simple goal; get the addict clean and sober.  </p>
<p>And everybody benefits when an addict gets clean and sober in Drug Court. The most conservative estimates by researchers show that for every 1.00 invested in Drug Court, $3.36 are saved by the justice system and up to $12.00 (per $1 investment) are saved by the community on reduced emergency room visits and other medical care, foster care, and property loss.  </p>
<p>In Drug Court, we have an effective intervention that is not being fully implemented. Now is not the time to change course. It is our hope that a drug-addicted citizen should not need to be arrested in order to receive the help they require. But for the 1.2 million drug-addicted arrestees currently involved in the adult criminal justice system, the verdict is in: Drug Court is the solution and the passport to a new way of life. Now we must make the investment and finish the job.</p>
<p><em>Source: http://www.nadcp.org/setting-the-record-straight   2010<br />
</em><br />
</span></p>
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		<title>Can These Leopards Change Their Spots?</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/can-these-leopards-change-their-spots/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/can-these-leopards-change-their-spots/#comments</comments>
		<pubDate>Fri, 04 Nov 2011 13:17:47 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Political Sector (Drug Politics)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7884</guid>
		<description><![CDATA[RESPONSE TO THE NTA BUSINESS PLAN 2010/2011 Deirdre Boyd, CEO of the Addiction Recovery Foundation Kathy Gyngell, chair of the Centre for Policy Studies&#8217; Addictions working group With the threat of abolition hanging over its head, the National Treatment Agency has cleverly extended its longevity by promising to mend its ways. It will, it announced [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;">RESPONSE TO THE NTA BUSINESS PLAN 2010/2011</p>
<p>Deirdre Boyd, CEO of the Addiction Recovery Foundation<br />
Kathy Gyngell, chair of the Centre for Policy Studies&#8217; Addictions working group</p>
<p>With the  threat of abolition  hanging over its head, the  National Treatment Agency has cleverly extended its longevity by promising to mend its ways. It will, it announced on Friday, use the final two years of  its now-extended life to change the policy it has promulgated over the past nine years.<br />
“We’ve got to get rid of the centralised bureaucracy that wasteS money and undermines morale,” prime minister David Cameron stated in July.  But the NTA would seem to have got the last laugh, with over £42.8million of taxpayer‘s money now  allocated to it for two more years to change the disastrous system it created and has so steadfastly defended even in face of the indefensible.<br />
The NTA will, it promises, help people get off the methadone dependency, tier 2/3 organisation dependency and state dependency which it created via its performance-managed targets.  Its new  Business Plan 2010/11, in a truly Orwellian &#8220;four legs bad, two legs good&#8221; style, now seemingly advocates the very abstinence approach its spokespeople have repeatedly declared to be unviable.<br />
It will even consult rehabs, the NTA  graciously announced – those very rehabs it has ignored for almost a decade and of whose success in getting addicts into drug-free and rewarding recovery Paul Hayes (yes, still the NTA’s CEO) has publicly belittled, scorned or downright denied.  Could it be less than two years ago that  the NTA’s ‘first point-of-contact’ told BBC Home Affairs editor Mark Easton that “rehab doesn’t work”?  (see Comment 5th from bottom here for more derogatory comments from NTA senior managers).<br />
But maybe this was not such a hard promise for the NTA top brass to make, as they look forward to their ‘brobdingnagian’ pension pots in two years’ time. After all, there are fewer rehabs to consult&#8230; For under the NTA regime, only 2-4% of addicts seeking help to quit drugs were actually referred to them. The result?  Financial hardship, redundancies, the closure of over 20 specialist rehabs, more wing and bed closures and a loss of the real expertise required to rehabilitate addicts. And with their own personal futures well secured, would success of change be in their interest?<br />
There isn’t any evidence for abstinence or for rehab, they have repeated declared. This is despite two national treatment outcomes surveys &#8211; Ntors and Doris &#8211; which indicate  strongly to the contrary. It is also in face of experience. As Sir Ian Gilmore said yesterday,  the &#8220;absence of evidence&#8221; about school milk for under fives is only that; it does not mean that it is not a good thing and has not helped children’s health. All experience suggests it certainly has, he insisted. Similarly with rehab: a joint report in 2008 by the Commission for Social Care Inspectorate with the NTA itself that “residential rehabs outstrip other sectors in every outcome group we measure&#8221;.<br />
The NTA seems to have bamboozled the Department of Health and a too readily believing government.  For who have they tasked to change their policy and now shift people into ‘recovery’?  Brazenly, it has appointed as one of the duo the addiction psychiatrist most closely associated with the failed medico-clinical treatment approach of the past 20 years years, one of the the proponents and instigators of the last government&#8217;s failed treating-drugs-with-drugs approach so loved by the NTA, key lobbyist for counterintuitive, expensive and ethically questionable prescribing programmes: John Strang of the National Addiction Centre.<br />
In his capacity as a director of the UKDPC &#8211; recipient of millions of charitable funds to, among other briefs, redefine for the nation the notion of (addiction) recovery &#8211; Strang chose to use this remit to ensure that any new official definition of recovery excluded full abstinence, ignoring all expert advice to the contrary.<br />
Nor did he stop there. His UKDPC&#8217;s plan was to use this new definition of “recovery” to replace real total drug-free outcomes as the measure for the NTA’s Treatment Outomes Profile forms, meaning that their  targets could be easily be hit. Very convenient. For, in one Orwellian sleight of hand, the NTA could claim a recovery outcome when no such thing had been achieved. A reduction in injection frequency would suffice. This would be the basis of NTA’s (aspirationless) claims of treatment success. In face of the derision this deserved, the  NTA has gone on record saying it does not define recovery at all now – despite the fact that “recovery” is the raison d’etre of its Janus-faced Business Plan 2010/2011. That all the goals and actions therein are meaningless can thus be taken as read.<br />
For example, there is apparently no plan to replace the discredited and bureaucratically heavy Top form. It will be forced on ever more people. The NTA states, too, that it has looked at the ASAM patient placement criteria. Yet instead of contacting the creators of this highly-researched method, it plans to reinvent the wheel and spend taxpayer money developing a version for its own purposes. It also plans to spend more taxpayer money on a mutual aid directory. Yet this is already provided free by Addiction Today. Under Championing abstinence-focussed treatment in the business plan&#8230; well, for further help interpreting  the Business Plan’s double speak, read our glossary.<br />
It is, however, commendable that Dr David Best, who has wriiten so cogently and expertly on abstinence-based recovery in the pages of Addiction Today and other professional journals, has been appointed as the other half of the recovery duo.  We wish him the very  best of luck in counterbalancing his former mentors, and getting them on the true road to recovery with a Damascene conversion. They should heed him, for he is the only person giving this exercise any credibility.<br />
As David Cameron said in June,“There is a problem in our national health service, in that we spend too much time treating the symptoms rather than necessarily dealing with the causes&#8230; All addictions need proper attention, and proper treatment and therapy, to rid people of their addictions”.<br />
We really would love to believe, as he and many in government must wish to believe, that we will witness the NTA&#8217;s respecting the trust that has been placed in it and seeking the rehab expertise that actually helps people to get off life-destroying drugs and rebuild their lives and their families’ lives. But the serious worry is that this initiative for change get will be lost in adherence to disinformation and blowback, and submerged in intransigent ideology about the non-recoverability from addiction. Of even more concern, will its lack of understanding continue to marginalise the expertise necessary to help the 330,000 or so addicts desperate for the sobriety which is the basis for them to get back, or get for the first time, their self esteem and their lives?<br />
We will be happy to be proved wrong. But we are not holding our breath.</p>
<p><em>Source: www.addictiontoday.org.   10th August 2010<br />
</em><br />
</span></p>
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		<title>Methadone is linked to one in three drug deaths</title>
		<link>http://drugprevent.org.uk/ppp/2011/11/methadone-is-linked-to-one-in-three-drug-deaths/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/11/methadone-is-linked-to-one-in-three-drug-deaths/#comments</comments>
		<pubDate>Fri, 04 Nov 2011 13:14:12 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Drug Specifics (Drug Politics)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7882</guid>
		<description><![CDATA[CALLS have been made for a rethink on the use of methadone in Scotland after official figures revealed the number of deaths in which it was implicated reached a ten-year high last year. Amid a general fall in people being killed by drugs, fatalities in which the heroin substitute was cited as a contributory factor [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;">CALLS have been made for a rethink on the use of methadone in Scotland after official figures revealed the number of deaths in which it was implicated reached a ten-year high last year.<br />
Amid a general fall in people being killed by drugs, fatalities in which the heroin substitute was cited as a contributory factor rose to 173 in 2009, up from 169 in 2008 and a surge of 51 per cent since 2007 when it was associated with 114 deaths.  The controversial drug treatment was found to be at least partly responsible for more than a third (32 per cent) of all of the 545 drug-related fatalities in Scotland last year, and was associated with the second-highest number of drug-addict deaths after heroin or morphine, which contributed to 322 losses of life &#8211; 59 per cent of the total.</p>
<p>The 2009 methadone figure also equates to roughly one death every 48 hours.</p>
<p>The rising number of deaths linked to methadone led to calls for the policy of wide prescription of the treatment to addicts to be reviewed, with one drug-misuse expert describing the current situation as being of &#8220;enormous concern&#8221;.</p>
<p>Professor Neil McKeganey, the director of the Centre for Drug Misuse Research at Glasgow University, said: &#8220;The situation in relation to methadone &#8211; where it appears we have around a third of addict deaths associated with the drug we are prescribing most widely to treat drug addiction &#8211; is of enormous concern.   We really ought to be looking again at this policy of widespread methadone prescribing. The statistics are inescapable &#8211; we ought to be looking at why we are doing it and whether all of those to whom it is being prescribed are deriving benefit from it.&#8221;</p>
<p>Peter McCann, the chairman of the Castle Craig Hospital for alcoholism and drug addiction, lent his weight to the calls, adding: &#8220;Today&#8217;s drug-death figures would have been described as totally catastrophic just a few years ago. There must now be a total rethink in Scotland along the lines of the National Treatment Agency in England which totally reversed its policy earlier this month.   &#8220;They will be limiting the use of methadone with strict multi-disciplinary assessments at regular intervals. The policies prescribing methadone in Scotland have obviously failed and must be revised.&#8221;</p>
<p>Murdo Fraser, Scottish Conservative health spokesman, said the focus of the Scottish drugs strategy should be on recovery and abstinence.  He said: &#8220;The attempts of the last decade to merely manage the problem, based on harm reduction and an over-reliance on methadone, just have not worked.  The challenge now is to expand the range of rehabilitation services on offer and move to abstinence and recovery.&#8221; </p>
<p>But the treatment was defended by Biba Brand of the Scottish Drugs Forum: &#8220;We know from research that staying on methadone tends to prolong their life by about 13 per cent.  &#8220;We also know that of those deaths that are occurring (overall], two-thirds are outwith treatment, so being in treatment &#8211; and generally that involves methadone &#8211; is helping people stay alive. Methadone can help save lives, but we need to help people progress through treatment.&#8221;</p>
<p>A Scottish Government spokesman added: &#8220;We do not favour one form of treatment over any other. Decisions on the most appropriate treatment to prescribe an individual are for clinicians, in discussion with their patients and in line with national guidelines.&#8221; </p>
<p>Overall, the number of people killed by drugs in Scotland fell by 5 per cent since 2008, but the 545 drug-related deaths during 2009 equated to the second-highest total ever recorded; an increase of 20 per cent since 2007 and a rise of 87 per cent since 1999.</p>
<p>A wider analysis, using figures recorded by the Office for National Statistics, showed the number of deaths related to drugs in Scotland last year was 716, down from 737 in 2008, but a rise from the 2007 total of 630. </p>
<p>This figure included people killed by solvent abuse, legal highs and through overdoses of prescription medication. It also included people dying with mental-health problems linked to drug abuse, as well as those killed by the health complications allied to contaminated drugs.  More than a third of all deaths in Scotland, some 200, were in the Greater Glasgow and Clyde NHS board area, and this represented the highest total on record. Deaths in Lothian dropped, by 13 to 81, as did fatalities in Fife (37 to 32) and Forth Valley (23 to 14). </p>
<p>There was also a rise in the number of older people dying from drugs, with deaths among those aged 35 and over rising from 271 in 2008 to 296 in 2009, while at the same time deaths among users under 35 dropped from 303 to 249.</p>
<p><em>Source: News.Scotsman.com  18th Augutst 2010<br />
</em></p>
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		<title>Planning Commission to consider ban on medical marijuana dispensaries</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/planning-commission-to-consider-ban-on-medical-marijuana-dispensaries/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/planning-commission-to-consider-ban-on-medical-marijuana-dispensaries/#comments</comments>
		<pubDate>Fri, 07 Oct 2011 11:03:23 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Political Sector (Drug Politics)]]></category>
		<category><![CDATA[USA]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7869</guid>
		<description><![CDATA[by Eric Pierce The Planning Commission will consider on Wednesday asking the City Council to revise the city charter to permanently bar medical marijuana dispensaries from operating in Downey. Citing federal law that still makes it a crime to grow, use or possess marijuana, city administrators recommend the charter be amended to prohibit the dispensaries. [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;"></p>
<p>by Eric Pierce </p>
<p>The Planning Commission will consider on Wednesday asking the City Council to revise the city charter to permanently bar medical marijuana dispensaries from operating in Downey.<br />
Citing federal law that still makes it a crime to grow, use or possess marijuana, city administrators recommend the charter be amended to prohibit the dispensaries. </p>
<p>The City Council last year enacted a moratorium on medical marijuana clinics that is scheduled to expire Nov. 10.</p>
<p>In a report prepared by community development director Brian Saeki and senior planner David Blumenthal, city officials also cited reports of violent crime &#8212; specifically robberies and homicides &#8212; at dispensaries in neighboring cities. </p>
<p>“Besides crimes against persons and property, the operation of medical marijuana dispensaries has been linked to organized criminal activity, money laundering and firearm violations,” the report states.</p>
<p>California voters approved the use of marijuana for medicinal purposes in 1996. The state created a voluntary medical marijuana identification card program in 2003 to protect residents from state marijuana laws.   The San Diego Union-Tribune reported in June that of California’s 481 incorporated cities, 132 have banned medical marijuana dispensaries. Another 101 have enacted temporary moratoriums.</p>
<p>Best, Best &#038; Krieger, before they were fired as the city’s law firm, wrote a whitepaper suggesting Downey had the discretion to either regulate or prohibit medical marijuana clinics. The law firm also warned the city against “adverse secondary impacts” dispensaries could pose.   “On balance, any utility to medical marijuana patients in care giving and convenience that marijuana dispensaries may appear to have on the surface is enormously outweighed by a much darker reality that is punctuated by the many adverse secondary effects created by their presence in communities,” Best, Best &#038; Krieger wrote. “These drug distribution centers have even proven to be unsafe for their own proprietors.” </p>
<p>The city of Los Angeles recently approved a restrictive ordinance aimed at corralling the city’s estimated 400 medical marijuana dispensaries. Attorneys representing marijuana dispensaries given shut-down notices have said they will sue Los Angeles to remain open.</p>
<p>Only one medical marijuana dispensary has operated legally in Downey. It closed after the city&#8217;s moratorium went into effect late last year.</p>
<p><em>Source: www.thedowneypatriot.com  31st Aug.2010<br />
</em><br />
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		<title>Drug seizures almost treble at city prison</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/drug-seizures-almost-treble-at-city-prison/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/drug-seizures-almost-treble-at-city-prison/#comments</comments>
		<pubDate>Fri, 07 Oct 2011 11:01:39 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Effects of Drugs (Drug Politics)]]></category>
		<category><![CDATA[Legal Sector (Drug Politics)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7867</guid>
		<description><![CDATA[Scottish Government figures show 168% increase at Craiginches since 2007 Drug seizures at Craiginches Prison in Aberdeen have nearly trebled in the last three years. Scottish Government figures show there were 134 seizures at the jail last year, a 168% increase since 2007 when there were 50. The increase was far higher than the total [...]]]></description>
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<p>Scottish Government figures show 168% increase at Craiginches since 2007<br />
Drug seizures at Craiginches Prison in Aberdeen have nearly trebled in the last three years.<br />
Scottish Government figures show there were 134 seizures at the jail last year, a 168% increase since 2007 when there were 50.  The increase was far higher than the total across Scotland where drug seizures went up by 12% from 1,626 to 1,829 over the same period.<br />
Labour called for a redoubling of efforts to rid Scotland’s jails of drugs. Yesterday, Chief Inspector of Prisons Brigadier Hugh Munro warned that drug testing needed to be tightened up because addiction programmes were rendered pointless by ineffective testing regimes.<br />
The only other prison with a similar number of drug seizures in the north-east was Perth where the number has remained relatively static with an average of 138 over three years.<br />
At Inverness Prison seizures were up from 11 to 19. The number at the two open prisons, Castle Huntly and Noranside, in Tayside, fell from 63 to 53, as did those at Peterhead, down from six to one.<br />
North-east MSP and Labour justice spokesman Richard Baker said: “Drugs are far too prevalent in Scotland’s prisons and Brig Munro is quite right to say more needs to be done.   “With a rising tide of drugs getting into our prisons there is a need to redouble our efforts to rid our prisons of drugs.”<br />
The Scottish Prison Service (SPS) said increased seizures were a sign that efforts to reduce drug taking and smuggling into jails were working.   An SPS spokesman said money had been invested in new technology such as mobile drug tracing and X-ray machines, and the “most effective deterrent” – sniffer dogs.<br />
“New legislation will also tackle the issue of mobile phones which are a key element in drug trafficking in prisons,” he said.  “High levels of finds, such as those at HMP Aberdeen which doubled in two years, are an indicator of success.”<br />
The Tories released figures showing a 37% increase in the number of prisoners receiving the heroin substitute methadone. A snapshot of one day showed the number on the drug went up from 1,228 in 2006 to 1,679 this year. The percentage of the prison population on methadone went up from 17.1% to 21.5%.<br />
Tory justice spokesman John Lamont said: “This is extremely worrying. This rise in prisoners in receipt of methadone suggests that efforts to move drug addicts towards abstinence are not working properly.”<br />
A Scottish Government spokesman said the percentage of prisoners prescribed methadone had risen by less than 3% since the current SNP administration came into office in 2007.  “Getting people into treatment is the most effective way of reducing drug use and breaking the links between drugs and crime,” he said.  “Methadone has a role to play among a range of treatments and support available to help people recover from their drug problems.”<br />
The SPS said 85% of prisoners on methadone were continuing medication prescribed before they were sentenced while 15% were on new prescriptions initiated in custody.  “According to the latest prisoner survey in 2009, almost a quarter of prisoners are currently on a reducing methadone dose as part of their recovery programme,” a spokesman said.</p>
<p><em>Source: www.pressandjournal.co.uk  3rd Sept. 2010<br />
</em><br />
</span></p>
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		<title>Marijuana and Youth – Experiences From a Practising Physician</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/marijuana-and-youth-%e2%80%93-experiences-from-a-practising-physician/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/marijuana-and-youth-%e2%80%93-experiences-from-a-practising-physician/#comments</comments>
		<pubDate>Fri, 07 Oct 2011 10:59:50 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Drug Specifics (Drug Politics)]]></category>
		<category><![CDATA[Political Sector (Drug Politics)]]></category>
		<category><![CDATA[USA]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7864</guid>
		<description><![CDATA[The impact medical marijuana has had on our adolescent substance-abuse treatment program in Denver is profound. The 2009 boom in marijuana distribution coincides with a tripling of teens referred to our program. Currently, 51 percent of our patients report getting their marijuana from someone with a medical marijuana license. Not surprisingly, patient attitudes about marijuana [...]]]></description>
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<p>The impact medical marijuana has had on our adolescent substance-abuse treatment program in Denver is profound.</p>
<p>The 2009 boom in marijuana distribution coincides with a tripling of teens referred to our program. Currently, 51 percent of our patients report getting their marijuana from someone with a medical marijuana license.</p>
<p>Not surprisingly, patient attitudes about marijuana are changing – and in ways that make it much more difficult for us to help them stop using the drug. Recently, a teenage boy said he couldn&#8217;t stop smoking marijuana because &#8220;it is my medicine for anger.&#8221; </p>
<p>Even worse, a few young adult patients in treatment for marijuana addiction have marijuana licenses. These patients struggle with conflicting messages from one physician who recommends smoking marijuana and another who recommends stopping.</p>
<p>In Denver, marijuana is advertised on billboards and in magazines and newspapers using themes that appeal to young people. Because youth are highly vulnerable to both the effects of advertising and the addictive potential of marijuana, it is not surprising that 60 percent of the state&#8217;s medical marijuana users are under 44 years old. </p>
<p>We must act swiftly to prevent situations such as this from getting worse.<br />
Christian Thurstone, M.D. is the Medical Director of Adolescent Substance Treatment, Education and Prevention at Denver Health and Hospital Authority and Assistant Professor, Department of Psychiatry, University of Colorado Denver.</p>
<p><em>Source:  http://ofsubstance.gov/cs/blogs  Wednesday, October 13, 2010<br />
</em><br />
</span></p>
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		<title>New habits for old</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/new-habits-for-old/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/new-habits-for-old/#comments</comments>
		<pubDate>Fri, 07 Oct 2011 10:56:45 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Drug Specifics (Drug Politics)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7862</guid>
		<description><![CDATA[The extension of “payment by results” to the treatment of drug addicts will test the method’s limits AT PHOENIX FUTURES in Birmingham, Karen is six weeks into a programme of group therapy sessions, life-skills training and one-on-one meetings with her keyworker, Dean. Things are looking pretty good. A former heroin user, she was on methadone [...]]]></description>
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<p>The extension of “payment by results” to the treatment of drug addicts will test the method’s limits </p>
<p>AT PHOENIX FUTURES in Birmingham, Karen is six weeks into a programme of group therapy sessions, life-skills training and one-on-one meetings with her keyworker, Dean. Things are looking pretty good. A former heroin user, she was on methadone for years before going into residential rehab last October. Karen now takes a relatively low dose of Subutex, a weaker heroin substitute, which she intends to come off altogether over the next six weeks. She credits her treatment with giving her the stability to have her three-year-old son to stay with her at weekends, and hopes to take a course or get a job—and eventually to work with drug users—once she is fully abstinent.<br />
There are many Karens in Britain, though most are not doing as well as she is. Around 320,000 people are thought to be on heroin or crack cocaine or both in England alone. Many more use cannabis (the most popular drug), powder cocaine or a constantly changing clutch of designer drugs and legal highs: in all, almost 3m in England and Wales used some sort of illegal drug in 2009-10. A big push by the previous Labour government lifted the numbers in treatment (see chart), and drug use seems to be falling a bit now. But it remains high by European standards. Some argue that too many users have been “parked” on methadone rather than encouraged to kick chemical dependence altogether.<br />
Intent on remedying what the Conservatives see as the persistent ills of “broken Britain”, the Tory-led coalition government has big ambitions in drugs policy. It wants to get more people through treatment and functioning again—free of drugs if possible, but also employed, housed and law-abiding. There is a moral dimension to its emphasis on recovery rather than harm reduction, but also an economic one. Use of heroin or crack cocaine is linked to between a third and a half of all acquisitive crimes; an estimated 400,000 benefit claimants who are dependent on drugs or drink cost the Treasury £1.6 billion a year; and demands on the health service and criminal-justice system are great. The coalition’s commitment is real: at a time of screaming budget cuts, central-government funds for drug treatment in communities and prisons have barely been hit.<br />
A key plank of the strategy is “payment by results”. This approach to delivering public services—rewarding charities, community groups or private firms not for what they do but for how well they do it—has been seized on gratefully by a cash-strapped government. Versions are being tried to get welfare recipients into work and discourage criminal reoffending. Now eight drug-treatment pilots are to be launched. This breaks new ground internationally, says Martin Barnes, the head of DrugScope, a drug-information charity.<br />
No magic wand<br />
The theoretical argument for payment by results is that, by rewarding only success, it drives up standards while reducing costs. “It will make organisations focus on delivering quality services because they won’t survive if they don’t,” says David Biddle, deputy chief executive of CRI, a charity whose drug and alcohol services have grown rapidly. Kent is one area chosen for a payment-by results pilot. “Commissioners will now have the opportunity to reward those who innovate, and deliver efficient and effective services,” says Amanda Honey of Kent County Council.<br />
Not everyone is sure that payment by results will work in drug treatment, however. Outcomes are hard to measure. In welfare-to-work schemes, a claimant either gets and holds a job or he doesn’t. A prisoner is convicted of reoffending or he isn’t. With drugs, progress often consists of baby steps on various fronts, which is why the government proposes to pay for a range of positive outcomes including jobs, housing and so forth. Coming up with precise measures is proving hard.<br />
Setting tariffs is difficult, too. Payment by results works only if risk is transferred to the provider. But drug users are prone to relapses, and recovery can take years. Most not-for-profits in drug treatment are small; they need payment along the way to cover their costs. If instead they become subcontractors to larger outfits, a one-size-fits-all approach could replace the tailored solutions seen by many as a key to success. Whoever is contracted, “if the basic tariff isn’t enough, it will wipe out the chances of the provider doing anything good. If it’s too much, then there is no risk transfer. If it’s the wrong mix [of incentives], then it encourages gaming,” says Lord Adebowale, the chief executive of Turning Point, a health and social-care organisation.<br />
Despite the challenges, Turning Point and CRI are both interested in the trials. For its part, smaller Phoenix Futures has started offering a payment-by-results option off its own bat. “I wanted us to be ready,” says Karen Biggs, its chief executive.<br />
But other uncertainties loom, as more administrative power is pushed down from Whitehall. From 2013 the funds earmarked by central government for drug treatment in the community (currently about £500m) will be handed over by Public Health England, a new bit of the NHS, to local authorities; drug and alcohol funding will merge, and perhaps disappear into the overall public-health pot. New elected police and crime commissioners will have a say in this area, as might local GPs newly charged with commissioning health care.<br />
Anne Milton, the minister for public health, is determined that money will not leak away from drug treatment, counting on a national “outcomes framework” to make sure that needs which are not regarded as a priority locally continue to be met. Payment by results in this complicated and difficult area might prove transformative in all the right ways—or it might turn out an unholy mess. If it does work, says one sceptical charity, “they can use payment by results to deliver absolutely anything.”</p>
<p><em>Source: www.economist.com 14th April 2011<br />
</em><br />
</span></p>
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		<title>Letter &#8211; Portugal is hardly a resounding success</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/letter-portugal-is-hardly-a-resounding-success/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/letter-portugal-is-hardly-a-resounding-success/#comments</comments>
		<pubDate>Fri, 07 Oct 2011 10:54:27 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Europe]]></category>
		<category><![CDATA[Legal Sector (Drug Politics)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7860</guid>
		<description><![CDATA[Letter published in The Times April 25th 2011 Sir, Portugal is hardly the resounding success that the drugs liberalising lobby would have us believe That fewer young people are trying drugs in Portugal may be the case (“Radical drug law could be imported to Britain”, April 22). But this simply reflects a Europe-wide trend, nowhere [...]]]></description>
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<p>Letter published in The Times April 25th 2011<br />
Sir,<br />
Portugal is hardly the resounding success that the drugs liberalising lobby would have us believe<br />
 That fewer young people are trying drugs in Portugal may be the case (“Radical drug law could be imported to Britain”, April 22). But this simply reflects a Europe-wide trend, nowhere more evident than in the United Kingdom. The alarming Europe-wide increase in young people’s illicit drug use between 1995 and 2003 has come to a halt and is decreasing — in Portugal by rather less than the European average.<br />
The picture painted by your report is less rosy overall when the data is examined fully. For according to Portugal’s Special Registry of the National Institute of Forensic Medicine, there has actually been an increase in Portugal’s drug-related deaths since decriminalisation was enacted, from 280 in 2001 to 314 in 2007. In well over half of these cases, opiates or opiates in combination with other substances (mainly cocaine or alcohol) were cited as the main substance involved.<br />
Furthermore Portugal has been the only European country to show a significant increase in [drug-related] homicides between 2001 and 2006, by 40 per cent over a five-year period (2009 UNODC World Drug Report).<br />
Finally, Portugal’s Instituto da Droga e da Toxicodependência reports that the overall prevalence of life time drug use increased from 7.8 per cent to 12.0 per cent in the period from 2001 to 2007, cocaine more than doubling and ecstasy close to doubling, with the prevalence of heroin abuse up from 0.7 per cent of the adult population to 1.1 per cent in the same period.<br />
As to the decreases in new cases of HIV/Aids, not only is this also in line with a Western European trend but it is just as, if not more, plausible to associate this with Portugal’s annual increases in funding for treatment, detox and harm reduction than with the act of decriminalisation per se.<br />
Portugal is hardly the resounding success that the drugs liberalising lobby would have us believe. And if it is what they are relying on to convert politicians and public to their cause it makes for a poor case.<br />
Kathy Gyngell<br />
Research Fellow, Centre for Policy Studies<br />
Neil McKeganey<br />
Professor of Drug Misuse, Centre for Drug Misuse Research, University of Glasgow<br />
Mary Brett<br />
Trustee, Cannabis Skunk Sense </p>
<p><em>Source:  http://www.thetimes.co.uk/tto/opinion/letters/article2997948.ece  25.4.2011<br />
</em> </p>
<p></span></p>
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		<title>Radio 4 Any Questions &#8211; Drug Police Debate</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/radio-4-any-questions-drug-police-debate-2/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/radio-4-any-questions-drug-police-debate-2/#comments</comments>
		<pubDate>Fri, 07 Oct 2011 10:51:31 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Drug Specifics (Drug Politics)]]></category>
		<category><![CDATA[Europe]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7858</guid>
		<description><![CDATA[BBC Radio 4’s Any Questions: The drug policy debate in early June mentioned an organisation in which I am involved – the UK National Drug Prevention Alliance – many times, so we must respond. In doing so, I hope to convey proven facts about the dangers of legalising drugs. Nadine Dorries was correct that much [...]]]></description>
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<p>BBC Radio 4’s Any Questions: The drug policy debate in early June mentioned an organisation in which I am involved – the UK National Drug Prevention Alliance – many times, so we must respond. In doing so, I hope to convey proven facts about the dangers of legalising drugs.</p>
<p>Nadine Dorries was correct that much modern cannabis is stronger than years ago but we do not agree with her figures. Typically, modern cannabis is three to four times stronger in THC, the psychoactive ingredient, than even the strongest cannabis of the 1960s and 1970s. This has been achieved by selective breeding and in response to consumer demand.</p>
<p>But the picture is more complex than ‘just’ THC strength. The presence – or rather absence in modern forms – of another chemical, CBD, appears to have aggravated the brain-damaging potential of cannabis. Use has also changed. Age of first use and regular use is earlier than in the 1960s and that is another damaging factor. The evidence caused the UK government, with cross-party agreement, to reclassify cannabis upwards two years ago. At the time (Sky News, 6 April 2008), prime minister David Cameron admitted that a parliamentary committee, of which he had been a member, had been wrong about lowering the classification of cannabis. Lessons have been learned and are unlikely to be overturned.  Cannabis contributes substantially to academic under-achievement and very poor mental health, regardless of other effects.</p>
<p>On the wider question of decriminalisation and even legalisation of all drugs, the NDPA believes that a monstrous, well financed and very slick fraud is being perpetrated internationally and that this fraud has fooled some of the “great and good” who signed up as supporters. There is no evidence at all that either measure could reduce the total harm from drugs. The reverse is very much the case, with academic opinion saying that either measure would inevitably normalise and increase, use.  The manifest harm from the legal drugs and the legislation on alcohol and tobacco, as variously applied around the world, confirms that.  Comments on wishful good effects from decriminalisation were profoundly incorrect and reflect manipulative messages. For years, we have been bombarded with the Netherlands as the example of sound drug policy – despite the fact that the country, through its policies, created the largest base for drugs-related criminality in Europe with supply, warehousing, distribution and manufacture at astonishing levels. At one stage, the Netherlands had more drug related murder than anywhere else in Europe. The Netherlands is changing. It spends proportionally more than the UK on enforcement and is currently more effective and better organised than the UK.</p>
<p>Portugal and decriminalisation appears now to be “the new orthodoxy” for those with a certain direction of travel and for those “user advocates” who want more freedom to use, regardless of the wider social effects. But Portugal is being misrepresented, as demonstrated below.</p>
<p>1.	The number of new cases of HIV and Hepatitis C in Portugal is eight times the average in other EU countries.<br />
2.	Portugal has the most cases of injected drug related Aids, with 85 new cases per million citizens.  Other EU countries average 5 per million.<br />
3.	Since decriminalisation, drug-related homicides have increased 40%.<br />
4.	Drug overdoses have increased substantially, by over 30% in 2005.<br />
5.	There has been an increase of 45% in post mortems testing positive for illegal drugs.<br />
6.	Amphetamine and cocaine consumption has doubled in Portugal, with cocaine seizures increasing sevenfold between 2001 and 2006.</p>
<p>Finally, the suggestion that legalisation would somehow remove criminality from drug supply is ridiculous. Criminality loves use-reinforcing substances and behaviours. Over 20% of the UK tobacco market is smuggled, counterfeit or both. In some other countries, the figures are worse. Legalisation or decriminalisation of substances unfit for human consumption should occur only if a demonstrable “public good” can be evidenced. The problem for the legalisation lobby is that it<br />
cannot.<br />
DAVID RAYNES is executive councillor of the<br />
UK National Drug Prevention Alliance (http:// drugprevent.org.uk/ppp/about-us).</p>
<p><em>Source: Addiction Today July/August 2011<br />
</em></p>
<p></span></p>
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		<title>Genetic Risk Factors for both Marijuana and Alcohol Misuse Similar</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/genetic-risk-factors-for-both-marijuana-and-alcohol-misuse-similar-2/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/genetic-risk-factors-for-both-marijuana-and-alcohol-misuse-similar-2/#comments</comments>
		<pubDate>Tue, 04 Oct 2011 13:51:59 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Addiction]]></category>
		<category><![CDATA[Brain and Behaviour]]></category>
		<category><![CDATA[USA]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7855</guid>
		<description><![CDATA[• Marijuana is the most commonly used illicit drug in the United States. • New research shows that the use and misuse of alcohol and marijuana are influenced by a common set of genes. Marijuana is the most commonly used illicit drug in the United States. Roughly eight to 12 percent of marijuana users are [...]]]></description>
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<p> •	Marijuana is the most commonly used illicit drug in the United States.<br />
•	New research shows that the use and misuse of alcohol and marijuana are influenced by a common set of genes.<br />
Marijuana is the most commonly used illicit drug in the United States. Roughly eight to 12 percent of marijuana users are considered “dependent” and, just like alcohol, the severity of symptoms increases with heavier use. A new study has found that use and misuse of alcohol and marijuana are influenced by a common set of genes.<br />
Results will be published in the March 2010 issue of Alcoholism: Clinical &#038; Experimental Research and are currently available at Early View.<br />
“Results from a large annual survey of high-school students show that in 2008, 41.8 percent of 12th graders reported having used marijuana,” explained Carolyn E. Sartor, a research instructor at Washington University School of Medicine and corresponding author for the study. “Although many may have used the drug on only a few occasions, 5.4 percent of 12th graders reported using it daily within the preceding month.”<br />
“The active ingredient in marijuana is THC, which mimics natural cannabinoids that the brain produces,” added Christian Hopfer, associate professor at the University of Colorado School of Medicine. “The cannabinoid system is critical for learning, memory, appetite, and pain perception. Most users of marijuana will not develop an ‘addiction’ to it, but perhaps one in 12 will. What is not commonly appreciated about marijuana use is that strong evidence has emerged that it increases the risk of developing mental illnesses and possibly exacerbates pre-existing mental illnesses.”<br />
“Like any drug, marijuana can be used in a way that negatively impacts quality of life, interfering with functioning at school or work or leading to problems with family and friends,” said Sartor. “Although at least three of six symptoms listed in the Diagnostic and Statistical Manual of Mental Disorders &#8211; Fourth Edition (DSM-IV) are needed to meet full criteria for cannabis (marijuana) dependence … the presence of even one or two of these symptoms could create distress or interfere with day-to-day functioning. There is strong evidence for a genetic component to use and dependence on marijuana as well as alcohol, and the use (and misuse) of these substances frequently occur together.”<br />
Researchers examined 6,257 individuals (2,761 complete twin pairs and 735 singletons) listed in the Australian Twin Registry, 24 to 36 years of age. Alcohol and marijuana use histories were gathered in telephone diagnostic interviews and used to derive levels of alcohol consumption, frequency of marijuana use, and DSM-IV alcohol and cannabis dependence symptoms.<br />
“Our findings indicate that … many of the same genetic factors that contribute to alcohol use also contribute to marijuana use,” said Sartor. “Likewise, alcohol dependence symptoms and cannabis dependence symptoms can be traced to some of the same genetic influences. For both alcohol and marijuana, the majority of genetic factors that contribute to use also contribute to dependence symptoms.”<br />
“In other words,” said Hopfer, “the genetic influences on drug use are not specific to individual drugs, but seem to influence a general tendency to engage in drug use. This is important to note because there is a tendency to study drugs in isolation – alcohol, tobacco, marijuana, cocaine, etc. These findings add support to the notion of common mechanisms underlying all addictions.”<br />
“The fact that very little of the environmental influences on alcohol and marijuana use, or on alcohol and cannabis dependence symptoms, could be traced to common sources indicates that there may be important distinctions between those environmental factors that influence alcohol-related outcomes and those that influence marijuana-related outcomes,” said Sartor. “Identifying alcohol- and marijuana-specific risk factors is an important next step in this line of research.”<br />
“Marijuana research is relatively sparse compared to alcohol or nicotine research,” added Hopfer. “However, if you look at reports of at least adolescents and young people using, it becomes clear that marijuana use, including daily marijuana use, is quite common and the effects of this are not well understood. The mental illness/marijuana connection has not received much press, although I think the evidence has grown substantially that marijuana is a causal risk factor for the development of mental illness.”</p>
<p><em>Source: http://www.attcnetwork.org/explore/priorityareas/science/tools/asmeDetails.asp?ID=643<br />
</em><br />
</span></p>
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		<title>Genes Help Determine Brain Response to Alcohol, Medication, NIAAA Says</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/genes-help-determine-brain-response-to-alcohol-medication-niaaa-says/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/genes-help-determine-brain-response-to-alcohol-medication-niaaa-says/#comments</comments>
		<pubDate>Tue, 04 Oct 2011 13:49:30 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Alcohol]]></category>
		<category><![CDATA[Brain and Behaviour]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7853</guid>
		<description><![CDATA[Research Summary Alcohol consumption prompts the brain to release the pleasure chemical dopamine, but genes may influence the degree to which the brain responds to drinking and &#8212; by extension &#8212; how effective medications like naltrexone are in treating alcoholism. Researchers at the National Institute on Alcohol Abuse and Alcoholism (NIAAA) found that genetic variations [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;"></p>
<p><strong>Research Summary<br />
</strong><br />
Alcohol consumption prompts the brain to release the pleasure chemical dopamine, but genes may influence the degree to which the brain responds to drinking and &#8212; by extension &#8212; how effective medications like naltrexone are in treating alcoholism.<br />
Researchers at the National Institute on Alcohol Abuse and Alcoholism (NIAAA) found that genetic variations in the mu-opioid receptor sites in the brain&#8217;s reward system seem to influence the release of the neurotransmitter dopamine and the degree of pleasure that individuals get from drinking.<br />
Researchers also found that naltrexone &#8212; a drug that works to block the release of dopamine resulting from drinking &#8212; was more effective for patients with some genetic profiles than others.<br />
&#8220;Our data strongly support a causal role of the 118G variant of the mu-opioid receptor to confer a more vigorous dopamine response to alcohol in the ventral striatum,&#8221; said NIAAA researcher Vijay A. Ramchandani, Ph.D. &#8220;The findings add further support to the notion that individuals who possess this receptor variant may experience enhanced pleasurable effects from alcohol that could increase their risk for developing alcohol abuse and dependence. It may also explain why these individuals, once addicted, benefit more from treatment with blockers of endogenous opioids.&#8221;<br />
Markus Heilig, NIAAA&#8217;s clinical director, noted that naltrexone also worked better in the early stages of alcoholism, when the body still believes it is being rewarded for drinking (&#8216;reward craving&#8217;). At a certain point, however, the brain switches to a pattern called &#8216;relief craving&#8217; &#8212; what Heilig called a &#8220;pathological pattern of anxiety&#8221; &#8212; where naltrexone isn&#8217;t nearly as helpful.<br />
The latest findings were published online in the journal Molecular Psychiatry.</p>
<p><em>Source:  Join Together  May 20, 2010<br />
</em><br />
</span></p>
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		<title>Study Finds Hospitalization Increases for Alcohol and Drug Overdoses</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/study-finds-hospitalization-increases-for-alcohol-and-drug-overdoses/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/study-finds-hospitalization-increases-for-alcohol-and-drug-overdoses/#comments</comments>
		<pubDate>Tue, 04 Oct 2011 13:46:57 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Alcohol]]></category>
		<category><![CDATA[Drug use-various effects]]></category>
		<category><![CDATA[Health]]></category>
		<category><![CDATA[USA]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7851</guid>
		<description><![CDATA[Hospitalizations for alcohol and drug overdoses &#8211; alone or in combination &#8211; increased dramatically among 18- to 24-year-olds between 1999 and 2008, according to a study by researchers at the National Institute on Alcohol Abuse and Alcoholism (NIAAA), part of the National Institutes of Health. Led by Aaron M. White, Ph.D. and Ralph W. Hingson, [...]]]></description>
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<p>Hospitalizations for alcohol and drug overdoses &#8211; alone or in combination &#8211; increased dramatically among 18- to 24-year-olds between 1999 and 2008, according to a study by researchers at the National Institute on Alcohol Abuse and Alcoholism (NIAAA), part of the National Institutes of Health. </p>
<p>Led by Aaron M. White, Ph.D. and Ralph W. Hingson, Sc.D., of NIAAA&#8217;s division of epidemiology and prevention research, the study examined hospitalization data from the Nationwide Inpatient Sample, a project of the U.S. Agency for Healthcare Research and Quality designed to approximate a 20 percent sample of U.S. community hospitals. The findings appear in the September issue of the Journal of Studies on Alcohol and Drugs. </p>
<p>Drs. White, Hingson, and their colleagues report that, over the 10-year study period, hospitalizations among 18-24-year-olds increased by 25 percent for alcohol overdoses; 56 percent for drug overdoses; and 76 percent for combined alcohol and drug overdoses.</p>
<p>&#8220;In 2008, 1 out of 3 hospitalizations for overdoses in young adults involved excessive consumption of alcohol,&#8221; noted Dr. White. &#8220;Alcohol overdoses alone caused 29,000 hospitalizations, combined alcohol and other drug overdoses caused 29,000, and drug overdoses alone caused another 114,000. The cost of these hospitalizations now exceeds $1.2 billion per year just for 18-24-year-olds.&#8221; </p>
<p>According to the authors, this is a growing problem for those outside of the 18-24 age range, as well. </p>
<p>&#8220;Among the entire population 18 and older, 1.6 million people were hospitalized for overdoses in 2008, at a cost of $15.5 billion, and half of these hospitalizations involved alcohol overdoses,&#8221; added Dr. Hingson.<br />
The current study also showed an increase of 122 percent in the rate of poisonings from prescription opioid pain medications and related narcotics among 18-24 year olds. An alcohol overdose was present in 1 of 5 poisonings on these medications. </p>
<p>&#8220;The combination of alcohol with narcotic pain medications is particularly dangerous, because they both suppress activity in brain areas that regulate breathing and other vital functions,&#8221; Dr. White said. </p>
<p>The researchers noted that the steep rise in combined alcohol and drug overdoses highlights the significant risk and growing threat to public health of combining alcohol with other substances, including prescription medications. They call for stronger efforts to educate medical practitioners and the general public about the dangers of excessive alcohol consumption alone or in combination with other drugs.</p>
<p>&#8220;An increase in screening for alcohol misuse would help clinicians identify patients at particularly high risk for excessive drinking and for alcohol and medication interactions,&#8221; said NIAAA Acting Director Kenneth Warren, Ph.D. &#8220;Clinicians should use brief intervention techniques to help young adults evaluate their relationship with alcohol and other drugs and make wise choices regarding future use</p>
<p><em>Source  www.cadca.org  Sept. 2011</em><br />
</span></p>
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		<title>Marijuana Under the Guise of Medicine Contributes to the Rise in Marijuana Use</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/marijuana-under-the-guise-of-medicine-contributes-to-the-rise-in-marijuana-use/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/marijuana-under-the-guise-of-medicine-contributes-to-the-rise-in-marijuana-use/#comments</comments>
		<pubDate>Tue, 04 Oct 2011 13:44:59 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Cannabis]]></category>
		<category><![CDATA[Europe]]></category>
		<category><![CDATA[Marijuana and Medicine]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7849</guid>
		<description><![CDATA[(St. Petersburg, FL) The National Survey on Drug Use and Health conducted by the Substance Abuse and Mental Health Services Administration (SAMHSA) and released this week shows a significant rise in marijuana use. In 2007, 4.4 million Americans 12 and older used marijuana; as of 2010 that number has risen to 17.4 million. The National [...]]]></description>
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<p>(St. Petersburg, FL) The National Survey on Drug Use and Health conducted by the Substance Abuse and Mental Health Services Administration (SAMHSA) and released this week shows a significant rise in marijuana use.  In 2007, 4.4 million Americans 12 and older used marijuana; as of 2010 that number has risen to 17.4 million.  The National Office of Drug Control Policy’s Director, Gil Kerlikowske, said the increases are prominent in states in which “medical” marijuana is legal.  The survey also shows that 21.5 percent of young adults aged 18 to 25 used illicit drugs in 2010, an increase from 19.6 percent in 2008.</p>
<p>“Other than the lone voice of Director Kerlikowske and large marijuana dispensary raids by the DEA, the Obama Administration has basically turned a blind eye to the medi-pot issue, a matter that fuels the rise in marijuana use and continues to be the biggest scam ever to be perpetrated on the American public.  While a crude toxic weed is peddled to sick and dying people as a medicine, our government has done far too little to protect the public. It is absolutely no surprise to me that marijuana use has sharply increased,” said Calvina Fay, executive director of Drug Free America Foundation, Inc. and Save Our Society From Drugs. </p>
<p>“Surveys have shown for years that when the perception of the harms of drugs decreases, use rises. The ruse that marijuana is a medicine has created a false sense that this addictive, dangerous drug is not harmful, but in fact helpful. Clearly, this belief has contributed to the increase of marijuana use among young people. In order to protect the public, it is time for our government to take its head out of the sand and aggressively push back against marijuana legalization for any purposes!  Perhaps it’s time to withhold federal funds from states that fail to uphold our nation’s drug laws,” Fay concluded.</p>
<p><em>Source:  Press Release Drug Free America Foundation  9th Sept.2011<br />
</em></span></p>
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		<title>Increase in HIV infections in Greece</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/increase-in-hiv-infections-in-greece/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/increase-in-hiv-infections-in-greece/#comments</comments>
		<pubDate>Tue, 04 Oct 2011 13:42:30 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Europe]]></category>
		<category><![CDATA[Health]]></category>
		<category><![CDATA[HIV/Injecting-Drug-Users]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7847</guid>
		<description><![CDATA[A significant increase (more than 10-fold) in the number of newly diagnosed HIV-1 infections among injecting drug users (IDUs) was observed in Greece during the first seven months of 2011. Molecular epidemiology results revealed that a large proportion (96%) of HIV-1 sequences from IDUs sampled in 2011 fall within phylogenetic clusters suggesting high levels of [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;"></p>
<p>A significant increase (more than 10-fold) in the number of newly diagnosed HIV-1 infections among injecting drug users (IDUs) was observed in Greece during the first seven months of 2011. Molecular epidemiology results revealed that a large proportion (96%) of HIV-1 sequences from IDUs sampled in 2011 fall within phylogenetic clusters suggesting high levels of transmission networking. Cases originated from diverse places outside Greece supporting the potential role of immigrant IDUs in the initiation of this outbreak. </p>
<p><em>Source: Eurosurveillance, Volume 16, Issue 36, 08 September 2011<br />
</em><br />
</span></p>
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		<title>Plain packaging removes cigarettes&#8217; appeal</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/plain-packaging-removes-cigarettes-appeal/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/plain-packaging-removes-cigarettes-appeal/#comments</comments>
		<pubDate>Tue, 04 Oct 2011 13:41:02 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Nicotine]]></category>
		<category><![CDATA[Prevention]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7845</guid>
		<description><![CDATA[Removing branding and wrapping cigarettes in plain packaging helps remove the appeal of smoking according to new a Cancer Research UK-funded study published in Tobacco Control. The researchers found that more women than men smoked less and found smoking less enjoyable when using the plain packs. Some smokers also claimed that they would be more [...]]]></description>
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<p>Removing branding and wrapping cigarettes in plain packaging helps remove the appeal of smoking according to new a Cancer Research UK-funded study published in Tobacco Control.<br />
The researchers found that more women than men smoked less and found smoking less enjoyable when using the plain packs.<br />
Some smokers also claimed that they would be more likely to attempt quitting if all cigarettes came in the dark brown unbranded packs used in this study.<br />
In the first study of its kind nearly 50 young adult smokers used non branded cigarette packets in normal everyday situations for two weeks. The researchers then compared the reaction to this packaging to the reactions of using regular packs for two weeks.<br />
The plain brown packs were given a fictional name with standard branding and the health warning “Smoking Kills”. Twice weekly questionnaires were followed up with face to face interviews for more in depth analysis of reaction.<br />
Plainly wrapped cigarettes were rated negatively against the original packs. Taking out the cigarettes less often, handing out cigarettes less frequently and hiding the pack more were all reported as a result of the plain packaging.<br />
Dr Crawford Moodie, the study’s lead author based at the University of Stirling, said: “Despite the small size of this study it adds an important real world dimension to the research on the way smokers respond to plain packaging. The study confirms the lack of appeal of plain packs, with the enjoyment and consumption of cigarettes being reduced. We’re now looking to build on this research to understand more about the impact of packaging on smokers.”<br />
The UK government is expected to begin a public consultation on the future of tobacco packaging later this year.<br />
Australia should be the first country in the world to wrap cigarettes in plain packaging. The Australian government has announced that all tobacco must be sold in plain packaging from July 1, 2012. Picture health warnings will also cover 75 per cent of the front and 90 per cent of the back of packs.<br />
Jean King, Cancer Research UK’s director of tobacco control, said: “While a small study, this research provides important insights into the power of cigarette packaging. Colourful and slickly designed packs are one of the last remaining avenues for tobacco companies to market their deadly product, so it’s interesting to see what might happen if and when this is removed. It’s important to remember that smoking remains the single biggest preventable cause of death in the UK, so preventing more people from starting and helping smokers to quit is vital. We look forward to the possibility of removing the silent salesman of cigarette packets.”</p>
<p><em>Source:   http://www.cancerresearchuk.org/     8tj Sept. 2011<br />
</em></p>
<p></span></p>
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		<title>Warning over &#8216;very toxic&#8217; chemical in Guernsey cocaine</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/warning-over-very-toxic-chemical-in-guernsey-cocaine/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/warning-over-very-toxic-chemical-in-guernsey-cocaine/#comments</comments>
		<pubDate>Tue, 04 Oct 2011 13:34:59 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Cocaine]]></category>
		<category><![CDATA[Drug use-various effects]]></category>
		<category><![CDATA[Health]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7843</guid>
		<description><![CDATA[Guernsey&#8217;s Health and Social Services Department has issued a warning about the danger of a toxic chemical found locally in cocaine. The department said levamisole had been detected in recent samples of the drug. It said that some people who ingested the chemical developed agranulocytosis, a potentially fatal condition that harms the immune system. Dr [...]]]></description>
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<p>Guernsey&#8217;s Health and Social Services Department has issued a warning about the danger of a toxic chemical found locally in cocaine.<br />
The department said levamisole had been detected in recent samples of the drug.<br />
It said that some people who ingested the chemical developed agranulocytosis, a potentially fatal condition that harms the immune system.<br />
Dr Roland Archer, the States analyst, said: &#8220;This is the first time that it has been detected in Guernsey.&#8221;<br />
He said: &#8220;Once levamisole has been added to cocaine, it is nearly impossible to remove it and it even survives processing of cocaine into &#8216;crack&#8217;.&#8221;<br />
New equipment costing £80,000 has enabled the department to examine drugs at a molecular level.<br />
A gas chromatograph mass spectrometer, recently purchased by the department, helped find the substance.<br />
Dr Archer said: &#8220;It gives us a lot more confidence when presenting the data on controlled drugs.&#8221;</p>
<p><em>Source: www.bbc.co.uk  26th August 2011<br />
</em><br />
</span></p>
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		<title>Heart Warning Added to Label on Popular Antipsychotic Drug (Seroquel)</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/heart-warning-added-to-label-on-popular-antipsychotic-drug-seroquel/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/heart-warning-added-to-label-on-popular-antipsychotic-drug-seroquel/#comments</comments>
		<pubDate>Tue, 04 Oct 2011 13:33:17 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Drug use-various effects]]></category>
		<category><![CDATA[Health]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7841</guid>
		<description><![CDATA[AstraZeneca is adding a new heart warning to the labels of Seroquel, a antipsychotic drug, at the request of the Food and Drug Administration. The revised label, posted on the Federal Food and Drug Administration website, says Seroquel and extended-release Seroquel XR “should be avoided” in combination with at least 12 other medicines (including methadone) [...]]]></description>
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<p>AstraZeneca is adding a new heart warning to the labels of Seroquel, a antipsychotic drug, at the request of the Food and Drug Administration. The revised label, posted on the Federal Food and Drug Administration website, says Seroquel and extended-release Seroquel XR “should be avoided” in combination with at least 12 other medicines (including methadone) linked to a heart arrhythmia that can cause sudden cardiac arrest. </p>
<p><em>Source: http://www.nytimes.com/2011/07/19/health/19drug.html?_r=1  July 2011<br />
</em></p>
<p></span></p>
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		<title>Glutamate dehydrogenase as a marker of alcohol dependence.</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/glutamate-dehydrogenase-as-a-marker-of-alcohol-dependence/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/glutamate-dehydrogenase-as-a-marker-of-alcohol-dependence/#comments</comments>
		<pubDate>Tue, 04 Oct 2011 13:30:39 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Addiction]]></category>
		<category><![CDATA[Alcohol]]></category>
		<category><![CDATA[Europe]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7838</guid>
		<description><![CDATA[Slovenian study identifies which chemicals in the blood best identify dependent drinkers in the sense of not missing those who are dependent, confirming when they have stopped drinking, and not falsely identifying non-dependent people as dependent. Summary The aim of this study was to determine the value of biochemical tests for glutamate dehydrogenase (GLDH) in [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;"></p>
<p>Slovenian study identifies which chemicals in the blood best identify dependent drinkers in the sense of not missing those who are dependent, confirming when they have stopped drinking, and not falsely identifying non-dependent people as dependent.</p>
<p><strong>Summary </strong></p>
<p>The aim of this study was to determine the value of biochemical tests for glutamate dehydrogenase (GLDH) in the blood as way of diagnosing alcohol dependence, in particular as compared to or in combination with other biochemical markers including gama-glutamyltransferase (GGT), aspartate-aminotranferase (AST), alanine-aminotransferase (ALT) and erythrocyte mean cell volume (MCV). All these levels were assessed three times in 238 alcohol dependent patients admitted to hospital (on admission, after 24 hours and after seven days) and also in healthy members of the public.<br />
Main findings All the values were significantly higher in the patients than in healthy persons. GLDH exhibited the fastest decrease in levels after the resumption of abstinence. 24 hours of non-drinking is sufficient for a reliable evaluation of the fall in GLDH activity, even more so when alcohol dependants had not drunk for three to seven days, offering a way to confirm the cessation of drinking. The time course of changes in GLDH and AST were more applicable than for GGT after a week, but GLDH changes were most reliable. GLDH was the most specific marker with almost 90% specificity, correctly identifying nine in 10 of the healthy subjects as non-dependent. A decision tree combining MCV,<br />
GGT and GLDH markers was selected as the best diagnostic procedure because of its simplicity, easy examination and moderate cost. It gave a model with 84.5% accuracy, excellent specificity at 90% (correctly identifying 9 in 10 healthy subjects as non-dependent) and very high sensitivity at almost 80% (correctly identifying 8 in 10 alcohol dependent patients as dependent).</p>
<p><strong>Conclusions</strong></p>
<p>The high accuracy of our classification model provides an opportunity to apply it as a helping method in finding and diagnosing alcohol dependence in everyday practice, with our exclusion criteria and differential diagnostic cautions taken into consideration. We strongly believe that watching changes in the activity of laboratory markers of alcoholism is an effective yet overlooked aid.<br />
Thanks for their comments on this entry in draft to Matej Kravos of the Psychiatric Hospital Ormoz in Slovenia. Commentators bear no responsibility for the text including the interpretations and any remaining errors. </p>
<p><em>Source:  Kravos M., Malešic I.<br />
Alcohol and Alcoholism: 2010, 45(1), p. 39–44.  Revised 22 Aug.2011</em></p>
<p></span></p>
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		<title>ER admissions for Ecstasy increase 74% in just four years&#8230;and nearly 20% involve children as young as 12</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/er-admissions-for-ecstasy-increase-74-in-just-four-years-and-nearly-20-involve-children-as-young-as-12/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/er-admissions-for-ecstasy-increase-74-in-just-four-years-and-nearly-20-involve-children-as-young-as-12/#comments</comments>
		<pubDate>Tue, 04 Oct 2011 13:00:23 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Ecstasy]]></category>
		<category><![CDATA[Health]]></category>
		<category><![CDATA[Youth]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7833</guid>
		<description><![CDATA[It was the party drug of the 90s. But alarmingly Ecstasy&#8217;s popularity seems to be rising again. A worrying trend is re-emerging for the illegal substance after U.S. hospital admissions involving Ecstasy leapt 74.8 per cent in just four years, according to a national study. Most of the Ecstasy-related hospital visits involved patients aged 18 [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;"></p>
<p>It was the party drug of the 90s. But alarmingly Ecstasy&#8217;s popularity seems to be rising again.  A worrying trend is re-emerging for the illegal substance after U.S. hospital admissions involving Ecstasy leapt 74.8 per cent in just four years, according to a national study.<br />
Most of the Ecstasy-related hospital visits involved patients aged 18 to 29, but notably 17.9 per cent involved children as young as 12<br />
The Substance Abuse and Mental Health Services Administration (SAMHSA) study indicated the number of hospital emergency visits involving Ecstasy increased from 10,220 in 2004 to 17,865 visits in 2008.<br />
Slightly more than half (52.8 per cent) of the emergency visits were male, the study found.  More than a third of the Ecstasy-related visits were made in the South (34.0 per cent) while nearly a third were in the West (31.4 per cent).<br />
Nearly a fifth were made in the Midwest (18.5 per cent), and nearly a sixth were made in the Northeast (16.1 per cent).<br />
But in another alarming trend the study also found that 77.8 per cent of these visits &#8211; almost 8 in 10 cases &#8211; also involved the use of at least one of more other substances alongside Ecstasy.<br />
The most common drugs used in combination with Ecstasy are marijuana, alcohol and cocaine.<br />
 In cases where patients were 21 or older 39.7 per cent had taken Ecstasy with three or more other drugs.<br />
&#8216;The resurgence of Ecstasy use is cause for alarm that demands immediate attention and action,&#8217; said SAMHSA Administrator Pamela S Hyde, J D. </p>
<p> The drug induces feelings of euphoria but can produce psychedelic and stimulant side effects such as anxiety attacks, hypertension and even hypothermia.<br />
The variety and severity of these can increase when the drug is used in combination with other substances.<br />
Dr Peter Delany, director of the Centre for Behavioural Health Statistics and Qualities at SAMHSA, said the agency &#8216;needed to start digging&#8217; to find the cause of the spike in admissions. &#8216;Kids are getting it (Ecstasy) at raves and parties, which may indicate a return to social gatherings,&#8217; he said.  &#8216;It is also probably a very cheap drug,&#8217; he added.<br />
&#8216;The largest group of people (doing Ecstasy) are 18 to 29. These are people who have a lot more freedom and a lot more money,&#8217; he said.   He also cited the need for prevention education to continue well into adulthood to address this age group.<br />
 The more pressing issue, Dr Delany said was the people who were admitted to hospital with more than one substance in their system. &#8216;Ignorance is part of it,&#8217; he said. &#8216;There is a lot of risk taking in that age group.  &#8216;This (Ecstasy) is not a safe drug. The first time out of the door can have some serious side effects. When you are mixing it with multiple drugs you don&#8217;t know what the reaction will be. Everyone is different,&#8217; he said.<br />
Dr Delany also cited so-called &#8216;pharm&#8217; or &#8216;trail mixing&#8217; parties, when young people put a collection of drugs into a bowl and it becomes a very dangerous lucky dip.<br />
But these bowls don&#8217;t just contain illegal drugs, they also contain prescription drugs raided from parents&#8217; medical cabinets.   Another report by SAMHSA found there has also been a dramatic rise in emergency visits associated with the misuse of prescription drugs.<br />
From 2004 to 2008 these rose from 144,644 visits to 305,885 visits a year and occurred among men and women, as well as among those younger than age 21 and those 21 and older.<br />
The three prescription opioid pain relievers most frequently involved were Oxycodone products (rose 152 percent), Hydrocodone products (rose 123 per cent) and Methadone products (rose 73 per cent).<br />
&#8216;These alarming findings provide one more example of how the misuse of prescription pain relievers is impacting lives and our health care system,&#8217; said SAMHSA administrator Pamela S Hyde.   &#8216;This public health threat requires an all-out effort to raise awareness of the public about proper use, storage, and disposal of these powerful drugs.&#8217;</p>
<p><em>Source:  www.dailymail.co.uk  25th March 2011<br />
</em><br />
</span></p>
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		<title>Substance Use amongst Children in Scotland</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/substance-use-amongst-children-in-scotland/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/substance-use-amongst-children-in-scotland/#comments</comments>
		<pubDate>Tue, 04 Oct 2011 12:57:26 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Miscellaneous]]></category>
		<category><![CDATA[Youth]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7831</guid>
		<description><![CDATA[WEEKLY DRINKING Weekly drinking is reported among even the youngest children in the survey. At age 11, 3% of young people report drinking alcohol every week (4% of boys and 2% of girls)). One in ten 13-year olds (10%) and more than a quarter of 15- year olds (27%) are weekly drinkers. Among 13 and [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;"></p>
<p>WEEKLY DRINKING<br />
Weekly drinking is reported among even the youngest children in the survey. At age 11, 3% of young people report drinking alcohol every week (4% of boys and 2% of girls)). One in ten 13-year olds (10%) and more than a quarter of 15- year olds (27%) are weekly drinkers. Among 13 and 15-year olds, there is no gender difference in weekly drinking.<br />
In all six surveys since 1990, young people have been asked about their alcohol consumption frequency2. The highest rates of weekly drinking were found in 1998 (45%of girls and 44% of boys). Reporting of weekly drinking in 2010 is similar to that in 1990, with a particularly large decline since 2006 among both boys and girls (29% of boys in 2010 compared with 39% in 2006 and 25% of girls in 2010 compared with 36% in 2006) .</p>
<p>TYPES OF ALCOHOL DRINKS<br />
Young people were asked to report how frequently they drink each of seven listed alcoholic drinks. They were instructed to include those times when they only drink a small amount. Beer is the alcoholic drink most commonly consumed at least once a week by 15-year old boys, whereas, for 15-year old girls, spirits and alcopops are the preferred drinks.  Boys are almost 5 times more likely to drink beer weekly than girls. Girls are 1.5 times more likely to drink alcopops. </p>
<p>DRUNKENNESS<br />
Overall, a fifth of young people (20%) have been drunk on at least two occasions. Prevalence of drunkenness is much higher among older adolescents; 43% of 15-year olds report having been drunk at least twice compared with 15% of 13-year olds and 2% of 11-year olds .<br />
At age 15, girls are more likely than boys to report drunkenness (47% of girls compared with 40% of boys).<br />
Reporting of drunkenness among 15-year olds increased in the 1990s and then subsequently declined<br />
Among boys, prevalence in 2010 (40%) is similar to that in 1990 (44%). Among girls, rates of drunkenness have declined slightly since the late 1990s, but have not changed since 2006 (48%), and remain higher in 2010 (47%) than in 1990 (36%). </p>
<p>FREQUENCY OF CANNABIS USE<br />
Nineteen percent (19%) of 15-year olds and 4% of 13-year olds have used cannabis at least once in their lives<br />
Boys are more likely to have ever used cannabis than girls. Sixteen percent (16%) of 15-year olds and 3% of 13-year olds reported cannabis use within the previous year (Figure 12.13), with 15-year old boys being more likely to have used cannabis in the previous year than 15-year old girls (19% and 13% respectively). Nine percent (9%) of 15-year olds used cannabis in the previous month, compared to just 2% of 13-year olds. Among 15-year olds, boys are more likely than girls to report cannabis use in the last month (12% of boys and 6% of girls).<br />
Between 2002 and 2010, there has been a decrease in lifetime cannabis use among 15-year olds, from 39% to 23% among boys and from 35% to 15% among girls (Use of cannabis in the previous year has also decreased since 2002, from 31% to 19% among boys, and from 30% to 13% among girls . </p>
<p>CANNABIS USER GROUPS AMONG 15-YEAR OLDS<br />
Six percent (6%) of 15-year olds are classified as ‘experimental’ cannabis users (once or twice in the past 12 months), 7% as ‘regular’ users (between 3 and 39 times in past 12 months) and 2% as ‘heavy’ users (40 times or more in past 12 months) A small number (3%) report using cannabis, but not in the previous 12 months and were therefore classified as ‘former’ users. Boys are more likely to be heavy users, but there is no gender difference among other user groups. The proportion of young people in each category of cannabis use is lower than in 2002 </p>
<p><em>Source:THE HEALTH BEHAVIOUR IN SCHOOL-AGED CHILDREN: WHO COLLABORATIVE CROSS-NATIONAL STUDY (HBSC)  SCOTLAND NATIONAL REPORT 2010 SUBSTANCE USE<br />
</em></span></p>
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		<title>Study Shows Drug-Addicted Individuals May Have Less Brain Matter</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/study-shows-drug-addicted-individuals-may-have-less-brain-matter/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/study-shows-drug-addicted-individuals-may-have-less-brain-matter/#comments</comments>
		<pubDate>Tue, 04 Oct 2011 12:53:06 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Brain and Behaviour]]></category>
		<category><![CDATA[Drug use-various effects]]></category>
		<category><![CDATA[Health]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7829</guid>
		<description><![CDATA[A new study from the Department of Energy&#8217;s Brookhaven Natural Laboratory released this week suggests that people addicted to certain types of drugs might actually have lower density in crucial parts of their brain. This and previous studies have shown that cocaine-addicted individuals, relative to non-addicted individuals, have lower gray matter density in frontal parts [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;"></p>
<p>A new study from the Department of Energy&#8217;s Brookhaven Natural Laboratory released this week suggests that people addicted to certain types of drugs might actually have lower density in crucial parts of their brain.<br />
This and previous studies have shown that cocaine-addicted individuals, relative to non-addicted individuals, have lower gray matter density in frontal parts of the brain &#8211; which is important for paying attention and organizing one&#8217;s own behavior &#8211; and in the hippocampus, a brain region important for learning and memory.<br />
But it doesn&#8217;t stop at cocaine. The study revealed that persistent alcohol or cigarette consumption may have a similar effect.<br />
The longer cocaine, alcohol, and cigarettes were abused, the lower gray matter was found in the hippocampus and frontal regions of the brain. </p>
<p>This result means that curtailing drug use may be protective against such brain changes.<br />
The study did not test the effects of other substances. It did, however, clarify that genetic makeup may predispose certain individuals to lose brain matter over </p>
<p><em>Source: www.huffingtonpost.com  2011/03/13<br />
</em><br />
</span></p>
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		<title>Marijuana Use Precedes the Onset Of Psychotic Symptoms In Youth and Young Adults</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/marijuana-use-precedes-the-onset-of-psychotic-symptoms-in-youth-and-young-adults/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/marijuana-use-precedes-the-onset-of-psychotic-symptoms-in-youth-and-young-adults/#comments</comments>
		<pubDate>Tue, 04 Oct 2011 12:50:20 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Brain and Behaviour]]></category>
		<category><![CDATA[Cannabis]]></category>
		<category><![CDATA[Drug use-various effects on youth]]></category>
		<category><![CDATA[Youth]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7827</guid>
		<description><![CDATA[Mar 24, 2011 Marijuana use during adolescence and young adulthood increases the risk of psychotic symptoms, while continued cannabis use may increase the risk for psychotic disorder in later life, concludes a new study published in the British Medical Journal. Cannabis is the most commonly used illicit drug in the world, particularly among adolescents, and [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;"></p>
<p>Mar 24, 2011 </p>
<p>Marijuana use during adolescence and young adulthood increases the risk of psychotic symptoms, while continued cannabis use may increase the risk for psychotic disorder in later life, concludes a new study published in the British Medical Journal. </p>
<p>Cannabis is the most commonly used illicit drug in the world, particularly among adolescents, and is consistently linked with an increased risk for mental illness. However, it is hasn’t been clear whether the link between cannabis and psychosis is causal, or whether it is because people with psychosis use cannabis to “self- medicate” their symptoms.</p>
<p>So a team of researchers, led by Professor Jim van Os from Maastricht University in the Netherlands, investigated the association between cannabis use and the incidence and persistence of psychotic symptoms over 10 years. </p>
<p>The study occurred in Germany and involved a random sample of 1,923 teens and young adults from the ages of 14 to 24.</p>
<p>Incident cannabis use almost doubled the risk of later incident psychotic symptoms, even after accounting for factors such as age, sex, socioeconomic status, use of other drugs, and other psychiatric diagnoses. Furthermore, in those with cannabis use at the start of the study, continued use of cannabis over the study period increased the risk of persistent psychotic symptoms. There was no evidence for self medication effects as psychotic symptoms did not predict later cannabis use. </p>
<p>These results &#8220;help to clarify the temporal association between cannabis use and psychotic experiences,&#8221; the authors said in their study summary. &#8220;In addition, cannabis use was confirmed as an environmental risk factor impacting on the risk of persistence of psychotic experiences.&#8221; </p>
<p><em>Source:  British Medical Journal   March 2011<br />
</em><br />
</span></p>
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		<title>New research ‘makes the case’ for investment in young people’s drug and alcohol treatment</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/new-research-%e2%80%98makes-the-case%e2%80%99-for-investment-in-young-people%e2%80%99s-drug-and-alcohol-treatment/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/new-research-%e2%80%98makes-the-case%e2%80%99-for-investment-in-young-people%e2%80%99s-drug-and-alcohol-treatment/#comments</comments>
		<pubDate>Tue, 04 Oct 2011 12:46:53 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Treatment/Addiction]]></category>
		<category><![CDATA[Youth]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7825</guid>
		<description><![CDATA[24 February 2011 DrugScope has welcomed new research demonstrating that drug treatment services for young people are extremely cost effective, with long term savings of between £5 and £8 for every pound invested. Published by the Department for Education, the report, Specialist drug and alcohol services for young people – a cost benefit analysis, finds [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;"></p>
<p>24 February 2011</p>
<p>DrugScope has welcomed new research demonstrating that drug treatment services for young people are extremely cost effective, with long term savings of between £5 and £8 for every pound invested.<br />
Published by the Department for Education, the report, Specialist drug and alcohol services for young people – a cost benefit analysis, finds that drug and alcohol treatment for young people reduces otherwise significant economic, social and health costs. Immediate savings are achieved in reduced crime and improved health. In the longer term, there are reductions in costs associated with problematic drug use in adulthood, including unemployment, crime and drug and alcohol dependency.</p>
<p>Approximately 24,000 young people received specialist drug and alcohol treatment in the UK in 2008/09. Most were treatedprimarily for alcohol (37%) or cannabis (53%); one in ten were treated for problems associated with Class A drugs, including heroin and crack.<br />
A report published by DrugScope in 2009, Young people’s drug treatment at the crossroads, found that as well as helping young people with their drug or alcohol problems, treatment services also address wider needs, such as mental health issues, involvement with the criminal justice system and social exclusion.<br />
Despite evidence of the cost effectiveness of spending on substance misuse treatment, many young people’s services have contacted DrugScope to report significant cuts in local funding.<br />
Commenting on the report, Martin Barnes, Chief Executive of DrugScope said:<br />
“At a time when many drug and alcohol services for young people are facing funding cuts, this research makes a timely, compelling and robust case for continued investment. Even on quite cautious and conservative estimates, the evidence shows that there are immediate net gains in return for spending on drug and alcohol treatment. Not only will cuts in services have a negative impact on vulnerable young people, the research confirms that greater costs are likely to be incurred in terms of crime, unemployment and poor health.<br />
“The concern is that with a record number of young people not in education, employment or training there will be a greater demand on prevention and treatment services. It is far easier to prevent young people from developing problems at an early stage that it is to treat adults with addiction issues. A considered assessment of the benefits to local communities of investment in drug and alcohol treatment services needs to be made to inform decisions on funding.” </p>
<p><em>Source:  www.drugscope.org.uk  24 Feb 2011<br />
</em><br />
</span></p>
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		<title>Cannabis, synthetic cannabinoids, and psychosis risk: What the evidence says</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/cannabis-synthetic-cannabinoids-and-psychosis-risk-what-the-evidence-says/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/cannabis-synthetic-cannabinoids-and-psychosis-risk-what-the-evidence-says/#comments</comments>
		<pubDate>Mon, 03 Oct 2011 15:45:06 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Effects of Drugs (Papers)]]></category>
		<category><![CDATA[USA]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7822</guid>
		<description><![CDATA[Research suggests marijuana may be a ‘component cause’ of psychosis Joseph M. Pierre, MD Co-Chief, Schizophrenia Treatment Unit, VA West Los Angeles Healthcare Center, Health Sciences Associate Clinical Professor, Department of Psychiatry and Biobehavioral Sciences, David Geffen School of Medicine at UCLA, Los Angeles, CA Over the past 50 years, anecdotal reports linking cannabis sativa [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;"></p>
<p>Research suggests marijuana may be a ‘component cause’ of psychosis</p>
<p>Joseph M. Pierre, MD<br />
Co-Chief, Schizophrenia Treatment Unit, VA West Los Angeles Healthcare Center, Health Sciences Associate Clinical Professor, Department of Psychiatry and Biobehavioral Sciences, David Geffen School of Medicine at UCLA, Los Angeles, CA</p>
<p>Over the past 50 years, anecdotal reports linking cannabis sativa (marijuana) and psychosis have been steadily accumulating, giving rise to the notion of “cannabis psychosis.” Despite this historic connection, marijuana often is regarded as a “soft drug” with few harmful effects. However, this benign view is now being revised, along with mounting research demonstrating a clear association between cannabis and psychosis.<br />
In this article, I review evidence on marijuana’s impact on the risk of developing psychotic disorders, as well as the potential contributions of “medical” marijuana and other legally available products containing synthetic cannabinoids to psychosis risk.</p>
<p>CANNABIS USE AND PSYCHOSIS</p>
<p>Cannabis use has a largely deleterious effect on patients with psychotic disorders, and typically is associated with relapse, poor treatment adherence, and worsening psychotic symptoms.1,2 There is, however, evidence that some patients with schizophrenia might benefit from treatment with cannabidiol,3-5 another constituent of marijuana, as well as delta-9-tetrahydrocannabinol (?-9-THC), the principle psychoactive constituent of cannabis.6,7<br />
Three meta-analyses have concluded cannabis use is associated with an increased risk of psychosis</p>
<p>The acute psychotic potential of cannabis has been demonstrated by studies that documented psychotic symptoms (eg, hallucinations, paranoid delusions, derealization) in a dose-dependent manner among healthy volunteers administered ?-9-THC under experimental conditions.8-10 Various cross-sectional epidemiologic studies also have revealed an association between cannabis use and acute or chronic psychosis.11,12<br />
In the absence of definitive evidence from randomized, long-term, placebo-controlled trials, the strongest evidence of a connection between cannabis use and development of a psychotic disorder comes from prospective, longitudinal cohort studies. In the past 15 years, new evidence has emerged from 7 such studies that cumulatively provide strong support for an association between cannabis use as an adolescent or young adult and a greater risk for developing a psychotic disorder such as schizophrenia.13-19 These longitudinal studies surveyed for self-reported cannabis use before psychosis onset and controlled for a variety of potential confounding factors (eg, other drug use and demographic, social, and psychological variables). Three meta-analyses of these and other studies concluded an increased risk of psychosis is associated with cannabis use, with an odds ratio of 1.4 to 2.9 (meaning the risk of developing psychosis with any history of cannabis use is up to 3-fold higher compared with those who did not use cannabis).11,20,21 In addition, this association appears to be dose-related, with increasing amounts of cannabis use linked to greater risk—1 study found an odds ratio of 7 for psychosis among daily cannabis users.16<br />
There are several ways to explain the link between cannabis use and psychosis, and a causal relationship has not yet been firmly established (Table 1).1-7,11-19,21-25 Current evidence supports that cannabis is a “component cause” of chronic psychosis, meaning although neither necessary nor sufficient, cannabis use at a young age increases the likelihood of developing schizophrenia or other psychotic disorders.26 This risk may be greatest for young persons with some psychosis vulnerability (eg, those with attenuated psychotic symptoms).16,18<br />
The overall magnitude of risk appears to be modest, and cannabis use is only 1 of myriad factors that increase the risk of psychosis.27 Furthermore, most cannabis users do not develop psychosis. However, the risk associated with cannabis occurs during a vulnerable time of development and is modifiable. Based on conservative estimates, 8% of emergent schizophrenia cases and 14% of more broadly defined emergent psychosis cases could be prevented if it were possible to eliminate cannabis use among young people.11,26 Therefore, reducing cannabis use among young people vulnerable to psychosis should be a clinical and public health priority</p>
<p><em>Source: www.currentpsychiatry.com   Vol.10 Sept 2011<br />
</em><br />
</span></p>
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		<title>Volatile substance abuse</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/volatile-substance-abuse/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/volatile-substance-abuse/#comments</comments>
		<pubDate>Mon, 03 Oct 2011 15:42:07 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Education Sector (Papers)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7820</guid>
		<description><![CDATA[Volatile substance abuse can cause sudden death. Stephen Ream offers advice to youth workers on helping young people. What is volatile substance abuse? Volatile substances readily evaporate at room temperature, giving off a &#8220;sniffable&#8221; vapour. Volatile substance abuse (VSA) is when these substances are deliberately inhaled through the mouth and/or nose to achieve a change [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;"></p>
<p>Volatile substance abuse can cause sudden death. Stephen Ream offers advice to youth workers on helping young people.</p>
<p><strong>What is volatile substance abuse?<br />
</strong><br />
Volatile substances readily evaporate at room temperature, giving off a &#8220;sniffable&#8221; vapour. Volatile substance abuse (VSA) is when these substances are deliberately inhaled through the mouth and/or nose to achieve a change in mental state or &#8220;high&#8221;. The most commonly misused products are butane gas from cigarette lighter refills, aerosols (deodorants or hair sprays), petrol and some glues.<br />
Many people assume that, because these products are legal, they are safe. In fact, inhaled volatile substances can kill suddenly and unpredictably, and there is no way to avoid this risk.</p>
<p><strong>How many young people inhale volatile substances?<br />
</strong><br />
The cheapness and accessibility of products make younger and more vulnerable children particularly susceptible. In the annual NHS report Drug Use, Smoking and Drinking Among Young People in England, VSA continues to be the most common form of substance misuse among 11- to 13-year-olds, and second only to cannabis by the age of 15. However, we have seen the positive effects of preventive education, with usage falling from 5.5 per cent of pupils in 2009, to 3.8 per cent in 2010.<br />
According to the annual St George&#8217;s University of London report, VSA kills about 50 people a year in the UK. In the past decade it has killed more under-15s than all illegal drugs combined.</p>
<p><strong>Why do young people do it?</strong></p>
<p>VSA is an enticing high for teenagers in that it is cheap, accessible and fast-acting, and a volatile substance such as butane has little or no hangover effect. VSA is often a sign of problems in other areas of a young person&#8217;s life, such as bereavement, divorce or stress. But the motivating factors might just be sheer accessibility, peer pressure, boredom or a desire to shock parents or carers.</p>
<p><strong>What are the warning signs?<br />
</strong><br />
Like any drug, these can include mood or behavioural changes such as appearing drunk or dizzy, or seeming secretive, withdrawn, irritable, restless or inattentive. A chemical smell might be noticed, a runny nose, watery eyes, rashes or spots around the nose and mouth, throat irritation or nausea.<br />
Environmental evidence of use might include empty gas, aerosol or glue containers with teeth marks in the nozzle, or products disappearing from around the home. At least one parent told us that it was a &#8220;family joke&#8221; how much deodorant their teenager used until they realised what was going on.<br />
Social evidence might include truancy, poor academic performance, a new social group or isolation from previous friends, and a withdrawal from activities.</p>
<p><strong>What can youth workers do?<br />
</strong><br />
VSA can cause cardiac arrhythmia – a problem with the rate or rhythm of a heartbeat – and kill instantly. The only way to avoid this risk is to stop.<br />
If no advice is likely to encourage a user to stop VSA immediately, it might be appropriate to give information that helps them avoid other risks, such as: don&#8217;t do VSA alone or in dangerous or out-of-the-way places; don&#8217;t impede breathing in any way; don&#8217;t use near a naked flame or lit cigarette; and don&#8217;t drink alcohol or take other drugs. However, while these will reduce the risk of suffocation or fatal accident, the toxic effects of VSA can still kill at any point.<br />
If you find a young person intoxicated from VSA remain calm. Do not excite them or try to use force to remove the product. Any stress or physical exertion can trigger cardiac arrhythmia.<br />
When working with a young person engaging in VSA: strip the environment of temptations; have clear, visible policies on the use and storage of volatile substances; openly discuss the potential dangers to their health; explore carefully how and why VSA started; and arrange support from other agencies, such as generic drug services, GPs and counsellors.</p>
<p><em>Source:www.cypnow.co.uk 20th Sept 2011<br />
</em><br />
</span></p>
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		<title>Will the Real Drug Policy ‘Emphasis’ Please Stand Up!</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/will-the-real-drug-policy-%e2%80%98emphasis%e2%80%99-please-stand-up/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/will-the-real-drug-policy-%e2%80%98emphasis%e2%80%99-please-stand-up/#comments</comments>
		<pubDate>Mon, 03 Oct 2011 15:37:40 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Australia]]></category>
		<category><![CDATA[Prevention (Papers)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7818</guid>
		<description><![CDATA[A brief look at the confusing messages emerging from current ‘prevention’ application in Australian drug policy. QUIT – MODERATE – ACCOMMODATE? WHICH EMPHASIS ARE WE FOLLOWING? What is going on with Australian Drug Policy Prevention application? It appears to be struggling with, what can only be described, as a Dis-associative Identity Disorder (D.I.D). The current [...]]]></description>
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<p>A brief look at the confusing messages emerging from current ‘prevention’ application in Australian drug policy. </p>
<p><strong>QUIT – MODERATE – ACCOMMODATE? WHICH EMPHASIS ARE WE FOLLOWING? </strong></p>
<p>What is going on with Australian Drug Policy Prevention application? It appears to be struggling with, what can only be described, as a Dis-associative Identity Disorder (D.I.D). The current interpretation continues to baffle the average Australian, and leaves many of us who are active in the Alcohol and Other Drug (AOD) field scratching our heads in bewilderment and sometimes utter disbelief! </p>
<p><strong>SMOKING &#8211; The new leprosy? </strong></p>
<p>The growing and relentless assault against tobacco via the QUIT campaign is something only ‘mushrooms’ would know little of. This vital and effective demand-reduction and education ‘war’ has been clear from its inception, and has continued to burgeon, evermore aggressively to the crusade we now see today.<br />
The message is at the very least unambiguous, at times, bombastic! There is no guessing what the outcome of this endeavour is to be. The message and mandate is not ‘slow down’, it is not ‘moderate’ it is QUIT. The end game is the only game. There are no illusions about the time it may take to reach that goal, but that goal is the only target to aim at and as a consequence measures and outcomes are effective &#8211; more and more Australians are quitting!<br />
Let’s commence by acknowledging the following principle, which is all but irrefutable… accessibility, availability and permissibility all increase consumption. When you reduce these, you reduce consumption. For example, the following details shows how education and legislation all reduced demand. Accessibility, availability and permissibility are all restricted and consumption drops.<br />
In 1945 approximately 72% of Australian men smoked. The rate has been dropping ever since then. In 2007 only 18% of Australian males were daily smokers. In 1945 26% of Australian women smoked…In 2007 women were smoking at a lower rate than men with 15.2% still smoking daily. 1<br />
• increases in getting help to quit smoking, especially use of the Quitline (2% to 4%) and nicotine replacement therapy (7% to 10%);<br />
• increase in one year quit rate from 8% to 11% among smokers and recent quitters;<br />
• a statistically significant reduction of about 1.5% in the estimated adult prevalence of smoking. 2<br />
However, as successful as this message has been, the fight is not over yet, as the following excerpt so irrefutably affirms…<br />
“ANTI-SMOKING campaigners have far from finished their battle with the tobacco industry, with some pushing for a &#8221;license to smoke&#8221; and many predicting that cigarettes could be outlawed within a decade.” 3<br />
Well so was the bold opening statement in recent article ‘Now butt out: new push seeks to outlaw cigarettes’ in The Age Newspaper.-</p>
<p>Fascinating…outlawing cigarettes, even though around 17% of Australians are still smoking &#8211; outrageous! The article went on to note that if such a ban were to take place the government would stand to lose around $6 billion dollars in tax revenue, but save an estimated $31 billion dollars currently spent per annum on smoking related health problems.<br />
No doubt to everyone who is not a smoker this makes good health and fiscal sense…maybe even to some smokers too?<br />
So how is that we have managed to convince a society that a ban could actually be possible on a legal drug &#8211; tobacco, that in its boom era (during the 40’s, 50’s and 60’s) was a key social accessory, that a legal ban be actually possible? A quick inventory of the processes engaged may give us some insight… </p>
<p>•	A clear and uncompromising acknowledgement from health, government and fiscal sectors that cigarette smoking was damaging our community. </p>
<p>•	The ensuing resolve that this must change for both fiscal, but more importantly, health reasons. </p>
<p>•	The continuing single voice of disapproval of cigarettes from academics, politicians and health professionals. (Stopped the propaganda of the pro-smoking academics/doctors and started the recognition of the undeniable facts that ‘every cigarette is doing you damage’.) </p>
<p>•	The sustained political will to create and implement policies to bring about change, including increased taxation, total advertising ‘blackouts’ and bans on smoking in defined places. </p>
<p>•	These have been followed by the creation and implementation of Demand Reduction strategies that only grow in number and intensity and the relentless public education campaign on the dangers of smoking. </p>
<p>It would appear from both empirical data that such resolute policies work…even with a once widely accepted and socially palatable ‘legal drug’ like tobacco.<br />
In a recent war of words over the zealous, if not poorly thought through, ‘plain packaging’ strategy, the Federal Minister for Health Nicola Roxon was quoted as saying…. &#8220;Big tobacco are fighting to protect their profits, but we are fighting to save lives.” 4 If that vitriol wasn’t enough, she was also quoted in the Australian Newspaper, again in regard to challenges to the plain packaging strategy …‘&#8221;We&#8217;re Australians. We can make laws in Australia to protect Australians&#8230;” 5 Feisty! I like it! However, comes the question… protect Australians from what? Well, Captain Obvious may answer that in this context it would be protection from the health and health budget destroying wrecking ball that is tobacco.<br />
But is ‘health’ the real motivator that is underpinning this zeal for the wellbeing of Australians? I hope it is, but the utter inconsistency of this focused passion belies another agenda. Or is it that some people just can’t see the utter inconsistencies or, at worst, hypocrisies of this unbalanced policy focus?<br />
If ‘health’ was the sole or main issue, then wouldn’t that same zeal, that same passion for justice of Aussie’s Health be mirrored in other areas of drug policy too? I mean, Roxon is pursuing a policy – plain packaging – that has a number of downsides to it, and only small possibility of a reduction in smoking &#8211; But that was enough, it seems, for her to implement the policy! Great I say, go for it, but why doesn’t this same ‘doggedness’ apply to the two other big monsters in the drug arena?<br />
The Federal minister seems passionate about the anti-smoking message, passionate enough to make those sweeping statements we just read &#8211; ‘fighting to save lives!’ &#8211; ‘Making laws to protect Australians!’ and pursuing every possible vehicle to STOP people killing themselves (and our health budgets)on the way.<br />
In a very recent interview published by the Financial Review, we get a glimpse into some of the motivators behind Roxon’s campaign against tobacco – ‘This is a defining moment for Roxon one that transcends politics and is deeply personal. Her father, a one-time smoker, died of oesophageal cancer at the age of 42…“All of us girls keenly felt the loss of not having our father as we grew up but that is not the same as being out on the street as some families are…it has made me very aware of the impact that smoking can have,” Said Roxon. This mother of a 6 year old daughter went on in the interview to declare that, ”This fight is about the past and the future. “We might be making the world a healthier place for our children, and that is very motivating. I don’t think the political gains will be very high or very quick, but the long-term health impact and feeling [that] you are in government to do some good is rewarding.”’*<br />
I have no issue at all with this motivation from Roxon, I mean it is the personal encounter with tragedy and/or the grief of loss/dysfunction that adds undisputable weight to the abundance of health-destroying evidence that exists. But again, why isn’t this same passion for health/safety/future of children applied to the other life and health destroying drugs in the ‘recreational’ arena? Nicola would do well to spend time at Rehabilitation clinics, with families of alcohol and other drug using individuals who have not only shattered their lives but their families. Countless stories of lives and potential ruined at young ages because a drug was accessible, permissible, available and cheap. This very powerful evidence should also inform the prevention focused emphasis of alcohol and other drugs policy platform. All measures including high volumetric tax, plus clear and powerful warning labels should also be taken immediately to further ensure that children and families have the greatest protection from the damage of these drugs.<br />
Alcohol – The protected substance?<br />
When it comes to the other ‘legal drug’ the (it would appear) culturally entrenched alcohol &#8211; options for management have one glaring omission. Can you guess what it might be? No prizes if you said ‘QUIT’. The conspicuousness of the absence of this goal in the strategy is probably the noisiest of all elephants in the ‘Drug policy’ room. So, why is that?</p>
<p>We seem to have no problems creating what ‘defenders of the right to self destruct’ call a ‘Nanny State’ posture when it comes to cigarette smokers or our indigenous communities for that matter &#8211; But when it comes to the rest of the population quitting or abstaining from alcohol, then howls of derision chanting anti-‘Nanny State’ mantras are deafening!<br />
James Campbell in his article ‘wowsers enough to drive you to drink’ featured in Herald-Sun 6 drew out, in his classic libertine framework article, some of the same inconsistencies we are bringing to attention in this paper &#8211; but I’m quick to add, for very different reasons. (Of course James would never have used the term ‘wowser’ in his title if he had even an inkling of what it stands for – We Only Want Social Evils Rectified – This of course is what all socially responsible people want. Yes, a free society, but a freedom that doesn’t disregard a) the liberty, safety and wellbeing of others b) the protection of the young, and c) bestowal of dignity on every human being… all of which are casualties when the imbibing begins.)<br />
In his article he noted the data and subsequent recommendations recently released by the Cancer Council, but also what he has interpreted their seeming ‘double standard’ on the ‘drink’ issue. Professor Olver was quoted in the Age as saying… &#8221;If you want to reduce your cancer risk as far as possible [abstinence] would be the option you have.&#8221; 7 yet in his article, Campbell states they stopped short of recommending abstinence from alcohol and settled for NHRMC recommendations of ‘a couple of standard drinks at any time’.<br />
Now whilst I can see the point of incongruence, I would like to challenge Campbell’s ‘framing’ of the response. It is clear that not all cancers are caused or even added to, by alcohol, but it is equally clear, through evidence based science, that alcohol is carcinogenic.*<br />
The point now is what do we do with that information? Certainly promoting abstinence as an option should be absolutely imperative…but that’s the problem… the ‘A’ word isn’t permitted, even in the ‘optional’ category!<br />
Our culture is either so deeply addicted to this drug or so completely gripped by fear at being labelled something less than human because they don’t drink, that they actually cannot see the option of saying ‘No Thank you!’<br />
Now if this was just, fully developed ‘grown ups’ who don’t care about their health or even worse, are self-medicating the vicissitudes of life with the grog, and never venture into the public space and expose others in the community to their less than sober persona, I suppose it would make less difference if one ‘partook’ (except for the medical and health bills the tax payer will have to fund)! However, it is the vulnerable in our society &#8211; the young (under 25 &#8211; still developing brains), the mentally ill, the socially and relationally isolated, the violent, the elderly, children and often women, who end up casualties of not only their own drinking, but that of others!<br />
Whilst the link between cigarettes and disease is clear, it is no less clear with alcohol…<br />
Alcohol consumption is the world’s third largest risk factor for disease and disability; in middle-income countries, it is the greatest risk. Alcohol is a causal factor in 60 types of diseases and injuries and a component cause in 200 others. Almost 4% (1 in 25) of all deaths worldwide are attributed to alcohol, greater than deaths caused by HIV/AIDS, violence or tuberculosis. 8<br />
A couple of questions that are often conspicuous by their absence, when it comes to the inconsistencies in drug policy when dealing with tobacco and alcohol, are to do with impact on others. Yes, it is good to have gone to considerable lengths to minimise ‘passive smoking’, but what of the impact of what Professor Rob Moodie calls ‘passive drinking’? A couple of quick questions to ponder…<br />
The real tragedy in all this ‘cultural reinforcement’ is that the imperative message for this demographic of abstinence, and delayed onset of drinking as long as possible, has completely disappeared .<br />
When was the last time a cigarette caused a man to beat his wife to death?<br />
When was the last time a cigarette caused an automobile accident killing two and disabling one for life?<br />
When was the last time a cigarette caused a pub brawl or ‘glassing’ incident?<br />
For the sake of brevity (and being seen to be too merciless on the sensibility of the Aussie imbiber) the following are just some of the long known, but only recently quantified data on this so called ‘social lubricant’…. </p>
<p>a) Fiscal Cost: The research by the Australian Education and Rehabilitation Foundation (AER Foundation) has now put the total economic impact of alcohol misuse at $36 billion per annum which is over double 2005 estimates. This comprises $24.7 billion in tangible costs, which include out-of-pocket expenses, forgone wages or productivity and hospital and childcare protection costs. There are a further $11.6 billion in intangible costs, which includes lost quality of life from someone else&#8217;s drinking9 </p>
<p>b) Consumption: Drinking more than ever before, at least 10.2 litres pure alcohol per person per annum 10 </p>
<p>c) Cancer: “Alcohol use has been linked to thousands of cases of cancers including bowel, mouth, pharynx and larynx. 1 in 5 cases of breast cancer are linked to alcohol”. 11 </p>
<p>d) Violence: There are more than 70,000 Australians who are victims of alcohol related assaults each year&#8230;alcohol-fuelled violence and abuse affects one in five people 12 </p>
<p>e) Emergency Services: Ambulance Call outs in Greater Melbourne alone, for predominantly alcohol abuse have increased almost 600%: 1998-99: 1043 by 2008-09 it was 6924 13 </p>
<p>f) Crime &#8211; In just one State alone, alcohol-related crime in Queensland has increased by 30 per cent, and public disorder offences by 65 per cent just in the past few years alone&#8230;Alcohol abuse in Queensland is now responsible for 100,000 crimes annually, or one-quarter of all offences.14 </p>
<p>You get the point! This is, if not worse, then at the very least as bad as the smoking issue…. So, why aren’t all zeal, all passion and all strategies being implemented to prevent or stop the impact of alcohol on the Australian people and the economy?<br />
So entrenched is the alcohol culture that according to the Australian Drug Foundation, parental supply has eclipsed all other sources of supply of alcohol to children aged 12-17. Now the excuses tabled for this kind of outrageous conduct are as follows… </p>
<p>a) Parents want to either, initiate their child into alcohol ‘wisely’ or at least ‘know’ how much they are drinking. </p>
<p>b) Parents want to be friends with their child and not parents. Believing they are avoiding stress at home by giving in to negative social influences. </p>
<p>c) Parents believe that if their children are going to ‘experiment’ then it’s better to do so with the legal drug. </p>
<p>d) ‘It’s part of being Aussie, it’s gonna happen, so might as well try and be ‘responsible’ and give them a hand in using this legal drug ‘properly’.’ </p>
<p>So, how has that been working for us as a community? Well the evidence seems to correspond with the mindset. Again an Australian Drug Foundation recent release shows that by 16, one in five teenagers regularly binge drinks; by 18 it is 50 per cent.<br />
It would appear this level of permissibility has only added to accessibility and availability and thus consumption has increased. I mean… ‘after all Mum and Dad are giving it to me and they use it, so it must be ok?’<br />
The real tragedy in all this ‘cultural reinforcement’ is that the imperative message for this vulnerable demographic of abstinence and delayed onset of drinking as long as possible, has completely disappeared. All the scientific evidence reveals that their vulnerable developing brains need this option to be aggressively promoted as best practice and their parents, above all, need to get this reality check too.<br />
Again, what continues to generate this disconnect between policy emphasis around the legal drugs of tobacco and alcohol? Both drugs are legal, but perhaps smoking an easy target now that fewer Australians do it, and is marginalised so much that scathing vitriol and uncompromising legislation will have little opposition? “But, not so with alcohol &#8211; Whilst approximately 14% of Australians who are legally permitted to drink, don’t, the amount of alcohol being consumed per person, per annum is near record highs. It would seem that challenging this second ‘monster’ can prove a difficulty, if a) votes matter b) the power brokers themselves are unable to say NO to alcohol; c) It has become the central and often sole ‘social amenity’ or even worse, d) it becomes the medication of choice for the ever growing epidemic of community wide psycho-social dysthymia.<br />
Whatever the reason, a clear gulf exists in zeal, attention and endeavour when we juxtapose tobacco and alcohol. A gulf that screams, at best inconsistency, but at worst hypocrisy!<br />
A quick recap…<br />
When it comes to tobacco the policy aim for smoking is ‘quit’, and we have no problem aggressively challenging ‘smoking’ as a reckless act that needs stronger management. We have used Prohibition in its legal context to prevent smoking in a number of places and breaches of such prohibitions have met with not only social censure, but a fiscal punitive response &#8211; fines. And in this framework there appears no fear about attracting the pejorative ‘nanny state’ label.<br />
When it comes to Alcohol, the policy aim (at the moment at least) is to avoid the ‘nanny state’ label, calling instead for management, more like a caring friend provoking a peer to a healthier choice. So the push seems to be toward ‘moderation’.<br />
But what is happening in the arena of current illicit drug policy?<br />
We appear to be losing the plot – the pro-drug lobby is trying to take over the judiciary, if not legislature!<br />
When it comes to illicit drugs there appears to be a departure from all regulatory sanity. The ‘State’, on whose advice we can easily guess (George Soros funded propagandists) works ruthlessly to assassinate, mutilate and bury all processes that are focused on prevention or abstinence. Such processes the patronizingly dump into the ‘Nanny-State’ model/basket . Nor, would it seem are they interested in a Good Parent model, or even the ‘caring friend’ model… No, it would appear from all current debate this confederacy has opted for the ‘go with whatever feeling grabs you; it’s your ‘right’ and let the State clean up the mess’ approach!<br />
There appears little to no censure, no label of ‘bad’ or ‘harmful’ or ‘destructive’ to the conduct that is illicit drug using. In fact great pains are taken to remove all terms from public documents that could potentially ‘marginalise’ the drug user. Whilst ‘name calling’ should never be condoned, conduct that illegal and destructive needs to be called for what it is and measures taken to change it. Whether the terms are legal or medical, they can never be ‘neutral’, or worse complimentary and condoning.<br />
What is of greater concern is the tacit message oozing through the permissive interpretation of Harm Minimisation policy by the Harm Reduction Only Lobby, which is that the State sanctions and promotes &#8211; not challenges or changes &#8211; a drug user’s ‘habit’. (Yet it is the ‘habit’ that needs to change &#8211; more on that later.)<br />
For example, they seem to be saying : </p>
<p>a) Please come to a special place with your illegal substance and we will assist you to take the drug of your choice (Medically Supervised Injecting Centre &#8211; MSIC). At no point will anyone ‘judge’ you for your ‘lifestyle choice’. Instead we will ensure you are comfortable and enabled in your drug taking activity whilst funding this process with tax-payer’s money. (No matter that this process breaks international laws on illicit drug use) </p>
<p>b) We will give you as many clean ‘needles’ as you like and will not hold you accountable for the return of used ones. In fact we will pay someone to go around and pick up your discarded syringes so you can continue to be free (not irresponsible, that would be pejorative)to continue, unhindered in your substance use, wherever and whenever you choose. </p>
<p>c) If the substance user opts to seek a change in conduct, only then may we humbly recommend a referral to a treatment facility. However, after we have just enabled you to continue your substance abuse (in our MSIC) and you are ‘feeling’ better (yet getting worse) after your State assisted ‘fix’, then it is unlikely that you’ll ‘feel’ the need for detox, let alone rehabilitate. So, the passive referral is ignored or forgotten. </p>
<p>d) If you are one of the single digit percentage of substance users that actually ‘follows through’ on referral, then no requirement will be placed on you to become drug free. No, we are only interested in trying to minimise your potential to kill yourself and make you as comfortable as possible. We will introduce you to other substances that may, or may not lead you to drug free recovery, but again, that is NOT our aim. This, after all, is only for the ‘problematic’ drug user and we must not have anyone feeling discomfort or distress from the withdrawal from drug use, even if is for a week &#8211; That would be ‘unkind’. So rather than treat you like a precious, intelligent, whole human being, we’ll simply treat you like a wounded pet and only treat the symptoms and not address the real problem. </p>
<p>e) The recent aggressive upsurge of promotion and use of, so called, ‘legal highs’ has produced an even clearer manifestation of this policy D.I.D/hypocrisy/inconsistencies. As these synthesized ‘designer’ concoctions started getting a more public profile, several States in Australia were quick to react by imposing age restrictions and then applying significant financial penalties (six figure fines) for those involved in distributing/using these products. Yet in some of these same States the use of current illicit drugs such as cannabis (and other currently illicit drugs that have clearly documented health damaging properties) attracts no more than a slap on the wrist for use and little more for trafficking!<br />
It would seem no effort is spared, to ensure the drug user is rarely, if ever, is called to make changes. More than that, and at any point, an act of horrendous nature can be perpetrated against another citizen as we saw recently in the senseless murder of a deaf octogenarian pensioner, murdered by yet another (it would appear by the new label) ‘problematic drug user’. Diminished responsibility, mitigation, equivocation, even obfuscation, are employed to avoid ownership of the issue by the substance user. What’s more disturbing is that at no point is the abysmally interpreted Harm Minimisation Policy used to bring about change, let alone drug free wellness of these dysfunctional people.<br />
The following (conveniently) long forgotten words of the remarkable Statesmen, Edmund Burke, are even more appropriate today than at any other time in recent history…<br />
&#8220;Men are qualified for liberty in exact proportion to their disposition to put moral chains upon their own appetites&#8230; Society cannot exist, unless a controlling power upon will and appetite be placed somewhere; and the less of it there is within, the more there must be without. It is ordained in the eternal constitution of things, that men of intemperate minds cannot be free. Their passions forge their fetters.” Sir Edmund Burke </p>
<p>The very thing that is needed as outlined by Burke is the very thing the pro-drug lobby works tirelessly to negate. Morality is ‘off the table’ in this arena (The only time morality is invoked these days is when it comes to climate change; nowhere else is this allowed in the public discourse) In this ‘amoral’ space all attempts to impugn drug taking are perceivably removed. Terms like ‘wrong’, ‘bad’ ‘irresponsible’ are no longer permitted. So, if it is no longer referred to as ‘wrong’ then comes the next manipulative question: on what grounds should substance use still be illegal? The next step is to turn the debate into a purely ‘health’ issue. It is true, it is also a health issue, but, it is still a social, psychological and moral issue as well. But even just at the level of health policy, would think that all measures should be taken to rectify the dysfunction /disorder/ailment in order to remove the health damaging substances at least from the patient, even if not the community. Ah, but no, that’s not the agenda of this lobby faction is it!<br />
The health issue is invoked only to manage some of the damage of substance taking and other second tier outcomes of these bad health choices, such as blood borne infections and or death. The call now in this decriminalised, so called amoral and consequence avoiding space, is that all health measures be taken to keep the patient alive and as healthy as possible to continue their ‘lifestyle choice’ of drug consumption.<br />
This is not Australian – Time to Stand up!<br />
At the moment the vast majority of Australians are still smart enough to know (perhaps drug free enough to know) that ultimately there I absolutely no gain/benefit in illicit drug use for individuals or society;  The current National Household survey (2007) has the vast majority of Australians declaring their disapproval of illicit drugs and their use. </p>
<p>•	99% don’t want use of hard drugs accepted<br />
•	95% don’t want hard drugs legalized<br />
•	94% don’t want use of cannabis accepted<br />
•	79% don’t want cannabis legalized<br />
•	Most Australians want tougher penalties for drug dealers.15 </p>
<p>The largest youth survey done in our nation with a sample of around 50,000 young people saw alcohol and others drugs as the second highest on ‘what is an important issue for Australia’. This issue is the most worrying to the youngest in this most susceptible to damage of Australia’s demographic – the ones we need most protect – our children 16 </p>
<p>When the overwhelming majority of people disapprove of illicit drugs, it might just be a cue to do something more significant than concede ground to it. You’d think that even the process (let alone value) of democracy, had any weight then the above mentioned majority opinion would mandate all and every action be taken to eradicate illicit drug use from society. According to collected data, around 6% of the world’s population aged between 15 and 64 currently use illicit drugs. 17 Australia’s stats are only a little higher than that. So here we have a user group that is arguably (at most) between a half or a third of current tobacco users, who are involved in a wilful breaking of the law to their own and the wider community’s detriment generating an exorbitant cost to our community.<br />
So what has the response been to this? Well, it depends on where you look, who you talk to and who is playing the strings of the propaganda harp.<br />
In recent years there has been a rising noise, about the need for illicit drug policy change. The standard mantra has been ‘the war on drugs has failed!’ Consequently we need to stop and rethink our processes and priorities. </p>
<p>What ‘war on drugs’? Where did this notion come from?<br />
Well, let’s pretend for a moment there actually was a ‘war on drugs’. How could it possibly be won? Well, again it depends on how this ‘war’ was fought and what priorities were set. If the war on drugs simply attempted supply removal and arrest, then it will have limited success. However, as with most ‘battle strategies’, if they only have one tactic, then success will always be limited or the potential for failure increased. If a ‘war on drugs’ isn’t really waged as it should be then it is locked into only limited success and more likely subject to criticism of its limitation. However, as in all wars the first casualty is always truth and that is no different in this theatre of combat, as the following reveals… </p>
<p>The term “war on drugs” was not used in 1971 and is not used today by anyone except those who mischaracterize history and current drug policy in the US. However, if one were going to connect the term to President Nixon, then it would be more accurate to say that Nixon ended, rather than launched, the “war on drugs.”<br />
The Nixon Administration repealed federal mandatory minimum sentences for marijuana, and on June 17, 1971, for the first time in US history, the long-dominant law enforcement approach to | 12 drug policy, known as “supply reduction”, was augmented by an entirely new and massive commitment to prevention, intervention and treatment, known as “demand reduction”. President Nixon announced this new, balanced approach to drug policy and it received full bipartisan support. Since that time, the idea of taking a balanced approach has enjoyed strong and sustained support through the terms of the seven US Presidents that followed. The US drug prevention policy, fully described in the annual National Drug Control Strategy published by ONDCP, maintains this twin-commitment to supply reduction and demand reduction, with the aim of reducing illegal drug use and the corresponding medical and social burdens that drug abuse imposes upon our nation.18<br />
Supply reduction remains a key tactical component and criminalisation will always lend weight to that vital strategy component. Time and space here will not permit us to go into all the local and national impact on drug use that supply reduction has facilitated, but just two examples will give us a clear indication: </p>
<p>a) ABS 2000 death stats collection: Heroin: 417; methadone: 118;Benzos: 403; anti-depressants: 268; Cannabis: 49 Note the reduction in Heroin deaths the following year when the heroin drought (for whatever reason) caused availability to dry up, the ABS 2001 death stats collection showed: Heroin: 113; methadone: 107;Benzos: 252; anti-depressants: 194; cannabis: 28! </p>
<p>b) According to the Australian Institute of Criminology, the four top reasons why detained illicit drug users had not used in the previous month 19 was in order of main reason to least. </p>
<p>1) Dealer didn’t have drug of choice (highest reason by far) </p>
<p>2) No Dealers available </p>
<p>3) Poor quality product </p>
<p>4) Police presence </p>
<p>I want you to notice that supply reduction elements are the key factor in reducing illicit drug consumption. Again, when you reduce permissibility, accessibility and availability you reduce consumption. This is why complementary Supply Reduction strategies are imperative in conjunction with Demand Reduction strategies and compulsory detox and rehabilitation strategies.<br />
When Ethan Nadelmann and Dr. Alex Wodak, the well-known supporters/ purveyors of the George Soros brand of cultural chaos, were on the media stage peddling their brand of harm ‘reduction’( (including the decriminalisation of illicit drugs), the voices of dissent from any other quarter were hard to hear, but not because they don’t exist considering over 90% of Australians disapprove of illicit drugs. It was the classic situation where the sane majority simply expect the government to do all that is necessary to eliminate drug use without bothering to mobilise against that small, but very ‘squeaky wheel ‘of pro-drug propaganda at legislators doors. Consequently, the long standing anti-drug movements were given no space at all.<br />
The Nadelmann/Wodak ‘spin’ had people believing prohibition drug policy had failed and therefore the only option left was to decriminalise or legalise. They even used cleverly spun unrelated science and misrepresented data from other nations and calling that ‘enlightened’ (Such as the so called Portugal decriminalisation ‘success’). Or they hijacked the debate away from drug use and placed it in the framework of management of damage caused by drug use, which actually increases dysfunction.<br />
It is remarkable that few clinicians or policy makers care to see or even acknowledge that the current illicit drug policy in Australia (among other western nations) has be completely hijacked by the single dimensional ‘harm reduction’ element and that has distanced them even further from the problems of drug use.<br />
This one dimensional focus has barely anything to do with drug use and absolutely nothing to do with reducing drug use. ‘Harm Reduction’ as it currently stands, when it is all distilled down to its core (a one step process) is only focused on the attempted prevention of death and blood borne infections. Whilst this may be a noble aim, we need to move drug policy back to the forgotten reduction or prevention of drug use in our society. We are all for having a policy for reducing the spread of blood borne infections and death, but let’s call it that and move drug policy back to what drug policy is supposed to be about – the prevention and reduction of drug use in our society. Of course, even a ‘blind man’ could see, that if you prevent and/or reduce drug use, you reduce the incidence of the other damage so focused on &#8211; but that is the very thing the pro-drug lobby doesn’t want to happen, the reduction of drug use! They advocate continuation of drug use, funded by tax-payer’s who keep them alive and pay for their treatment.<br />
So in our mind, an unavoidable question is &#8211; Where was Federal Minister for Health, Roxon on these issues? Where was the same zeal that was focused on cigarettes? At the time where this ‘drug reform’ lobby has used special arguments to remove the protection, where was the declaration, ‘making laws that protect Australians’ from substances that have long been banned because of the undeniable damage they do?<br />
Is it utter ignorance that generates this silence? Or is it as one prominent AOD Clinician once said ‘Harm minimisation is just a euphemism for ‘we don’t know what the hell to do, so we’ve just given up!’. Or is it, reason spare us, a tacit yet wilful pursuit of cultural sabotage foisted on society because a minority of drug users who believe they can control their ‘habit’ have ‘friends’ in high places?<br />
Prohibition is a word that has been marginalised and disparaged, again by hijacking the meaning and reinterpreting it in a different context – the context of purely a moral control of a majority. However, prohibition is, in this context, a matter of law and not a simple moral based endeavour.<br />
 We prohibit by law things that are injurious to individuals and the community. With Tobacco law, cigarette smoking is prohibited in restaurants, government buildings, some public spaces, inside cars and so on. Illicit drugs are prohibited at a higher level because of the health, family and social damage and the impediment of function and increased danger they that create. The prohibiting is based on minimising the harms done by these toxins to the community and individuals. Decriminalisation will only lead to greater substance use and experimentation and simply bolsters well the ranks of the damaged and dysfunctional. It will perpetuate this damage in an emerging generation that has little capacity to handle it. This is a crime! </p>
<p>Will the real drug policy emphasis, please stand up and will it stand for health, justice, responsibility and protection of the young? </p>
<p><em>Source: Shane Varcoe – Executive Director, Dalgarno Institute.  www.dalgarnoinstitute.org.au    August 2011<br />
</em><br />
Endnotes<br />
1 http://www.cancercouncil.com.au/editorial.asp?pageid=371<br />
2 CHANGES ASSOCIATED WITH THE NATIONAL TOBACCO CAMPAIGN PRE AND POST CAMPAIGNSURVEYS COMPARED by Melanie Wakefield http://www.health.gov.au/internet/main/publishing.nsf/Content/health-pubhlth-publicat-document-metadata-tobccamp.htm/$FILE/tobccamp_c.pdf<br />
3 Stark , Jill The Age, 22.5. 2011 http://www.theage.com.au/victoria/now-butt-out-new-push-seeks-to-outlaw-cigarettes-20110521-1ey2s.html#ixzz1OBTg5SRQ<br />
4 http://www.smokernewsworld.com/market-cheap-cigarettes/<br />
5 Nicola Roxon solid on cigarette packaging Sallie Don and Sue Dunlevy From: The Australian May 27, 2011 http://www.theaustralian.com.au/national-affairs/nicola-roxon-solid-on-cigarette-packaging/story-fn59niix-1226063781056<br />
6 James Campbell – wowsers enough to drive you to drink, page 78, Sunday Herald-Sun May 28, 2011,<br />
7 http://www.theage.com.au/lifestyle/wellbeing/quit-drinking-to-cut-cancer-risk-20110501-1e38g.html<br />
8 Global Status Report on Alcohol and Health. Taken from Introduction page x, ISBN 978 92 4 156415 1 (NLM classification: WM 274) © World Health Organization 2011<br />
9 Alcohol Education and Rehabilitation Foundation &#8211; Range and Magnitude of Alcohol’s Harm to Others August 2010<br />
10 Wine link to rise in alcohol intake, Sikora, Kate; Page 16, Herald-Sun Edition 1 – 2/11/2010<br />
11 Medical Journal of Australia (published May 2011)<br />
12 Alcohol Education and Rehabilitation Foundation &#8211; Range and Magnitude of Alcohol’s Harm to Others August 2010<br />
13 http://www.heraldsun.com.au/news/more-news/mateship-abandoned-drunks-left-behind/story-fn7x8me2-1226063706968<br />
14 “Punch Drunk Campaign”, QLD Courier Mail – July 2009<br />
</span></p>
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		<title>Personality-targeted interventions delay uptake of drinking and decrease risk of alcohol-related problems when delivered by teachers.</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/personality-targeted-interventions-delay-uptake-of-drinking-and-decrease-risk-of-alcohol-related-problems-when-delivered-by-teachers/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/personality-targeted-interventions-delay-uptake-of-drinking-and-decrease-risk-of-alcohol-related-problems-when-delivered-by-teachers/#comments</comments>
		<pubDate>Mon, 03 Oct 2011 15:33:25 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Education Sector (Papers)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7816</guid>
		<description><![CDATA[O&#8217;Leary-Barrett M., Mackie C.J., Castellanos-Ryan N. et al. Addressing the substance use promoting tendencies of the personality traits of London secondary school pupils at particular risk of substance misuse led to fewer drinking and, among the drinkers, fewer drinking heavily. The study showed that school staff could effectively conduct the focus group interventions. Summary An [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;"></p>
<p>O&#8217;Leary-Barrett M., Mackie C.J., Castellanos-Ryan N. et al. </p>
<p>Addressing the substance use promoting tendencies of the personality traits of London secondary school pupils at particular risk of substance misuse led to fewer drinking and, among the drinkers, fewer drinking heavily. The study showed that school staff could effectively conduct the focus group interventions.<br />
Summary An alternative to prevention approaches applied to all children whatever their risk levels, the Preventure programme is a short intervention which targets youngsters who score highly on four personality dimensions which make different kinds of early-onset substance use and other risky behaviours particularly rewarding or hard to resist. As assessed by the Substance Use Risk Profile Scale personality questionnaire, these traits are:<br />
Hopelessness A tendency to unhappiness, depression and feeling a failure, feelings relieved by intoxication;<br />
Anxiety-sensitivity Fear of anxiety-related bodily sensations due to beliefs that such sensations will lead to catastrophic outcomes, for which substance use can represent a form of self-medication;<br />
Impulsivity An inability to restrain seeking gratification in the presence of immediate rewards (such as the feelings available through substance use) despite longer term negative consequences; and<br />
Sensation-seeking Desire for intense and novel experiences, which can be expressed as a desire to &#8216;get high&#8217; through drugtaking or heavy drinking.</p>
<p><strong>The Preventure intervention<br />
</strong><br />
The manualised Preventure intervention addresses these risk factors by drawing on psychoeducational approaches, motivational enhancement therapy, and cognitive-behavioural therapy, applied to real-life scenarios shared by high-risk young people in Britain. As implemented in the featured study, it occupied two 90-minute focus groups of on average six pupils led by two trained facilitators. Groups were formed of pupils who shared elevated scores on the same personality dimension, and the variant of the intervention applied to that group particularly targeted that dimension and the associated risks. In the first session participants were guided in a goal-setting exercise to enhance motivation to change behaviour, taught about the personality dimension and how it can predispose to problematic coping behaviours, and guided in breaking down personal experience according to the physical, cognitive, and behavioural components of an emotional response. All the exercises were specific to the personality risk factors identified in the children. The second session involved identifying and challenging personality-specific cognitive distortions which lead to problematic behaviours.<br />
Preventure interventions have been found to prevent the onset and escalation of drug use over the following two years, but so far only as delivered by skilled research therapists. The featured study tested whether school staff, with moderate levels of training and expertise and competing responsibilities, could be trained to effectively deliver this unfamiliar interactive, small group intervention. Another issue was whether pupils would be open with adults who may hold disciplinary positions. On the other hand, it was possible that the teachers&#8217; familiarity with their pupils would aid participation, and provide a platform for later addressing individual problems.<br />
The study<br />
Across nine randomly selected London boroughs, 21 randomly selected secondary schools were asked to join the study and randomly allocated to the Preventure intervention or to act as control schools which simply carried on (as all the schools had to) with the drug education components required by the national curriculum. Three schools could not be included in the featured analyses, leaving 18 schools and 2506 of the original 3021 year nine (ages 13–14) pupils. Of these pupils, 1159 or just under half scored as high risk on the Substance Use Risk Profile Scale; their responses were the basis for the featured report. 1008 could be followed up six months later; the probable responses of the remainder were estimated on the basis of earlier assessments and other data.<br />
School staff running the Preventure intervention were trained in a three-day workshop followed by at least four hours of supervised practice and feedback on their performance while practising the full intervention with year 10 pupils from their schools. Though broader and longer-term outcomes are being assessed, the featured report focused on drinking six months post-intervention.</p>
<p><strong>Main findings<br />
</strong><br />
Over 8 in 10 of the school staff members in the study completed training and supervision and qualified to facilitate the intervention. Each conducted on average six intervention sessions. Researchers observed at least one session by each facilitator. They judged that two thirds of the sessions had covered most of the core components of the intervention, and that two thirds also had been delivered in ways which embodied the required counselling skills of listening, enabling, involving the entire group, and being inquisitive and empathic. Facilitators themselves were all rated as at least satisfactory as cognitive therapists.</p>
<p>As expected, at the start of the study more of the high risk than the lower risk pupils (41% v. 32% ) had drunk alcohol in the past six months and more too had drunk heavily during that period (22% v. 12% ), defined as at least five drinks at one sitting for boys and four for girls. Six months later and compared to control schools, in schools allocated to Preventure the increase in the proportion of high risk pupils who were drinking was significantly less steep (rising from 43% to just 50% v. from 38% to 57%)  chart. Narrowly missing statistical significance was a similar disparity in trends in the proportion drinking heavily across the entire population of high risk pupils; in intervention schools this rose from 22% to 25%, in control schools, from 21% to 28%.<br />
A second set of analyses focused on the four in ten high risk pupils drinking at the start of the study. Among these drinkers, the proportion later drinking heavily actually fell in Preventure schools (from 52% to 48%) but rose in control schools (from 54% to 63%), another statistically significant difference. They were also consuming less alcohol overall, and were less likely to report drink-related problems.<br />
These effects were comparable to those noted in previous trials of the intervention with specialist interventionists.</p>
<p><strong>The authors&#8217; conclusions<br />
</strong><br />
The was the first evaluation of a school-based personality-targeted intervention for substance misuse delivered by trained educational professionals. Compared with controls, the intervention was associated with significantly decreased drinking and drink-related problems six months later, and with fewer &#8216;binge&#8217; drinkers among participants drinking at the start of the study – a particularly high risk group for future substance misuse. The potential health benefits of this delayed uptake of drinking are substantial: a one-year delay can decrease the risk for future alcohol-related problems by 10%.<br />
These results replicate findings from personality-targeted intervention trials in the UK and Canada, but within an implementation model that has a higher likelihood of being adopted in the real world. The demonstration that trained and supervised school staff can achieve results comparable to specialist therapists means the intervention has the potential to become a sustainable school-based early prevention strategy with youth most at risk for developing future alcohol-related problems. However, it remains unclear whether ongoing expert supervision and/or performance and outcome feedback is required to maintain standards.<br />
Among baseline drinkers, this trial and others have found that just from four to six young people need to be allocated to the intervention in order to later prevent one from drinking heavily – a ratio much more favourable than typically found for &#8216;universal&#8217; prevention programmes which target all the young people in a population rather than just those at high risk, and which are typically of much longer duration.<br />
The possibility that it was simply a group intervention which was effective rather than the particular content of that intervention is contradicted by studies which have compared the Preventure intervention to alternative group sessions, and by general findings that few interventions decrease substance misuse. From a similar UK trial which found reduced use of illicit drugs, it also seems unlikely that Preventure pupils in the featured study substituted these for alcohol.<br />
In sum, the evidence appears to strongly support the use of this programme in schools, whether delivered by trained clinicians external to the school or trained school staff. However, implementations should include the expert training and supervision components unless and until it is shown that schools are able to deliver the interventions autonomously and effectively.<br />
 Relative to basic education without much if any intended prevention content, this and other studies have demonstrated substantial effects in delaying the onset of and retarding the growth of substance use. Few of the usual limitations on the generalisability of the findings to the normal run of schools apply to this study. Neither schools nor pupils were highly selected, all but a small proportion of sampled pupils were followed up, and the schools&#8217; own staff conducted the intervention. As the authors comment, an impediment to widespread implementation may be the availability of expert trainers and supervisors. Another may be the willingness of schools to release four staff for three days training each followed by hours of supervision, and to let them spend many more hours addressing non-academic issues with a subset of high risk pupils. What may help convince them will be further results from the study if these demonstrate impacts not just on drinking but on mental health, other substance use, conduct, and academic achievement.<br />
Among the findings is however the narrow failure to find a statistically significant impact on regular heavy or &#8216;binge&#8217; drinking across all high risk pupils rather than just among those already drinking at the start of the study – a finding which seems to reflect the dilution of the results due to the inclusion of pupils unlikely to go on to drink heavily. This finding almost certainly also means no significant impact on regular heavy drinking across all the pupils in the school. Drinking as such at these ages is a concern, but in the British context, even more so is teenage binge drinking. That the intervention could not register even a short-term impact on this priority concern will lessen its appeal.<br />
Its matching strategy above all distinguishes the featured intervention from other approaches. Plausibly, the developers argue that addressing each individual&#8217;s particular personality vulnerability to substance use should more effectively reduce or prevent that use than a more scatter-gun or generic approach. However, this remains to be convincingly demonstrated in studies which have offered essentially the same intervention, but not matched to the individual&#8217;s personality. It is possible that the advance made by the broad matching strategy embodied in the intervention&#8217;s manuals is not sufficiently great to improve on the &#8216;natural&#8217; and possibly more fine-tune matching which occurs as a sensitive therapist or counsellor adapts their interpersonal style and the content of the intervention to the individual. Also at issue is the persistence of the effects past the first six months.<br />
Other studies of the featured intervention<br />
This study is one of the latest in a series investigating the same or similar interventions co-authored by the intervention&#8217;s developers. Given that allegiance to an intervention is associated with finding that it works, a fully independent demonstration by researchers with no personal investment in the intervention is desirable. Despite this, the body of work to date is methodologically sound, often convincing in its results and based on a plausible theory of how the intervention should work.<br />
Among the British trials was another in London, but this time of a highly selected set of 347 schoolchildren counselled by a professional psychologist rather than school staff. As in the featured study, the intervention was associated with drinking reductions six months later, but these effects dissipated to insignificance over the next six months and remained so over the remainder of the two-year follow-up. This was in contrast to drink-related problems, experience of which increased over the first six months in the control group and remained higher than in the intervention group over the follow-up period.<br />
Another similar study in London found that over the following six months the intervention delayed the expected increase in drinking among high risk pupils over the first six months of the follow-up, though again, by a year there was no significant difference in the drinking behaviour of pupils who had or had not been allocated to the intervention. The same trial found reduced uptake of cocaine and other drug use and a reduced frequency of drug use overall (but not cannabis in particular) over the two-year follow-up. In Canada too, the intervention was found to result in at least short-term (four months) drinking reductions in secondary school pupils.<br />
As well as these trials among schoolchildren, earlier versions of the intervention have been trialled with adults and young adults. One trial focused on female undergraduates in Canada characterised by one of the personality traits investigated in the featured study – anxiety-sensitivity. Over the next 10 weeks, drink-related problems were relatively lower (but not quite to a statistically significant degree) among women allocated to an intervention targeted to their personality profiles compared to those allocated to a &#8216;placebo&#8217; group intervention, but drinking itself was unaffected. Another study involved largely alcohol-dependent women in Canada aged 30 to 50 recruited via ads asking them to get in contact if they were concerned about their drinking or prescription drug use. A variant of the featured intervention was compared to a control intervention involving a motivational film on substance use problems and a supportive discussion with a therapist, a combination which it fairly consistently outperformed in reducing substance use. However, there were no statistically significant findings (though there were tendencies in this direction) indicating that the intervention bettered another intervention similar in every other way except that the content was not matched to the individual&#8217;s personality profile. These findings call in to question the matching strategy which above all distinguishes the featured intervention from other approaches.</p>
<p><em>Source.:   www.findings.org.uk  16 August 2011<br />
Journal of the American Academy of Child &#038; Adolescent Psychiatry: 2010, 49(9), p. 954–963<br />
</em><br />
</span></p>
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		<title>Public Policy Statement: Definition of Addiction</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/public-policy-statement-definition-of-addiction/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/public-policy-statement-definition-of-addiction/#comments</comments>
		<pubDate>Mon, 03 Oct 2011 15:27:13 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Addiction (Papers)]]></category>
		<category><![CDATA[Effects of Drugs (Papers)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7813</guid>
		<description><![CDATA[Addiction is a primary, chronic disease of brain reward, motivation, memory and related circuitry. Addiction affects neurotransmission and interactions within reward structures of the brain, including the nucleus accumbens, anterior cingulate cortex, basal forebrain and amygdala, such that motivational hierarchies are altered and addictive behaviors, which may or may not include alcohol and other drug [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;"></p>
<p>Addiction is a primary, chronic disease of brain reward, motivation, memory and related circuitry. Addiction affects neurotransmission and interactions within reward structures of the brain, including the nucleus accumbens, anterior cingulate cortex, basal forebrain and amygdala, such that motivational hierarchies are altered and addictive behaviors, which may or may not include alcohol and other drug use, supplant healthy, self-care related behaviors. Addiction also affects neurotransmission and interactions between cortical and hippocampal circuits and brain reward structures, such that the memory of previous exposures to rewards (such as food, sex, alcohol and other drugs) leads to a biological and behavioral response to external cues, in turn triggering craving and/or engagement in addictive behaviors.<br />
The neurobiology of addiction encompasses more than the neurochemistry of reward.1 The frontal cortex of the brain and underlying white matter connections between the frontal cortex and circuits of reward, motivation and memory are fundamental in the manifestations of altered impulse control, altered judgment, and the dysfunctional pursuit of rewards (which is often experienced by the affected person as a desire to “be normal”) seen in addiction&#8211;despite cumulative adverse consequences experienced from engagement in substance use and other addictive behaviors. The frontal lobes are important in inhibiting impulsivity and in assisting individuals to appropriately delay gratification. When persons with addiction manifest problems in deferring gratification, there is a neurological locus of these problems in the frontal cortex. Frontal lobe morphology, connectivity and functioning are still in the process of maturation during adolescence and young adulthood, and early exposure to substance use is another significant factor in the development of addiction. Many neuroscientists believe that developmental morphology is the basis that makes early-life exposure to substances such an important factor.<br />
Genetic factors account for about half of the likelihood that an individual will develop addiction. Environmental factors interact with the person’s biology and affect the extent to which genetic factors exert their influence. Resiliencies the individual acquires (through parenting or later life experiences) can affect the extent to which genetic predispositions lead to the behavioral and other manifestations of addiction. Culture also plays a role in how addiction becomes actualized in persons with biological vulnerabilities to the development of addiction.<br />
Other factors that can contribute to the appearance of addiction, leading to its characteristic bio-psycho-socio-spiritual manifestations, include:<br />
a.	The presence of an underlying biological deficit in the function of reward circuits, such that drugs and behaviors which enhance reward function are preferred and sought as reinforcers;<br />
b.	The repeated engagement in drug use or other addictive behaviors, causing neuroadaptation in motivational circuitry leading to impaired control over further drug use or engagement in addictive behaviors;<br />
c.	Cognitive and affective distortions, which impair perceptions and compromise the ability to deal with feelings, resulting in significant self-deception;<br />
d.	Disruption of healthy social supports and problems in interpersonal relationships which impact the development or impact of resiliencies;<br />
e.	Exposure to trauma or stressors that overwhelm an individual’s coping abilities;<br />
f.	Distortion in meaning, purpose and values that guide attitudes, thinking and behavior;<br />
g.	Distortions in a person’s connection with self, with others and with the transcendent (referred to as God by many, the Higher Power by 12-steps groups, or higher consciousness by others); and<br />
h.	The presence of co-occurring psychiatric disorders in persons who engage in substance use or other addictive behaviors.<br />
Addiction is characterized by2:<br />
a.	Inability to consistently Abstain;<br />
b.	Impairment in Behavioral control;<br />
c.	Craving; or increased “hunger” for drugs or rewarding experiences;<br />
d.	Diminished recognition of significant problems with one’s behaviors and interpersonal relationships; and<br />
e.	A dysfunctional Emotional response.<br />
The power of external cues to trigger craving and drug use, as well as to increase the frequency of engagement in other potentially addictive behaviors, is also a characteristic of addiction, with the hippocampus being important in memory of previous euphoric or dysphoric experiences, and with the amygdala being important in having motivation concentrate on selecting behaviors associated with these past experiences.<br />
Although some believe that the difference between those who have addiction, and those who do not, is the quantity or frequency of alcohol/drug use, engagement in addictive behaviors (such as gambling or spending)3, or exposure to other external rewards (such as food or sex), a characteristic aspect of addiction is the qualitative way in which the individual responds to such exposures, stressors and environmental cues. A particularly pathological aspect of the way that persons with addiction pursue substance use or external rewards is that preoccupation with, obsession with and/or pursuit of rewards (e.g., alcohol and other drug use) persist despite the accumulation of adverse consequences. These manifestations can occur compulsively or impulsively, as a reflection of impaired control.<br />
Persistent risk and/or recurrence of relapse, after periods of abstinence, is another fundamental feature of addiction. This can be triggered by exposure to rewarding substances and behaviors, by exposure to environmental cues to use, and by exposure to emotional stressors that trigger heightened activity in brain stress circuits.4<br />
In addiction there is a significant impairment in executive functioning, which manifests in problems with perception, learning, impulse control, compulsivity, and judgment. People with addiction often manifest a lower readiness to change their dysfunctional behaviors despite mounting concerns expressed by significant others in their lives; and display an apparent lack of appreciation of the magnitude of cumulative problems and complications. The still developing frontal lobes of adolescents may both compound these deficits in executive functioning and predispose youngsters to engage in “high risk” behaviors, including engaging in alcohol or other drug use. The profound drive or craving to use substances or engage in apparently rewarding behaviors, which is seen in many patients with addiction, underscores the compulsive or avolitional aspect of this disease. This is the connection with “powerlessness” over addiction and “unmanageability” of life, as is described in Step 1 of 12 Steps programs.<br />
Addiction is more than a behavioral disorder. Features of addiction include aspects of a person’s behaviors, cognitions, emotions, and interactions with others, including a person’s ability to relate to members of their family, to members of their community, to their own psychological state, and to things that transcend their daily experience.<br />
Behavioral manifestations and complications of addiction, primarily due to impaired control, can include:<br />
a.	Excessive use and/or engagement in addictive behaviors, at higher frequencies and/or quantities than the person intended, often associated with a persistent desire for and unsuccessful attempts at behavioral control;<br />
b.	Excessive time lost in substance use or recovering from the effects of substance use and/or engagement in addictive behaviors, with significant adverse impact on social and occupational functioning (e.g. the development of interpersonal relationship problems or the neglect of responsibilities at home, school or work);<br />
c.	Continued use and/or engagement in addictive behaviors, despite the presence of persistent or recurrent physical or psychological problems which may have been caused or exacerbated by substance use and/or related addictive behaviors;<br />
d.	A narrowing of the behavioral repertoire focusing on rewards that are part of addiction; and<br />
e.	An apparent lack of ability and/or readiness to take consistent, ameliorative action despite recognition of problems.<br />
Cognitive changes in addiction can include:<br />
a.	Preoccupation with substance use;<br />
b.	Altered evaluations of the relative benefits and detriments associated with drugs or rewarding behaviors; and<br />
c.	The inaccurate belief that problems experienced in one’s life are attributable to other causes rather than being a predictable consequence of addiction.<br />
Emotional changes in addiction can include:<br />
a.	Increased anxiety, dysphoria and emotional pain;<br />
b.	Increased sensitivity to stressors associated with the recruitment of brain stress systems, such that “things seem more stressful” as a result; and<br />
c.	Difficulty in identifying feelings, distinguishing between feelings and the bodily sensations of emotional arousal, and describing feelings to other people (sometimes referred to as alexithymia).<br />
The emotional aspects of addiction are quite complex. Some persons use alcohol or other drugs or pathologically pursue other rewards because they are seeking “positive reinforcement” or the creation of a positive emotional state (“euphoria”). Others pursue substance use or other rewards because they have experienced relief from negative emotional states (“dysphoria”), which constitutes “negative reinforcement.“ Beyond the initial experiences of reward and relief, there is a dysfunctional emotional state present in most cases of addiction that is associated with the persistence of engagement with addictive behaviors. The state of addiction is not the same as the state of intoxication. When anyone experiences mild intoxication through the use of alcohol or other drugs, or when one engages non-pathologically in potentially addictive behaviors such as gambling or eating, one may experience a “high”, felt as a “positive” emotional state associated with increased dopamine and opioid peptide activity in reward circuits. After such an experience, there is a neurochemical rebound, in which the reward function does not simply revert to baseline, but often drops below the original levels. This is usually not consciously perceptible by the individual and is not necessarily associated with functional impairments.<br />
Over time, repeated experiences with substance use or addictive behaviors are not associated with ever increasing reward circuit activity and are not as subjectively rewarding. Once a person experiences withdrawal from drug use or comparable behaviors, there is an anxious, agitated, dysphoric and labile emotional experience, related to suboptimal reward and the recruitment of brain and hormonal stress systems, which is associated with withdrawal from virtually all pharmacological classes of addictive drugs. While tolerance develops to the “high,” tolerance does not develop to the emotional “low” associated with the cycle of intoxication and withdrawal. Thus, in addiction, persons repeatedly attempt to create a “high”&#8211;but what they mostly experience is a deeper and deeper “low.” While anyone may “want” to get “high”, those with addiction feel a “need” to use the addictive substance or engage in the addictive behavior in order to try to resolve their dysphoric emotional state or their physiological symptoms of withdrawal. Persons with addiction compulsively use even though it may not make them feel good, in some cases long after the pursuit of “rewards” is not actually pleasurable.5 Although people from any culture may choose to “get high” from one or another activity, it is important to appreciate that addiction is not solely a function of choice. Simply put, addiction is not a desired condition.<br />
As addiction is a chronic disease, periods of relapse, which may interrupt spans of remission, are a common feature of addiction. It is also important to recognize that return to drug use or pathological pursuit of rewards is not inevitable.<br />
Clinical interventions can be quite effective in altering the course of addiction. Close monitoring of the behaviors of the individual and contingency management, sometimes including behavioral consequences for relapse behaviors, can contribute to positive clinical outcomes. Engagement in health promotion activities which promote personal responsibility and accountability, connection with others, and personal growth also contribute to recovery. It is important to recognize that addiction can cause disability or premature death, especially when left untreated or treated inadequately.<br />
The qualitative ways in which the brain and behavior respond to drug exposure and engagement in addictive behaviors are different at later stages of addiction than in earlier stages, indicating progression, which may not be overtly apparent. As is the case with other chronic diseases, the condition must be monitored and managed over time to:<br />
a.	Decrease the frequency and intensity of relapses;<br />
b.	Sustain periods of remission; and<br />
c.	Optimize the person’s level of functioning during periods of remission.<br />
In some cases of addiction, medication management can improve treatment outcomes. In most cases of addiction, the integration of psychosocial rehabilitation and ongoing care with evidence-based pharmacological therapy provides the best results. Chronic disease management is important for minimization of episodes of relapse and their impact. Treatment of addiction saves lives †<br />
Addiction professionals and persons in recovery know the hope that is found in recovery. Recovery is available even to persons who may not at first be able to perceive this hope, especially when the focus is on linking the health consequences to the disease of addiction. As in other health conditions, self-management, with mutual support, is very important in recovery from addiction. Peer support such as that found in various “self-help” activities is beneficial in optimizing health status and functional outcomes in recovery. ‡<br />
Recovery from addiction is best achieved through a combination of self-management, mutual support, and professional care provided by trained and certified professionals.</p>
<p><em>Source:  www.asam.org  April 2011</em></p>
<p>Explanatory footnotes:<br />
1. The neurobiology of reward has been well understood for decades, whereas the neurobiology of addiction is still being explored. Most clinicians have learned of reward pathways including projections from the ventral tegmental area (VTA) of the brain, through the median forebrain bundle (MFB), and terminating in the nucleus accumbens (Nuc Acc), in which dopamine neurons are prominent. Current neuroscience recognizes that the neurocircuitry of reward also involves a rich bi-directional circuitry connecting the nucleus accumbens and the basal forebrain. It is the reward circuitry where reward is registered, and where the most fundamental rewards such as food, hydration, sex, and nurturing exert a strong and life-sustaining influence. Alcohol, nicotine, other drugs and pathological gambling behaviors exert their initial effects by acting on the same reward circuitry that appears in the brain to make food and sex, for example, profoundly reinforcing. Other effects, such as intoxication and emotional euphoria from rewards, derive from activation of the reward circuitry. While intoxication and withdrawal are well understood through the study of reward circuitry, understanding of addiction requires understanding of a broader network of neural connections involving forebrain as well as midbrain structures. Selection of certain rewards, preoccupation with certain rewards, response to triggers to pursue certain rewards, and motivational drives to use alcohol and other drugs and/or pathologically seek other rewards, involve multiple brain regions outside of reward neurocircuitry itself.<br />
2. These five features are not intended to be used as “diagnostic criteria” for determining if addiction is present or not. Although these characteristic features are widely present in most cases of addiction, regardless of the pharmacology of the substance use seen in addiction or the reward that is pathologically pursued, each feature may not be equally prominent in every case. The diagnosis of addiction requires a comprehensive biological, psychological, social and spiritual assessment by a trained and certified professional.<br />
3. In this document, the term &#8220;addictive behaviors&#8221; refers to behaviors that are commonly rewarding and are a feature in many cases of addiction. Exposure to these behaviors, just as occurs with exposure to rewarding drugs, is facilitative of the addiction process rather than causative of addiction. The state of brain anatomy and physiology is the underlying variable that is more directly causative of addiction. Thus, in this document, the term “addictive behaviors” does not refer to dysfunctional or socially disapproved behaviors, which can appear in many cases of addiction. Behaviors, such as dishonesty, violation of one’s values or the values of others, criminal acts etc., can be a component of addiction; these are best viewed as complications that result from rather than contribute to addiction.<br />
4. The anatomy (the brain circuitry involved) and the physiology (the neuro-transmitters involved) in these three modes of relapse (drug- or reward-triggered relapse vs. cue-triggered relapse vs. stress-triggered relapse) have been delineated through neuroscience research.<br />
Relapse triggered by exposure to addictive/rewarding drugs, including alcohol, involves the nucleus accumbens and the VTA-MFB-Nuc Acc neural axis (the brain&#8217;s mesolimbic dopaminergic &#8220;incentive salience circuitry&#8221;&#8211;see footnote 2 above). Reward-triggered relapse also is mediated by glutamatergic circuits projecting to the nucleus accumbens from the frontal cortex.<br />
Relapse triggered by exposure to conditioned cues from the environment involves glutamate circuits, originating in frontal cortex, insula, hippocampus and amygdala projecting to mesolimbic incentive salience circuitry.<br />
Relapse triggered by exposure to stressful experiences involves brain stress circuits beyond the hypothalamic-pituitary-adrenal axis that is well known as the core of the endocrine stress system. There are two of these relapse-triggering brain stress circuits – one originates in noradrenergic nucleus A2 in the lateral tegmental area of the brain stem and projects to the hypothalamus, nucleus accumbens, frontal cortex, and bed nucleus of the stria terminalis, and uses norepinephrine as its neurotransmitter; the other originates in the central nucleus of the amygdala, projects to the bed nucleus of the stria terminalis and uses corticotrophin-releasing factor (CRF) as its neurotransmitter.<br />
5. Pathologically pursuing reward (mentioned in the Short Version of this definition) thus has multiple components. It is not necessarily the amount of exposure to the reward (e.g., the dosage of a drug) or the frequency or duration of the exposure that is pathological. In addiction, pursuit of rewards persists, despite life problems that accumulate due to addictive behaviors, even when engagement in the behaviors ceases to be pleasurable. Similarly, in earlier stages of addiction, or even before the outward manifestations of addiction have become apparent, substance use or engagement in addictive behaviors can be an attempt to pursue relief from dysphoria; while in later stages of the disease, engagement in addictive behaviors can persist even though the behavior no longer provides relief.</p>
<p></span></p>
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		<title>The Facts on Marijuana</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/the-facts-on-marijuana/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/the-facts-on-marijuana/#comments</comments>
		<pubDate>Mon, 03 Oct 2011 15:22:44 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Effects of Drugs (Papers)]]></category>
		<category><![CDATA[Medicine and Marijuana]]></category>
		<category><![CDATA[Prevention (Papers)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7810</guid>
		<description><![CDATA[Several jurisdictions in the U.S. have taken steps toward decriminalizing marijuana possession for personal use or when prescribed by a physician for medicinal purposes. Other jurisdictions have pending ballot initiatives or legislative bills proposing such changes in the law. The Board of Directors of the National Association of Drug Court Professionals (NADCP) has determined that [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;"></p>
<p>Several jurisdictions in the U.S. have taken steps toward decriminalizing marijuana possession for personal use or when prescribed by a physician for medicinal purposes. Other jurisdictions have pending ballot initiatives or legislative bills proposing such changes in the law.<br />
The Board of Directors of the National Association of Drug Court Professionals (NADCP) has determined that it is essential for drug court practitioners to be fully and objectively informed about the effects of marijuana on their participants and the public at-large. This document briefly reviews the scientific evidence concerning the effects of marijuana. </p>
<p><strong>Incarceration for Marijuana Possession</strong></p>
<p>It is exceedingly rare to be incarcerated in the U.S. for the use or possession of marijuana. According to the National Center on Addiction &#038; Substance Abuse at Columbia University (CASA, 2010), less than 1 percent (0.9%) of jail and prison inmates in the U.S. were incarcerated for marijuana possession as their sole offense.<br />
Excluding jail detainees who may be held pending booking or release on bond, the rates are even lower. Prison inmates sentenced for marijuana possession account for 0.7 percent of state prisoners and 0.8 percent of federal prisoners (see Table). And, considering that many of those prisoners pled down from more serious charges, the true incarceration rate for marijuana possession can only be described as negligible.<br />
State Prisoners Federal Prisoners<br />
Marijuana offense only 1.6% N.R.<br />
Marijuana possession only 0.7% 0.8%<br />
First-time marijuana possession 0.3% N.R. </p>
<p><em>Source: Office of National Drug Control Policy, Who’s Really in Prison for Marijuana? [NCJ #204299] (citing BJS, 1999, Substance abuse and treatment, state and federal prisoners, 1997 [NCJ #172871]; U.S. Sentencing Commission, 2001 Sourcebook of Federal Sentencing Statistics). N.R. = not reported. 2 </em></p>
<p><strong>Addiction Potential<br />
</strong><br />
By the early 1990’s, the scientific community had concluded from rigorous laboratory and epidemiological studies that marijuana is physiologically and psychologically addictive. Every drug of abuse has what is called a dependence liability, which refers to the statistical probability that a person who uses that drug for nonmedical purposes will develop a compulsive addiction. Based upon several nationwide epidemiological studies, marijuana’s dependence liability has been reliably determined to be 8 to 10 percent (Anthony et al., 1994; Brook et al., 2008; Budney &#038; Moore, 2002; Kandel et al., 1997; Munsey, 2010; Wagner &#038; Anthony, 2002). This means that one out of every 10 to 12 people who use marijuana will become addicted to the drug.<br />
Importantly, the dependence liability of any drug increases with more frequent usage. Individuals who have used marijuana at least five times have a 20 to 30 percent likelihood of becoming addicted to the drug, and those who use it regularly have a 40 percent likelihood of becoming addicted (Budney &#038; Moore, 2002).<br />
The hallmark feature of physical addiction is the experience of uncomfortable or painful withdrawal symptoms whenever levels of the substance decline in the bloodstream. This is, in part, what drives addicts to continue abusing drugs or alcohol despite suffering severe negative medical, legal and interpersonal consequences. Carefully controlled, rigorous laboratory studies have proven beyond further dispute that marijuana addiction is associated with a clinically significant withdrawal syndrome. When marijuana-addicted individuals stop using the drug, they experience symptoms of irritability, anger, cravings, decreased appetite, insomnia, interpersonal hypersensitivity, yawning and/or fatigue (Budney et al., 2001; Preuss et al., 2010). In fact, the features and severity of the marijuana withdrawal syndrome are virtually indistinguishable from those of nicotine (cigarette) withdrawal.<br />
A second hallmark feature of addiction is psychosocial dysfunction resulting from repeated use of the substance. The most commonly diagnosed symptoms of psychosocial dysfunction among marijuana addicts include persistent procrastination, bad or guilty feelings, low productivity, low self-confidence, interpersonal or family conflicts, memory problems and financial difficulties (Budney &#038; Moore, 2002; NIDA, 2005). This constellation of symptoms has been collectively referred to as an “amotivational syndrome” (e.g., Hubbard et al., 1999) because marijuana abusers tend to be characteristically languid and often achieve considerably below their true intellectual potentials.<br />
Based on this substantial body of empirical research, the American Psychiatric Association (APA) has long recognized cannabis dependence as a valid and reliable psychiatric disorder in the Diagnostic and Statistical Manual of Mental Disorders (DSM). The DSM is the official psychiatric diagnostic classification system in the U.S. A diagnosis of cannabis dependence has been continuously included in the 3rd and 4th editions of the DSM since 1980 (APA, 1980, 1987, 1994, 2000). In the soon-to-be published 5th edition of the DSM, a cannabis withdrawal syndrome will now also be officially recognized as part of the diagnostic criteria for cannabis dependence.  </p>
<p><strong>Medical Harm<br />
</strong><br />
In many respects, smoked marijuana has the potential to be as, or more, harmful than cigarettes. Although marijuana does not contain nicotine, it does contain 50 to 70 percent more carcinogenic compounds, including tar, than cigarettes (NIDA, 2005; Hubbard et al., 1999). Marijuana also produces high levels of a particular enzyme which converts certain hydrocarbons into their carcinogenic or malignant forms (NIDA, 2005).<br />
Although gram for gram, marijuana smoke is clearly more carcinogenic than cigarette smoke, it is difficult to predict whether actual incidence rates of induced cancers are likely to be as high as they are for cigarettes. On one hand, cannabis smokers tend to use the drug on fewer occasions than cigarette smokers. On the other hand, they typically inhale larger amounts of the drug per occasion, hold the smoke in their lungs for longer intervals of time, and are unlikely to employ filters. This makes it difficult to compare the predicted magnitudes of the harms. The best estimate from the National Institutes of Health (NIH) is that a person who smokes five marijuana cigarettes per week is likely to be inhaling as many cancer-causing chemicals as one who smokes a full pack of cigarettes every day.1<br />
 See U.S. Dept. of Justice, Drug Enforcement Administration, Exposing the myth of medical marijuana: The facts. Available at http://www.justice.gov/dea/ongoing/marijuanap.html.<br />
Like nicotine, cannabis increases heart rate, alters blood pressure, can induce tachycardia (rapid or irregular heartbeat), increases myocardial (heart) stress, decreases oxygen levels in the circulatory system, and exacerbates angina (Hubbard et al., 1999). As a result, a person’s risk of a heart attack is increased four-fold during the first hour after smoking marijuana (NIDA, 2005).<br />
There is no question that regular marijuana use is associated with a wide spectrum of chronic respiratory ailments. A nationally representative study of 6,728 adults found heavy marijuana use to be substantially associated with chronic bronchitis, coughing on most days, wheezing, abnormal chest sounds and increased phlegm (Moore et al., 2005).<br />
Marijuana has undisputed negative effects on cognitive functioning, including memory, learning and motor coordination. These negative effects persist long after the period of acute intoxication, averaging approximately 30 days of residual cognitive impairment (Bolla et al., 2002; NIDA, 2005; Pope et al., 2001). This means that individuals are apt to wrongly believe they are capable of performing critical tasks, such as driving a car, operating heavy machinery, caring for children or solving work-related intellectual problems, when in fact they may be performing in the mildly to moderately impaired range of functioning.<br />
Like any drug, marijuana’s negative effects tend to be most pronounced in elderly persons, individuals with chronic medical illnesses, and those with compromised immune systems. This is of particular concern given that marijuana is being specifically touted for “medicinal” use by elderly patients, cancer patients, and those with immunodeficiency<br />
syndromes such as HIV/AIDS (e.g., Munsey, 2010). Rather than benefiting such individuals, marijuana has the serious potential to further suppress or compromise their immune systems and exacerbate the disease process (NIDA, 2005). </p>
<p><strong>Medicinal Effects<br />
</strong><br />
Marijuana is a “Schedule I” drug according to the Drug Enforcement Administration (DEA), meaning it has a high abuse potential and no recognized medical indication. However, the Food and Drug Administration (FDA) has approved a particular ingredient within marijuana (THC) in a non-smoked form for certain medical indications, such as for treatment of nausea, vomiting and poor appetite. Recent studies have also supported its use in treating chronic neuropathic pain (e.g., Munsey, 2010).<br />
To date, research indicates that oral THC (when administered at adequate doses) is as effective as smoked marijuana in achieving these therapeutic effects (e.g., Munsey, 2010). Anecdotal testimonials are the only evidence favoring smoked marijuana over oral THC for therapeutic purposes. Further research is called for to determine whether other compounds within marijuana might have medicinal properties as well, but at this juncture any such indications are purely experimental and speculative.<br />
Regardless, smoked marijuana could no more be considered a “medication” than cigarettes or alcohol. Although cigarettes and alcohol have undeniable effects that many people may find palliative (such as alleviating short-term stress), they are very “dirty” drugs. This means they contain dozens, if not hundreds, of other physiologically active compounds which are irrelevant to their palliative effects and may actually work at cross-purposes against those effects. For example, many people believe alcohol and nicotine lower their stress level, but in fact these drugs are proven to increase anxiety, lower stress tolerance and exacerbate insomnia over the longer term. These drugs are also associated with a host of serious medical conditions, including cancer, heart disease, liver disease and respiratory illnesses. For these reasons, physicians would rarely, if ever, “prescribe” these drugs to treat a medical condition.<br />
More research is needed to isolate the potential therapeutic effects of specific compounds within marijuana, and to determine how to administer those compounds in a manner that is medically safe and does not threaten to cause heart, lung and other diseases. Administering the “dirty” form of the drug would never be a legitimate medical end-goal. </p>
<p><strong>Impact on Crime<br />
</strong><br />
Two recent meta-analyses (advanced statistical procedures) have concluded that marijuana use during adolescence or young adulthood significantly predicts later involvement in criminal activity and criminal arrests (Bennett et al., 2008; Pedersen &#038; Skardhamar, 2010). The risk of criminal involvement was determined to be between 1.5 and 3.0 times greater for cannabis users than for non-users. 5 The results suggest that, all else being equal, cannabis users are at a statistically increased risk for associating with antisocial individuals, engaging in illegal conduct, and eventually getting a criminal record. </p>
<p><strong>Conclusion<br />
</strong><br />
Marijuana is an intoxicating and addictive drug that poses serious medical risks akin to those of nicotine and alcohol. Although some physicians may consider it to have palliative indications, no national or regional medical or scientific organization recognizes marijuana as a medicine in its raw or smoked form.<br />
If marijuana becomes decriminalized or legalized in a given jurisdiction, this does not necessarily require drug court practitioners to abide its usage by their participants. The courts have long recognized restrictions on the use of a legal intoxicating substance (i.e., alcohol) to be a reasonable condition of bond or probation where the offender has a history of illicit drug involvement.  If there is a rational basis for believing cannabis use could threaten public safety or prevent the offender from returning to court for adjudication, appellate courts are likely to uphold such restrictions in the drug court context.<br />
Individuals who have a valid medical prescription for marijuana present a more challenging issue, but one that is probably also not insurmountable. Under such circumstances, the judge might subpoena the prescribing physician to testify or respond to written inquiries about the medical justification for the prescription. In addition, the court may be authorized by the rules of evidence or rules of criminal procedure to engage an independent medical expert to review the case and offer a medical recommendation or opinion. Having a Board-certified addiction psychiatrist on hand to advise the drug court judge may provide probative evidence about whether a particular marijuana prescription is medically necessary or indicated.<br />
It remains an open question what degree of deference appellate courts are likely to give to the conclusions of a treating physician. In the absence of clear precedent, the best course of action is to develop a factual record and make a particularized decision in each case about the medical necessity for the prescription and the rationale for restricting marijuana usage during the term of criminal justice supervision.<br />
If judges make these decisions based on a reasonable interpretation of medical evidence presented by qualified experts, it seems unlikely that drug courts — which were specifically designed to treat seriously addicted individuals — could not restrict access to an intoxicating and addictive drug as a condition of criminal justice supervision.</p>
<p><strong>About NADCP<br />
</strong><br />
It takes innovation, teamwork and strong judicial leadership to achieve success when addressing drug-using offenders in a community. That’s why since 1994 the National Association of Drug Court Professionals (NADCP) has worked tirelessly at the national, state and local level to create and enhance Drug Courts, which use a combination of accountability and treatment to compel and support drug-using offenders to change their lives.<br />
Now an international movement, Drug Courts are the shining example of what works in the justice system. Today, there are over 2,400 Drug Courts operating in the U.S., and another thirteen countries have implemented the model. Drug Courts are widely applied to adult criminal cases, juvenile delinquency and truancy cases, and family court cases involving parents at risk of losing custody of their children due to substance abuse.<br />
Drug Court improves communities by successfully getting offenders clean and sober and stopping drug-related crime, reuniting broken families, intervening with juveniles before they embark on a debilitating life of addiction and crime, and reducing impaired driving.<br />
In the 20 years since the first Drug Court was founded in Miami/Dade County, Florida, more research has been published on the effects of Drug Courts than on virtually all other criminal justice programs combined. The scientific community has put Drug Courts under a microscope and concluded that Drug Courts significantly reduce drug abuse and crime and do so at far less expense than any other justice strategy.<br />
Such success has empowered NADCP to champion new generations of the Drug Court model. These include Veterans Treatment Courts, Reentry Courts, and Mental Health Courts, among others. Veterans Treatment Courts, for example, link critical services and provide the structure needed for veterans who are involved in the justice system due to substance abuse or mental illness to resume life after combat. Reentry Courts assist individuals leaving our nation’s jails and prisons to succeed on probation or parole and avoid a recurrence of drug abuse and<br />
Today, the award-winning NADCP is the premier national membership, training, and advocacy organization for the Drug Court model, representing over 27,000 multi-disciplinary justice professionals and community leaders. NADCP hosts the largest annual training conference on drugs and crime in the nation and provides 130 training and technical assistance events each year through its professional service branches, the National Drug Court Institute, the National Center for DWI Courts and the National Veterans Treatment Court Clearinghouse. NADCP publishes numerous scholastic and practical publications critical to the growth and fidelity of the Drug Court model and works tirelessly in the media, on Capitol Hill, and in state legislatures to improve the response of the American justice system to substance-abusing and mentally ill offenders through policy, legislation, and appropriations.<br />
For more information please visit us on the web at www.AllRise.org. </p>
<p><em>Source: National Association of Drug Court Professionals.  Sept. 2010<br />
</em><br />
</span></p>
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		<title>Newly Born, and Withdrawing From Painkillers</title>
		<link>http://drugprevent.org.uk/ppp/2011/10/newly-born-and-withdrawing-from-painkillers/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/10/newly-born-and-withdrawing-from-painkillers/#comments</comments>
		<pubDate>Mon, 03 Oct 2011 15:17:33 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Effects of Drugs (Papers)]]></category>
		<category><![CDATA[Social Affairs (Papers)]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7808</guid>
		<description><![CDATA[BANGOR, Me. — The mother got the call in the middle of the night: her 3-day-old baby was going through opiate withdrawal in a hospital here and had to start taking methadone, a drug best known for treating heroin addiction, to ease his suffering. The mother had abused prescription painkillers like OxyContin for the first [...]]]></description>
			<content:encoded><![CDATA[<p><span style="font-size: 10pt; font-family: Verdana;"></p>
<p>BANGOR, Me. — The mother got the call in the middle of the night: her 3-day-old baby was going through opiate withdrawal in a hospital here and had to start taking methadone, a drug best known for treating heroin addiction, to ease his suffering.<br />
The mother had abused prescription painkillers like OxyContin for the first 12 weeks of her pregnancy, buying them on the street in rural northern Maine, and then tried to quit cold turkey — a dangerous course, doctors say, that could have ended in miscarriage. The baby had seizures in utero as a result, and his mother, Tonya, turned to methadone treatment, with daily doses to keep her cravings and withdrawal symptoms at bay.<br />
As prescription drug abuse ravages communities across the country, doctors are confronting an emerging challenge: newborns dependent on painkillers. While methadone may have saved Tonya’s pregnancy, her son, Matthew, needed to be painstakingly weaned from it.    Infants like him may cry excessively and have stiff limbs, tremors, diarrhea and other problems that make their first days of life excruciating. Many have to stay in the hospital for weeks while they are weaned off the drugs, taxing neonatal units and driving the cost of their medical care into the tens of thousands of dollars.<br />
Like the cocaine-exposed babies of the 1980s, those born dependent on prescription opiates — narcotics that contain opium or its derivatives — are entering a world in which little is known about the long-term effects on their development. Few doctors are even willing to treat pregnant opiate addicts, and there is no universally accepted standard of care for their babies, partly because of the difficulty of conducting research on pregnant women and newborns.<br />
Those who do treat pregnant addicts face a jarring ethical quandary: they must weigh whether the harm inflicted by exposing a fetus to powerful drugs, albeit under medical supervision, is justifiable.   “I’ve had pharmacies that have just called back and said: ‘This lady’s pregnant. Why do you want me to fill this scrip? I can’t do that,’ ” said Dr. Craig Smith, a family practitioner in Bridgton, Me. “But when you stop and think about what actually happens during withdrawal and how violent it can be, that would certainly be not in the baby’s best interest.”<br />
Still, even doctors who advocate treating pregnant addicts have had moments of doubt.   “At first I was going, ‘Gosh, what am I doing?’ ” said Dr. Thomas Meek, a primary care physician in Auburn, Me. “ ‘Am I really helping these people?’ ”<br />
There are no national figures that document the extent of the problem, but interviews with doctors, researchers, social workers and women who abused painkillers while pregnant suggest that it has grown rapidly, especially in rural regions, where officials say such abuse is most common.<br />
In Maine, which has been especially plagued by prescription drug abuse, the number of newborns treated or watched for opiate withdrawal, known as neonatal abstinence syndrome, at the state’s two largest hospitals climbed to 276 in 2010 from about 70 in 2005. Hospitals in states including Florida and Ohio reported similar increases, and experts said the numbers were probably higher since pregnant women are rarely tested for drug use and many mothers do not admit to abusing opiates.<br />
Tonya, 24, said she was introduced to painkillers like OxyContin, Percocet and Vicodin while working the overnight shift at an industrial bakery an hour from her home. Everyone — including co-workers, the boyfriend she met on the job and their manager — was taking pills, she said.     “It was a lot easier to get through life and have energy,” Tonya said at Eastern Maine Medical Center here in January, holding Matthew a month after his birth. He was still being weaned off methadone.<br />
Before she was pregnant, Tonya said, she quickly became addicted, spending all of her money on pills bought on the street. She and her boyfriend, Josh, needed to stave off withdrawal and get through the day, she said.    Now that she is in treatment, Tonya, who like most mothers interviewed for this article did not want her last name used, said her focus was on Matthew. “We put him in this situation,” she said, “and we have to help him out of it.”<br />
‘How Little We Know’<br />
Rigorous studies on treating infant withdrawal are scarce, and the American Academy of Pediatrics has not published guidelines since 1998.    “It’s really remarkable how little we know about the effect of prescription drugs and even nonprescription drugs on the fetus,” said Dr. Nora D. Volkow, director of the National Institute for Drug Abuse. “There are real roadblocks in terms of helping us advance the field.”<br />
Dr. Mark L. Hudak, a neonatologist in Jacksonville, Fla., is helping to revise the pediatrics academy’s guidelines. “There are commonalities, but it’s not like you can go to a Web site that says, ‘This is what should be used by everyone,’ ” Dr. Hudak said. “No one knows what the best approach is.”<br />
Within states, every hospital that delivers babies exposed to painkillers may have its own approach. Eastern Maine treats affected newborns with tiny doses of methadone, while Maine Medical Center in Portland uses morphine combined with phenobarbital, a barbiturate that prevents seizures. Some hospitals are also experimenting with clonidine, a mild sedative that can relieve withdrawal symptoms.<br />
There is growing debate over treatment for pregnant women addicted to prescription drugs, in light of concerns over the effects on their babies. Many are slowly weaned from their dependence with methadone, the standard of care for decades. Methadone, when taken in prescribed doses, keeps a steady amount of opiate in the body, preventing withdrawal and drug cravings that occur when levels dip. But it, too, can be addictive and cause nagging side effects like drowsiness. And for addiction treatment, it can be obtained only at federally licensed clinics where most users have to report for a daily dose.<br />
A growing number of addicts are instead taking buprenorphine, another drug used to treat addiction that some studies suggest staves off drug cravings as effectively as methadone but is less likely to cause withdrawal in newborns. In rural areas of the nation, where methadone clinics are few, buprenorphine is considered a promising alternative because it can be prescribed by primary care doctors and taken at home.    But buprenorphine also appears not to work for some addicts.<br />
Still, a study published in December in The New England Journal of Medicine showed that babies whose mothers had taken buprenorphine required significantly less medication after birth and less time in the hospital than did babies whose mothers were treated with methadone. But researchers cautioned that exposure to buprenorphine in utero can still cause withdrawal symptoms and that further study was needed.    “We don’t want it misconstrued that buprenorphine is a miracle drug,” said Hendrée E. Jones, a Johns Hopkins University researcher and the study’s lead author.<br />
Even less is known about longer-term effects on babies exposed to painkillers, though in a second leg of their study, Dr. Jones and her fellow researchers plan to follow the 131 babies in the cohort until they turn 3.     A recent study by the Centers for Disease Control and Prevention found that babies exposed to opiates in utero, in this case legally prescribed painkillers, had slightly higher rates of birth defects, including congenital heart defects, glaucoma and spina bifida.<br />
Experts say that since many drug users also smoke and abuse alcohol, not to mention that they face extenuating circumstances like poverty, it is difficult to tease out the effects of each substance on their offspring.    “Most of the literature suggests consistently that the drug exposure itself is not the primary concern,” said Karol Kaltenbach, a professor at Jefferson Medical College in Philadelphia who studies addiction in pregnant women. “It’s the cumulative effect of the drug-using lifestyle — poverty, chaos in the home, domestic violence. All those things affect development.”<br />
Not all newborns exposed to opiates have severe enough withdrawal to need medicine; at Maine Medical Center since 2003, about 55 percent of babies exposed to buprenorphine and 80 percent of those exposed to methadone have needed treatment. But it is hard to predict which ones will need it: a newborn whose mother was on a high dose of either drug might need none, while a baby whose mother took a low dose might experience acute withdrawal.    Babies known to have been exposed to drugs are often kept in the hospital for at least five days because withdrawal symptoms usually do not set in immediately. Nurses examine them for a checklist of symptoms every few hours, assigning each baby a score that, if high enough, calls for treatment.<br />
“They don’t stop crying, they can’t settle down, they don’t relax,” said Geraldine Tamborelli, nursing director of the birthing unit at Maine Medical Center, which in 2010 diagnosed opiate withdrawal in 121 newborns. “They’re struggling in your arms instead of snuggling into you like a baby that is totally fine.”<br />
In the neonatal intensive care unit at Eastern Maine, Kendra, 3 days old, was sleeping in a dark, silent room one morning, away from the bustle and bright lights that can be especially irritating to babies going through withdrawal. Nurses frequently crept in to observe her, though, and by the afternoon her limbs had stiffened and she was crying excessively and having tremors; it was enough to begin treatment.    “This seems to be ramping up fairly quickly for her,” said Dr. Mark Brown, the hospital’s chief of pediatrics, “so the decision was to start treatment more quickly.”<br />
On the pediatric ward, Matthew started fussing while his mother, Tonya, talked to reporters that afternoon in January; his cry had a strange, reedy pitch that nurses say is common to babies with his condition. The small dose of methadone he had received gave him gas and heartburn, for which he was given two stomach medications. He also was on clonazepam, a muscle relaxant and anti-anxiety drug that helped him metabolize the methadone more slowly.<br />
Tonya said that at first she “didn’t believe in” methadone treatment during pregnancy and that doctors had to persuade her that it would not hurt her fetus. She had experienced wrenching withdrawal when she stopped using painkillers after learning she was pregnant, she said, and the doctors had warned her that “when I was feeling that bad, he was feeling 1,000 times worse.”    Tonya said that in a previous pregnancy, she quit using drugs altogether and miscarried a month later. “That was the last thing I wanted to happen this time,” she said.<br />
Avoiding Addicts, and Liability<br />
Treating drug-dependent mothers and babies is often lonely work, with little communication among the doctors who take it on. As Dr. Brown said, “My network for people who do this is really very small.”<br />
Dr. Mark R. Publicker, an addiction medicine specialist at Mercy Recovery Center in Westbrook, Me., is on a mission to get more of the state’s doctors to treat pregnant prescription drug abusers and more hospitals to deliver their babies. Only a handful of doctors here treat pregnant women with buprenorphine, Dr. Publicker said, partly because they fear liability and do not want to deal with addicts.   The fact that most hospitals will not deliver the babies makes doctors even less likely to treat the women.    “It’s mostly ignorance,” Dr. Publicker said. “It’s a concern that it’s a risky proposition and that they’re going to wind up with an ill baby.”<br />
In February, Dr. Smith persuaded Bridgton Hospital, which has only 25 beds, to deliver the babies of women on buprenorphine — a major victory, he said, because until then women in rural southwestern Maine had to drive an hour or more to Maine Medical to deliver.   Courtney, a patient of Dr. Smith’s who discovered she was pregnant while in jail for stealing OxyContin from her landlord, said buprenorphine treatment seemed the best of her bleak options.   “I just don’t want to mess up,” she said.<br />
Tonya, too, said she was determined to make things right for Matthew, who was five weeks old when she took him home to a trailer outside Bangor. He is off the methadone now and appears healthy, but Tonya still has to go to a methadone clinic in Bangor every day for her dose and resist the pressures to return to illicit drug use. Her boyfriend began using opiates as a young teenager, she said, and his father and grandmother abused OxyContin along with him.   “I’m proud that I changed my life,” Tonya said. “But at the same time, when you see your child in pain and you know your child is in pain because of a life decision you made, it’s the hardest thing in the world.” </p>
<p><em>Source:  New York Times April 9th 2011<br />
</em></span></p>
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		<title>Crystal Meth Detected In Newborns&#8217; Hair</title>
		<link>http://drugprevent.org.uk/ppp/2011/09/crystal-meth-detected-in-newborns-hair/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/09/crystal-meth-detected-in-newborns-hair/#comments</comments>
		<pubDate>Tue, 20 Sep 2011 19:10:57 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Methamphetamine/GHB/Hallucinogens/Oxycodone]]></category>
		<category><![CDATA[Parents]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7801</guid>
		<description><![CDATA[TORONTO, Nov. 2 &#8212; Methamphetamine can be detected in the hair of newborns whose mothers used the drug during pregnancy, researchers here have found. Action Points Note that this study shows that methamphetamine used during pregnancy can be found in the hair of neonates, suggesting it crosses the placental barrier with effects that are not [...]]]></description>
			<content:encoded><![CDATA[<p>TORONTO, Nov. 2 &#8212; Methamphetamine can be detected in the hair of newborns whose mothers used the drug during pregnancy, researchers here have found.<br />
Action Points</p>
<ul>
<li>Note that this study shows that methamphetamine used during pregnancy can be found in the hair of neonates, suggesting it crosses the placental barrier with effects that are not completely understood.</li>
<li>Advise patients who ask that drug abuse during pregnancy can be detrimental to the fetus, with a range of physical and intellectual sequelae, as well as hazardous to the mother.</li>
</ul>
<p>It represents the first direct evidence in humans that crystal meth, which is a growing drug-abuse problem in North America, can cross the placenta and affect the growing fetus, according to Facundo Garcia-Bournissen, M.D., of the Motherisk program at the Hospital for Sick Children.<br />
Researchers at the program have been testing hair samples from parents and adults across Canada for several years, usually when there is clinical suspicion of drug abuse on the part of parents, Dr. Garcia-Bournissen and colleagues reported in the online issue of Archives of Disease in Childhood.<br />
From June 1997 through December 2005, the database accumulated results of 34,278 tests for drugs in hair, representing 8,270 people. Nearly 60% (or 4,926) of these people were positive for at least one drug of abuse, the researchers said.<br />
In a retrospective analysis, Dr. Garcia-Bournissen and colleagues examined the incidence of methamphetamine in hair samples:</p>
<ul>
<li>The first methamphetamine was found in hair in 2003, when six samples tested positive, with a slight increase in 2004, with eight cases.</li>
<li>There were 372 cases in 2005 and the researchers said preliminary data for 2006 indicates that the surge has not stopped.</li>
<li>The study identified 11 mother-neonate pairs in which each was positive for methamphetamine.</li>
<li>Also, one newborn was negative, although the mother was positive.</li>
</ul>
<p>The median methamphetamine values in the mother-baby pairs were 1.75 ng/mg for the mothers and 1.63 ng/mg for the newborns. Dr. Garcia-Bournissen and colleagues said.<br />
The median concentrations were not significantly different, &#8220;suggesting that the transplacental transfer of methamphetamine is extensive,&#8221; the researchers said. On an individual level, maternal and neonatal drug levels correlated significantly (at P=0.003, using Spearman&#8217;s rho test, with r=0.8).<br />
Interestingly, among the 171 subjects who were positive for methamphetamine and whose hair was tested for other drugs, 83.5% were positive for at least one other drug, usually cocaine, Dr. Garcia-Bournissen and colleagues found.<br />
In contrast, among the 1,053 subjects negative for methamphetamine but positive for some other drug, only 38% were positive for more than one drug, they said.<br />
&#8220;Positive exposure to methamphetamine strongly suggests that the person is a polydrug user, which may have important implications for fetal safety,&#8221; the researchers said.<br />
The effects of the drug on the exposed child remain unclear, Dr. Garcia-Bournissen and colleagues noted, although there is some evidence that &#8220;children exposed in utero to methamphetamine are at risk of developmental problems, because of either the effect of direct exposure to the drug during pregnancy or growing in the environment associated with parental methamphetamine misuse, or probably both.&#8221;<br />
Because the study was retrospective and anonymous, clinical information on the exposed infants is not available, the researchers said.</p>
<p>Source: www.medpage.today.com 2nd Nov. 2006</p>
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		<title>Comprehensive gene atlas underlying drug addiction</title>
		<link>http://drugprevent.org.uk/ppp/2011/09/comprehensive-gene-atlas-underlying-drug-addiction/</link>
		<comments>http://drugprevent.org.uk/ppp/2011/09/comprehensive-gene-atlas-underlying-drug-addiction/#comments</comments>
		<pubDate>Tue, 20 Sep 2011 19:02:27 +0000</pubDate>
		<dc:creator>ndpa</dc:creator>
				<category><![CDATA[Addiction]]></category>

		<guid isPermaLink="false">http://drugprevent.org.uk/ppp/?p=7798</guid>
		<description><![CDATA[Using an integrative meta-analysis approach, researchers from the Center for Bioinformatics at Peking University in Beijing have assembled the most comprehensive gene atlas underlying drug addiction and identified five molecular pathways common to four different addictive drugs. This novel paper appears in PLoS Computational Biology on January 4, 2008. Drug addiction is a serious worldwide [...]]]></description>
			<content:encoded><![CDATA[<p>Using an integrative meta-analysis approach, researchers from the Center for Bioinformatics at Peking University in Beijing have assembled the most comprehensive gene atlas underlying drug addiction and identified five molecular pathways common to four different addictive drugs.<br />
This novel paper appears in PLoS Computational Biology on January 4, 2008.<br />
Drug addiction is a serious worldwide problem with strong genetic and environmental influences. So far different technologies have revealed a variety of genes and biological processes underlying addiction. However, individual technology can be biased and render only an incomplete picture. Studying individual or a small number of genes is like looking at pieces of a jigsaw puzzle &#8211; only when you gather most of the pieces from different places and arrange them together in an orderly fashion do interesting patterns emerge.<br />
The team, led by Liping Wei, surveyed scientific literature published in the past 30 years and collected 2,343 items of evidence linking genes and chromosome regions
